拮抗抑制制 的英文怎麼說

中文拼音 [jiékàngyìzhìzhì]
拮抗抑制制 英文
antagonistic inhibition
  • 抗 : Ⅰ動詞1 （抵抗; 抵擋） resist; combat; fight 2 （拒絕; 抗拒） refuse; defy 3 （對等） contend with...
  • 抑 : Ⅰ動詞（向下按; 壓制） restrain; repress; curb Ⅱ連詞[書面語]1 （表示抉擇） or 2 （表示轉折） but3 ...
  • 制 : Ⅰ動詞1 （製造） make; manufacture 2 （擬訂; 規定） draw up; establish 3 （用強力約束; 限定; 管束...
  • 抑制 : 1 （控制） restrain; control; check; hold up; curb; stop; repress; bridle; choke; prehension; sup...
  1. Among antihypertensive measures, blockade of the raas by means of ace inhibitors or angiotensin ii receptor antagonists recently emerged as a potentially polyvalent approach, not only for treating hypertension and reducing cardiovascular events, but also to prevent or reduce albuminuria, counteract diabetic nephropathy and lower the occurrence of new type 2 diabetes in individuals at risk

    在抗高血壓藥物治療中，通過用ace抑制劑或血管緊張素受體拮抗劑可阻滯腎素-血管緊張素-醛固酮系統激活，最近其可作為一種潛在多能的治療方法，不僅可治療高血壓和減少心血管事件，也能夠預防或減少蛋白尿，阻止糖尿病性腎病變，並使新糖尿病患者發生風險性降得更低。
  2. So far, six kinds of medicines are authorised to be applied to the treatment of hypertension by the world health organization ( who ), including diuretics, - receptor blocking agent, - receptor blocking agent, angiotensin receptor antagon, calcium channal blocking agent and angiotensin converting enzyme inhibitors ( aceis )

    目前，世界衛生組織（ who ）批準用於治療高血壓病的一線藥物主要有利尿劑、受體阻斷劑、血管緊張素轉換酶抑制劑（ acei ） 、鈣通道阻滯劑（ caa ） 、受體阻斷劑、血管緊張素（ ang ）受體拮抗劑等六大類。
  3. Follistatin antagonises myostatin by a direct protein - protein interaction, which prevents myostatin from executing its inhibitory effect on muscle development

    Myostatin是目前所知的最強的肌肉抑制因子， follistatin通過直接的蛋白結合而拮抗myostatin對肌肉發育的抑制作用，表現出增肌效應。
  4. Research interests : organic synthesis, including synthetic methodology, divergent oriented organic synthesis, asymmetric synthesis, asymmetric synthesis of small molecules with biological significance ( alkaloids, amino acids, pheromones, nucleosides analogues, enzyme inhibitors, receptor agonists and antagonists, bioactive heterocycles etc ) and chiral drugs

    研究興趣：主要從事有機合成化學及相關的化學生物學研究，包括合成方法學、多樣性導向有機合成、不對稱合成、有機小分子調節劑（生物堿，氨基酸，昆蟲信息素，酶抑制劑，受體拮抗劑，核苷類似物等）和手性藥物的不對稱合成。
  5. Bp23 celb genes, b. pwnilus endoglucanase and b. polymyxa beta - 1, 4 - endoglucanase " genes, respectively. it was recognized as a new gene encoding for endoglucanase of b. mega terium. the recombinant plasmid tvchi ( pmd18 ~ t inserted with chitinase encoding gene from aplls ) and e. coli - bacillus shuttle vector physooplk were digested by ecori and sail completely, and the chitinase gene was ligated with shuttle vector, and the recombinant vector was used to transform b. megaterium ap25 competent cell

    平板拮抗實驗同野生菌株相比，轉化子對麥長蠕抱菌的抑制作用最明顯，抑制百分數最高可達33 . 3 % ，而apll3和ap25分別是23 . 1 %和25 . 6 % ，同時轉化子對小麥紋枯病菌、棉花立枯病菌、棉花枯萎病菌和小麥的全蝕病菌也具有較為明顯的抑制作用。
  6. Clinical study of the protective effect of acei and arb on patients with diabetic nephropathy

    血管緊張素轉換酶抑制劑和血管緊張素受體拮抗劑對糖尿病腎病患者腎功能的保護作用
  7. The effects of arsenic on anticomplementary competence of testis of rat and the antagonism of selenium

    砷對睪丸補體抑制水平的影響及硒的拮抗作用
  8. Compared to the single hg2 + treated, se4 + + hg2 + the mixed exposure could restrain the fall of the activity of t ? aoc, sod and gsh ? px, reducing the level of lipid peroxidation, and alleviating membrane damage, represented with the heighten na + - k + - atpase activity and reduced mda concentration, resulting in the increased resistance to hg2 + damage. however, se4 + were incapable of preventing from the damage of hg2 + ultimately, along with the exposure period, regardless of the fact that the obvious enhancement of t - aoc and atpase activity

    混合處理后，與單一汞處理相比， se ~ （ 4 + ）在一定程度上能有效抑制汞脅迫引起的t ? aoc ， sod ， gsh ? px的降低，降低膜脂質過氧化水平，減輕了對膜的硒對汞致劍尾魚抗氧化系統的毒害和生理損傷的拮抗作用損傷，表現為隨一k + / atpase活力升高，而mda含量降低，提高了機體對hg葉毒害的抗性;但隨著染毒時間延長，硒不能最終阻止毒害的加深。
  9. " the concept is that systemic sclerosis, which is a severe disease characterized by systemic fibrosis, is at the beginning mainly a vascular disease, and we hypothesize that using major vascular drugs ( calcium channel blockers, endothelin antagonists, prostacyclins, phosphodiesterase inhibitors, etc. ) from the beginning and at appropriate dosage may interfere with the fibrotic process which seems secondary, " dr

    「從概念上講，系統性硬化是一種以系統性的纖維化變性為特徵的嚴重疾病，早期主要表現在血管性的病變，因此我們推測早期、適量地應用主要作用於血管的藥物（如鈣離子阻斷劑，內皮素拮抗劑，前列環素，磷酸二酯酶抑制劑等）可能可以干預似乎是繼發發生的纖維化過程。 」
  10. " the concept is that systemic sclerosis, which is a seere disease characterized by systemic fibrosis, is at the beginning mainly a ascular disease, and we hypothesize that using major ascular drugs ( calcium channel blockers, endothelin antagonists, prostacyclins, phosphodiesterase inhibitors, etc. ) from the beginning and at appropriate dosage may interfere with the fibrotic process which seems secondary, " dr

    「從概念上講，系統性硬化是一種以系統性的纖維化變性為特徵的嚴重疾病，早期主要表現在血管性的病變，因此我們推測早期、適量地應用主要作用於血管的藥物（如鈣離子阻斷劑，內皮素拮抗劑，前列環素，磷酸二酯酶抑制劑等）可能可以干預似乎是繼發發生的纖維化過程。 」
  11. Mechanism of glutamate - induced inhibition of pulmonary surfactant lipid synthesis ? mda receptor ( nmdar ) antagonist mk - 801 could reverse glutamate - induced inhibition of [ 3h ] - choline incorporation in lung explants ; ? he expression of nmdar1 subtype existed in alveolar type ii epithelial cells ; ? he effect of glu on pc synthesis required l - arginine in cultured lung explants, nos activity and no production were increased significantly in lung explants with glu treatment ; and mk - 801 and nos inhibitor l - nna could block the increase of nos activity and no production induced by glu ; l - nna also could reverse the inhibition of [ 3h ] - choline incorporation induced by glu ; ( 4 ) mda content increased and sod level decreased in cultured lung explants of glu treament groups, mk - 801 and l - nna could reverse these effects ; ( 3 ) mk - 801, l - nna could block the release of ldh induced by glu

    谷氨酸對肺表面活性物質脂質合成抑制的機制研究nmda受體拮抗劑mk - 801能逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應；肺泡上皮細胞有nmda受體nmdar1亞單位的表達； glu對ps脂質合成的影響依賴l -精氨酸的存在， glu可使肺組織勻漿中nos活性、 no合成量升高； mk - 801 、 nos的抑制劑l - nna均可阻斷glu所致的這些效應； l - nna可逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應； glu可使肺組織勻漿中mda碩士學位論文中文摘要含量增高、 sod水平下降。 mk七0 、 l nna能阻斷q所致的這些效應； mk80 、 l nna還能阻斷gill誘導的ldh釋放增多的效應。
  12. The experimental results showed that tongbian fang had the antagonistic effect on the inhibition of small intestine propulsion induced by adrenaline, atropine and morphine hydrochloride in mice, increased the water volume in intestinal cavities, excited the spontaneous movement of rabbit duodenum in vitro, antagonized the adrenaline - induced inhibition of duodenum movement in vitro and the acetylcholine - induced contraction of ileum in vitro

    研究結果表明，通便方能拮抗腎上腺素、阿托品、鹽酸嗎啡引起小鼠小腸推進的抑制；能顯著增加小鼠腸腔水分；對家兔離體十二指腸自發運動有興奮作用，能拮抗腎上腺素引起的離體十二指腸運動抑制和乙酰膽堿引起離體回腸的強直性收縮。
  13. It is well known that tumor necrosis factor a ( tnfa ) can play very different character in defence mechnism of body and pathologic injury, in which it can inhibit or kill tumor cell, and induce inflammation against infection ; on the other hand, tnfa can be as a very important mediator to cause some serious pathologic processes such as septic shock, gvhr and some autoimmune diseases

    腫瘤壞死因子（ tumornecrosisfactor ， tnf ）具有對某些惡性腫瘤的抑制或殺傷作用，也是致內毒素性休克、急性移植物抗宿主病及一些自身免疫病的重要效應因子，因此tnf的改造和tnf拮抗劑的研究受到各國學者的重視。
  14. The main physiology - biochemistry changes were determined in the two fruits after treating with trifluoperizine, a caimodulin antagnist, and verapamil, a calcium channel blocker. the results showed that trifluoperizine and verapamil weakened the effects of the exogenous ethylene on the the main physiology - biochemistry

    鈣調素拮抗劑tfp和鈣通道阻塞劑ver抑制了外源乙烯促進果實衰老時的乙烯釋放量、 o _ ~ -的積累和膜脂過氧化產物mda的增加。
  15. 10 should ace inhibitors be combined with angiotensin ii receptor antagonists

    Ace抑制劑應與血管緊張素受體拮抗劑合用嗎？
  16. Effect of vitamin c antagonism on fluorine - induced inhibition of antioxidant enzymes activities in human lymphocyte in vit

    拮抗氟抑制體外培養淋巴細胞抗氧化酶活性的研究
  17. At discharge, 86 % of patients were receiving beta blockers and 76 % were receiving either ace inhibitors of angiotensin receptor blockers

    在出院的時， 86 %的患者服用受體阻滯劑，而76 %的患者服用ace抑制劑或者血管緊張素受體拮抗劑。
  18. During hs at 44, the cam antagonists w7 and cpz als o inhibited dna - binding activity of the hsf in a concentration - dependent manner, but not by w5, an inactive structural analogue of w7

    Cam拮抗劑w7和cpz都可抑制熱激誘導的hsf的dna結合活性，而w7的類似物w5無此抑制作用。
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