毒害細胞的 的英文怎麼說

中文拼音 [dúhàixìbāode]
毒害細胞的 英文
cytotoxinic
  • 毒 : Ⅰ名詞1 （對生物體有害的性質或物質; 毒物） poison; toxin 2 （毒品） drug; narcotics 3 （姓氏） a s...
  • 細 : 形容詞1 （條狀物橫剖面小） thin; slender 2 （顆粒小） in small particles; fine 3 （音量小） thin ...
  • 胞 : Ⅰ名詞1 （胞衣） afterbirth2 （同一個國家或民族的人） fellow countryman; compatriot Ⅱ形容詞（同胞...
  • 的 : 4次方是 The fourth power of 2 is direction
  • 毒害 : poison （sb. 's mind）; infect
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  1. Recently, the chinese plant protection agencies reported the growing hardships in controlling some field populations of oriental migratory locust with organophosphate ( op ) compounds. up to now there are more than 600 arthropod species with documented resistance to one or more insecticides and / or acaricides since resistance phenomenon was first described in san jose scale to lime - sulfur in washington in 1908. enhanced metabolic detoxification and reduced sensitivity of insecticide target - sites are the two major mechanisms in resistance development, involving three primary metabolic enzymes of esterases, glutathione s - transferases and cytochrome p450 polysubstrate monooxygenases

    代謝抗性是指對殺蟲劑起水解或隔離作用的酶發生改變，從而對殺蟲劑起到解毒作用，昆蟲主要解毒酶系有酯酶、谷胱甘肽- s -轉移酶、細胞色素p450單加氧酶等，這三種酶系任何一種的組成部分發生改變均會引起害蟲抗性的改變；靶標抗性是指由於殺蟲劑作用靶標敏感度降低而產生的抗性。
  2. Using cd, cu, zn and cr as experimental toxicants and crucian as experimental creature, we employed the environmental biotechniques ( flame atomic absorption spectrophotometry, the method of improved pyrogallol autoxidation and electron microscopy ) to study the acute toxicity and secure concentrations of heavy metals to crucian ; the accumulation and distribution of mixed heavy metals to fingling crucian ; the activities of superoxide dismutase ( sod ) of gill and liver tissues and the hispathological and ultrastructural change of superfine structure of liver and kidney of crucian after cadmium exposure. through which, we intended to fully and systemically study the toxic influence of heavy metals to fish, and preliminarily discuss the influence of heavy metals pollution to the diversified level of fish such as individual, organ, tissue, cell and molecule. thus we tried to provide scientific evidence to establish preventative management measures, avoid and relieve the harm of heavy metals pollution to aquicolous ecosystem in time

    隨后以這幾種重金屬作為實驗毒物，以鯽魚為實驗動物，應用環境生物技術、火焰原子吸收分光光度法、鄰苯三酚自氧化法、透射電鏡等技術研究了重金屬cu 、 zn 、 cd和cr對鯽魚的急性毒性及其安全濃度評價、混合重金屬在鯽魚幼體組織內的積累和分佈、 cd對鯽魚鰓和肝臟中sod活性的影響、 cd對鯽魚肝細胞和腎細胞超微結構的影響等，全面和系統地研究了水環境中重金屬暴露對魚類的毒性影響，初步探討了重金屬污染對魚類的個體? ?器官? ?組織? ?細胞? ?分子等各水平層次的影響，為制定漁業生產上預防性的管理措施提供科學依據，及時避免或減輕重金屬污染對水生生態系統造成的損害。
  3. 3 the salt - resistant characteristic which is adapt to poisonousness of salt hydronium, such as salt - gland, crystal cell, and soon

    適應鹽離子毒害的抗鹽結構特徵。如鹽腺、含鹽液泡包、含晶細胞等。
  4. Plant defensin disrupts the cell membrane of phytopathogen differently from cema that contacts directly with the cell membrane. plant defensins are thought to interact with specific receptor harboring in the cell membrane

    由於對真菌，特別是絲狀真菌有很強的抑制作用，而對植物細胞沒有毒害作用，防禦素有望在植物抗病基因工程中得到廣泛應用。
  5. Plants are thought to remove na + from the cytoplasm by transporting it into the vacuolar or out of the cell using na + / h + exchangers localized in the vacuolar and plasma membranes, respectively. sos1 encoding a plasma membrane na + / h + antiporter and atnhxl encoding a vacuolar na + / h + antiporter were isolated from glycophytic arabidopsis thaliana, and overexpression of atnhxl and sos1 in arabidopsis thaliana increased the salt tolerance of transgenic plants significantly

    目前，擬南芥細胞內控制na ~ +外排的基因sos1及離子區隔化基因atnhx1均已克隆， sos1及atnhx1在擬南芥中的過量表達顯著提高了轉基因植株的耐鹽性，開創了降低na ~ +毒害的基因操作新途徑。
  6. One is the accumulation of the solute osmo - protectants, the other is the mechanisms of ion homeostasis including na + extrusion system and na + compartmentation into the vacuolar to reduce the toxic effects of this cation

    一方面增加細胞內可溶性物質，另一方面則通過na ~ +外排或na ~ +區隔化機制來維持胞質內較低的na ~ +濃度，以消除na ~ +的毒害。
  7. The variety of the activity of na + / k + - atpase, ca2 + - atpase and sod by increase calcium concentration in the water, and the effects of cd2 +, estradiol - 17 and ca2 + on the lymphocyte proliferation from blood and head kidney of ctenopharyngodon idellus were also investigated the significance of this paper is that to research the toxicity of cadmium to the fish and to search the mechanism of the cadmium toxicity to the fish. and the protection of calcium on cd2 + toxicity

    本論文研究了鎘對草魚的毒性作用，比較了在不同鈣濃度條件下，其鰓、肝臟、腦等組織中sod 、 na ~ + k ~ + ? atp酶、 ca ~ （ 2 + ） ? atp酶的活性變化規律以及鎘和17 ?雌二醇對草魚血液及頭腎淋巴細胞增殖的影響，評價鎘對魚類的毒害作用，探索鎘的毒害機理。
  8. This paper expounds the progresses of the researches on the life science and relevant subjects, which include the research of cultivating the arterial vessel with fine cell tissue, the research on the genetically modified animal, the research on the pest control with bio - virus pesticide, the research on the death mechanism of protein and the diseases of immune system ( including cancer ), the research on anti - senile protein and the research on compound - type aids vaccine

    闡述了生命科學及其相關學科的研究進展，包括用細小細胞組織培製成動脈血管的研究、轉基因動物的研究、生物病毒農藥防治蟲害的研究、蛋白質死亡機理及免疫系統疾病（包括癌癥）的研究、抗衰老蛋白的研究以及復合型艾滋病疫苗的研究。
  9. The propagules of b. gymnorrhiza and a. corniculatum undergo some growth before becoming detached from the parent plant. they accumulated large amounts of ions during the development while the level of ion concentrations are consistently lower in their embryos compared to leaves of parent plant. before detachment, the growth of hypocotyls were reduced, the water content declined, while the osmotic potential was approaching to or more negative than that of the solution on which their parent plant vegetating

    另一方面維持較大比例的有機滲透調節劑，控制細胞液中鹽離子的水平以減少代謝毒害；木欖成熟胚軸的密度接近或低於生境中海水的密度，適于隨水漂浮和傳播；胚軸內離子濃度呈下降趨勢，但最終以達到和母樹生境中的離子濃度水平相一致為目標。
  10. In this paper, signal peptide was added before the n - terminus of cema with the intention to reduce its toxic to plant cells while maintaining the strong antibacterial activity

    本文通過在cema的n端添加信號肽，對其進行改造，以探索不降低抑菌作用的同時，降低其對植物細胞毒害作用的可能性。
  11. La3 + has greater ability of relieving the toxic effect of ni3 + to hydrocharis dubia b l. backer than that of ce3 +. however, the relif ability of rare earth to the stress of heavy metal was limited

    在狐尾藻葉片細胞超微結構水平上也能見到cr ~ （ 6 + ）的毒害作用在稀土元素la ~ （ 3 + ）的作用下得到緩解。
  12. The chances of heavy metal into the plant cell were reduced and stress alleviated. ultrastructural observation of leaf cells of myriophyllum verticillatum linn, showed that la3 + coule alleviate the injury of cell structure produced by cr6 +

    對元素含量的測定表明，稀土元素降低了植物對重金屬的富集量，兩者表現出拮抗作用，減少重金屬離子進入植物葉片細胞的機會，從而減輕了毒害作用。
  13. The protein encoded by orf1 show strong homology with cation efflux family protein from geobacter sulfurreducens pca [ identities = 65 / 272 ( 23 % ) ], and contains mmt1 - like putative conserved domains kog1485, mmt1 contains conserved domains kog1485 is mitochondrial fe2 + transporter ( cation diffusion facilitator superfamily ). so it can be considered that the protein encoded by o rf1 is fe2 + transporter involved in magnetosome biosynthesis in magnetospirillum gryphiswaldense msr - 1

    經同源比較和保守性結構域分析，推測orf1編碼的蛋白具有fe ~ （ 2 + ）轉運功能，在磁小體合成過程中將細胞質中fe ~ （ 2 + ）轉運到磁小體囊泡中，合成磁小體的同時去除fe ~ （ 2 + ）對細胞的毒害作用。
  14. In terms with the principle of fusarium oxysporiun caused plant disease : bundles were blocked and fusarid acid killing cells was formed by hyphae so that caused water metabolism abnormal and plant wilting. in order to find out effective method of anti - fiisarium oxysporuin, long ya lillium was taken as material with plant tissue culture and genetic transformation techniques in this paper

    針對尖孢鐮刀菌的致病機理：菌絲阻塞維管束引起水分代謝失常和菌絲在植物體內產生毒素（鐮刀菌酸）損害膜結構造成代謝失常，從而導致植物萎焉。本實驗以龍牙百合為研究對象，應用細胞工程中的離體培養方法並結合轉基因技術，以期找到抗尖孢鐮刀菌的有效途徑。
  15. The alpha particles released from polonium - 210 have a very low penetrating power and can be stopped easily by the outer layer of skin or a sheet of paper. it is unlikely that the particles can affect human body through direct irradiation. however the ionization power of alpha particles is very high

    釙210釋放的阿爾法粒子穿透能力很弱，人類的皮膚或一張紙已能隔阻它，粒子經照射影響人體的機會很微，不過由於它的電離能力很強，如果人類吸入或進食含有釙210的物質，其釋放的阿爾法粒子就能直接破壞內臟細胞，即使所吸收的份量不多，也會損害身體健康，急性高劑量阿爾法輻射可以引致輻射中毒。
  16. The major factors for progressie loss of ? - cell function and mass are glucotoxicity, lipotoxicity, proinflammatory cytokines, leptin, and islet cell amyloid

    導致細胞的功能與體積進行性損害的因素包括糖毒性、脂肪毒性、免疫炎性因子、瘦蛋白與胰島細胞澱粉樣變。
  17. The major factors for progressive loss of ? - cell function and mass are glucotoxicity, lipotoxicity, proinflammatory cytokines, leptin, and islet cell amyloid

    導致細胞的功能與體積進行性損害的因素包括糖毒性、脂肪毒性、免疫炎性因子、瘦蛋白與胰島細胞澱粉樣變。
  18. Autoclaves, microwave systems and chemical disinfection equipment cannot remove the toxicity or destroy the residual amount of cytotoxic drugs, pharmaceuticals and other chemicals in the syringes and on sharp instruments such as scalpels

    蒸壓消毒微波消毒及化學消毒設備往往不能去除或破壞留于針筒及尖銳器材如解剖刀的殘餘毒素和毒害細胞的藥物及其他化學劑。
  19. Hence, it is important to bring these cells down in number in order to leave room in the " immunological space " for others to expand as needed ; we probably don ' t need to worry that this would allow the virus to harm us ( though it would certainly be good to get rid of the virus too if we can, and work is intensive on this, because people with a compromised immune system, such as aids sufferers, often experience serious symptoms from these viruses )

    因此，重要的是把這些細胞的數目降下來，把"免疫學空間"留給需要的免疫細胞類型去擴展；我們也許不必擔心這會讓病毒傷害我們（雖然，如果我們能夠，也把病毒清除掉，那敢情好，在這方面研究工作正在緊張進行，因為有危及安全的免疫系統的人，如艾滋病患者，常常因這些病毒而經受嚴重的癥狀） 。
  20. The maladies caused by salt stress arise from the disruption of cellular aqueous and ionic equilibrium. na + accumulates excessively in the cytoplasm can be toxic to the metabolism and affects the regulation mechanism of the plant cell

    鹽脅迫條件下，植物細胞滲透均衡和離子正常均衡狀態被破壞，特別是胞質中過多的na ~ +滲透調節對植物細胞的新陳代謝產生毒害，影響到植物細胞的調節機制。
分享友人
分享到 Line

分享到臉書