突觸可塑性 的英文怎麼說
中文拼音 [tūchùkěsùxìng]
突觸可塑性
英文
synaptic plasticity-
Subject covers all major areas of cellular and molecular neurobiology including excitable cells and membranes, ion channels and receptors, synaptic transmission, cell type determination, axon guidance and targeting, neuronal cell biology, synapse formation and plasticity
本學科涵蓋了細胞分子神經生物學的所有主要領域，包括興奮細胞和細胞膜、離子通道和受體、突觸傳遞、細胞類型決定、軸突導向和定位、神經元細胞生物學、突觸形成和可塑性。Liu qian, supervisor : wang huai - jing. title : effect of cuzhifang on ethology and synaptic plasticity in hippocampus of aged learning and memory impaired rate
劉謙，導師：王懷經教授。論文題目： 「促智方對老年學習記憶減退大鼠行為學及海馬突觸可塑性的實驗研究」 。 2001 - 2004 。Postsynaptic calcium pathway contributes to synaptic plasticity between cones and l - type horizontal cells of the retina
突觸后鈣通路有助於視網膜視錐與l型水平細胞之間的突觸可塑性The effects of metabotropic glutamate receptors in synaptic plasticity
代謝型谷氨酸受體在突觸可塑性中的作用研究進展Using this system, we have studied matrine - inhibittory effect and trifluoperation - neuroprotection effect in hippocampal slices, also discussed the mechanism of long - term potentiation using anesthetic rats. the experiment results showed that matrine can inhibit the hyperactivity induced by penicillin sodium in dosage by changing the relative parameters of field potential ; trifluoperation can alter ps change with the time, enhance the degree and the ratio of ps recovery, then minis the hypoxic injury ; high frequency stimulate can increase ps amplitude and epsp slope for long time, buildup the in / out function of nerve cells, and enhance synaptic plasticity
結果表明，苦參堿能夠劑量依賴性地抑制青霉素誘導的神經元順向信號傳導激活過程，使細胞外記錄到的場電位各個參數發生相應改變；三氟拉嗪可以改變ps的時相變化，提高ps的恢復程度和恢復率，減小了神經元因缺氧引起的不可逆損傷；高頻刺激（ highfrequencystimulate ， hfs ）可以長時間的增強ps的幅度和epsp的斜率，進而增強神經元的輸入輸出功能，增加了突觸的可塑性。Accumulating evidence indicates that nmda receptors are involved in many complex physiological and neuropathological mechanisms, such as neuronal developmental plasticity, long - term potentiation ( ltp ), learning and memory, excitotoxicity and neurodegenerative diseases etc. three gene families that encode nmda receptor subunits have been identified : nr1, nr2a - d, and nr3 subunit
已知nmda受體與許多復雜的生理和病理機制有關，如突觸的可塑性、長時程增強作用（ ltp ） 、學習和記憶、興奮性神經毒性、神經退行性變性疾病等。已克隆到3個編碼nmda受體亞單位蛋白的基因家族，即nr1 、 nr2和nr3 。1. the relationship between protein kinase c and remember behavior alteration of mice exposed to lead. introduction lead has high affinity to nerve and it can be accumulated in nerve tissues, which can cause long - period damage to nervous system, especially to memory and learning function
一、慢性鉛暴露小鼠腦組織蛋白激酶c活性變化及其與記憶行為改變的關系前言學習記憶是腦的高級功能，其物質基礎是中樞神經系統高度的可塑性，包括神經網路、神經環路及突觸連接等不同水平的可塑性。The predominantly expressed nr2 subunits in hippocampus and neocortex are nr2a and nr2b, they have different developmental expression profiles - there is a developmental decrease in expression of the nr2b subunit and an increase in expression of the nr2a subunit, which endow the nmda receptor with distinct channel properties
而隨著年齡的增長， nr2a亞單位的表達量和突觸含量逐漸增加。不同的nr2亞單位賦予nmda受體不同的通道動力學特性，如在老年動物中觀察到nr2b向nr2a亞單位的轉換降低了突觸可塑性。The related substance of synaptic plasticity and its change after cerebral ischemia
突觸可塑性相關物質及其在腦缺血后的變化In the present work, we investigated the synaptic plasticity and neural circuitry in the outer retina using the electrophysiological method and model analysis
本論文從實驗和模型兩方面研究了視網膜外網狀層的突觸可塑性和神經元迴路。Influence of tiantai no. 1 recipe on synaptic plasticity of the hippocampal ca1 region of spontaneous alzheimer disease models : quantitative research of ultrastructure
1區分子層突觸可塑性的影響超微結構定量研究Effects of electroacupuncture on synaptic plasticity of hippocampal neurons in model rats with alzheimer disease
電針對阿爾茨海默病模型大鼠海馬神經元突觸形態可塑性的影響機制分享友人