細胞勻漿 的英文怎麼說

中文拼音 [xìbāoyúnjiāng]
細胞勻漿 英文
cell homogenate
  • 細 : 形容詞1 （條狀物橫剖面小） thin; slender 2 （顆粒小） in small particles; fine 3 （音量小） thin ...
  • 胞 : Ⅰ名詞1 （胞衣） afterbirth2 （同一個國家或民族的人） fellow countryman; compatriot Ⅱ形容詞（同胞...
  • 勻 : Ⅰ形容詞（均勻） even Ⅱ動詞1. （使均勻） even up; divide evenly 2. （分出一部分） spare
  • 漿 : 漿同「糨」
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  1. The tumor is composed of small, uniform, round acini and tubular structures separated by scant stroma. the lining epithelial cells are uniform with hyperchromatic nuclei and hae scant cytoplasm

    腫瘤的胞核小而圓，大小一致，形成管狀結構，與周圍少量的間質分隔開。瘤細胞大小均勻，核深染，胞漿少。
  2. Finally, the anion exchanger was used to recover dehalogenase from unclarified cell homogenate, while the cation exchanger was introduced to purify nattokinase directly from fermentation broth

    最後，所開發的陰離子吸附劑被應用於從細胞勻漿中提取脫鹵酶，而陽離子交換劑則被用來從發酵液中提純納豆激酶。
  3. The glandular clusters hae punched out lumens. the cytoplasm is clear or pale eosinophilic. the nuclei are uniform and lack nucleoli. basal cells can be clearly seen

    簇狀分佈的腺體已經突入腺腔。胞漿透明或蒼白，呈嗜酸性染色。胞核大小均勻，核仁不明顯。基底細胞清晰可見。
  4. Mechanism of glutamate - induced inhibition of pulmonary surfactant lipid synthesis ? mda receptor ( nmdar ) antagonist mk - 801 could reverse glutamate - induced inhibition of [ 3h ] - choline incorporation in lung explants ; ? he expression of nmdar1 subtype existed in alveolar type ii epithelial cells ; ? he effect of glu on pc synthesis required l - arginine in cultured lung explants, nos activity and no production were increased significantly in lung explants with glu treatment ; and mk - 801 and nos inhibitor l - nna could block the increase of nos activity and no production induced by glu ; l - nna also could reverse the inhibition of [ 3h ] - choline incorporation induced by glu ; ( 4 ) mda content increased and sod level decreased in cultured lung explants of glu treament groups, mk - 801 and l - nna could reverse these effects ; ( 3 ) mk - 801, l - nna could block the release of ldh induced by glu

    谷氨酸對肺表面活性物質脂質合成抑制的機制研究nmda受體拮抗劑mk - 801能逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應；肺泡上皮細胞有nmda受體nmdar1亞單位的表達； glu對ps脂質合成的影響依賴l -精氨酸的存在， glu可使肺組織勻漿中nos活性、 no合成量升高； mk - 801 、 nos的抑制劑l - nna均可阻斷glu所致的這些效應； l - nna可逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應； glu可使肺組織勻漿中mda碩士學位論文中文摘要含量增高、 sod水平下降。 mk七0 、 l nna能阻斷q所致的這些效應； mk80 、 l nna還能阻斷gill誘導的ldh釋放增多的效應。
  5. 3. calcitonin gene - related peptide attenuates glutamate - induced inhibition of pulmonary surfactant lipid synthesis ( d cgrp could reverse the decrease of [ 3h ] - choline incorporation induced by glu with dose - dependence ; ( 2 ) cgrp could block the decrease of the content of cct a mrna induced by glu in lung tissue ; ( 3 } cgrp could reduce the impairment of the ultrastructure of at ii cells induced by glu ; ( 4 ) cgrp could reverse the increase of mda content and decrease of sod level induced by glu in cultured lung explants, respectively ; ? grp had no significant effect on nos activity and increase of no production induced by glu

    降鈣素基因相關肽減輕谷氨酸所致肺表面活性物質脂質合成抑制的保護作用降鈣素基因相關肽grp ）可顯著減輕0所致肺組織h一膽堿摻入pc量的降低，並且呈劑量依賴保護效應； cgrp可逆轉gill所致cctqinrna含量的降低； cgrp可減輕o所致肺11上皮細胞超微結構的損傷； cgrp可逆轉o所致肺組織勻漿中mda含量增多、 sod水平降低的效應，並可逆轉q所致肺組織ldh釋放增多的效應； cg販對gill引起的nos活性和no含量的升高均沒有顯著性影響。
  6. Results the levels of endotoxin, leukocyte, mda and sod were all signifificantly higher than control group, while, the level of glutathione ( gsh ) was much lower in experimental group ( p < 0. 01 )

    結果模型組內毒素、肺勻漿脂質過氧化物，以及白細胞計數均明顯增高；而還原谷胱甘肽（ gsh ）明顯降低，與對照組比差異有顯著性（ p < 0 . 05 ） 。
  7. When the approach to get a direct binding molecule was proved not fruitful, attention was turned to find binding sources for the purpose of further isolating the binding protein. the first considered and also the relatively easier - obtained materials are some adhesion molecules. results showed, only n - cam 140 showed ambiguous binding

    ， idin砰proteina磁珠為結合反應載體，從小鼠成年全腦勻漿作為結合起始材料，以蔗糖密度梯度離心法進4行細胞亞組分分離，以sds page顯示陽性結合條帶，以westernblot確定蛋白身份。
  8. Methods : in cultured lung explants without serum, the lipid component synthesis of pulmonary surfactant was evaluated in [ 3h ] - choline incorporation ; mrna content of phosphocholine cytidylyltransferase ( cct ) in lung explants was investigated in rt - pcr ; the changes of the ultrastructure of the at ii cells were observed with electron microscope ; the expression of nmdar1 subtype was observed in immunohistochemistry staining ; nitric oxide synthase ( nos ) activity, nitric oxide ( no ) content, superoxide dismutase ( sod ) level, malondialdehyde ( mda ) content and lactae dehydroase ( ldh ) level were determined by biochemistry methods. results : 1. influence of glutamate on synthesis of the lipid component of pulmonary surfactant ? with l - arginine, glu inhibited [ 3h ] - choline incorporation with good dose - dependence and time - dependence ; ( 2 ) mrna content of cct of the glu treatment groups was decreased ; ( 3 ) glu increases the release of ldh in cultured lung explants ; ( dwith electron microscope histochemistry, glu induced the changes of the ultrastruture of at ii iv cells

    方法：採用成年大鼠肺組織無血清培養，運用[ ~ 3h ] -膽堿摻入法測定ps主要脂質磷脂酰膽堿（ pc ）合成量； rt - pcr擴增檢測肺組織中pc合成限速酶磷酸膽堿二胞苷酰基轉移酶（ cct ） mrna含量；透射電子顯微鏡法觀察肺泡型上皮細胞和ps系統超微結構的變化；免疫組織化學染色檢測glu的受體nmdar1亞單位的表達；生化測定肺組織乳酸脫氫酶（ ldh ）釋放量和肺組織勻漿中一氧化氮合酶（ nos ）活性、一氧化氮（ no ）生成量、超氧化物歧化酶（ sod ）水平以及丙二醛（ mda ）含量。
  9. The cellular localization of hse was done by using in situ hybridization, the results showed that primary spermtatocytes and spermatids in seminiferous tubular have positive signals. the subcellular localizations of hsei and hseii were identified by gfp fusion protein. hsei - gfp fusion protein was evenly distributing in cytoplasm, while hseii - gfp was not evenly distributing in cytoplasm and there were many bright spots in cytosol

    用gfp高合蛋白的技術確定hsei和hsell在細胞中勺定位，結果顯示hsei gfp均勻地分佈於細胞漿中，而hsell0fp則分佈不均勻，在胞漿有聚集成許多熒光信號很強的點。
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