細胞毒性機制 的英文怎麼說

中文拼音 [xìbāodúxìngjīzhì]
細胞毒性機制 英文
cytotoxic mechanism
  • 細 : 形容詞1 （條狀物橫剖面小） thin; slender 2 （顆粒小） in small particles; fine 3 （音量小） thin ...
  • 胞 : Ⅰ名詞1 （胞衣） afterbirth2 （同一個國家或民族的人） fellow countryman; compatriot Ⅱ形容詞（同胞...
  • 毒 : Ⅰ名詞1 （對生物體有害的性質或物質; 毒物） poison; toxin 2 （毒品） drug; narcotics 3 （姓氏） a s...
  • 性 : Ⅰ名詞1 （性格） nature; character; disposition 2 （性能; 性質） property; quality 3 （性別） sex ...
  • 機 : machineengine
  • 制 : Ⅰ動詞1 （製造） make; manufacture 2 （擬訂; 規定） draw up; establish 3 （用強力約束; 限定; 管束...
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  • 毒性 : [藥理學] toxicity; virulence; poisonousness毒性測定 toxicity test
  • 機制 : 1 （機器製造或加工的） machine processed; machine made 2 （機器的構造和工作原理; 有機體的構造、功...
  1. This commensal organism is part of the gastrointestinal tract flora and can become extremely virulent, often in the setting of immuno - suppression such as neutropenia, occult malignancy ( commonly caecal ) and poorly controlled diabetes

    這種（與人類）共生生物是胃腸道菌群的一部分，可以變得毒性巨大，通常發生於機體免疫受抑制，如中性粒細胞減少癥，隱蔽的腫瘤（一般如盲腸） ，或者控制不佳的糖尿病。
  2. The mechanism enhancement of the optical brightener is not known. shapiro et al. postulated that selected brightener including m2r inhibit or alter the chitinous peritrophic membrane ( pm ), creating gaps in the membrane or gut lining and perhaps allowing more virions to pass from the gut lumen into the hemocoel

    光增白劑對桿狀病毒的增效作用的機理存在兩種推測一種觀點認為光增白劑是通過破壞圍食膜結構的完整性，促使更多的病毒粒子穿越圍食膜而發動感染的；另一種意見認為光增白劑能延遲中腸上皮細胞的脫落，促進病毒的復制繁殖。
  3. Objective the present study was to investigate the effects of endotoxin on capacitation and acrosome reaction ( ar ) of mouse, golden hamster and human sperm, on the sperm - oocyte fusion in mouse, and on the development of 1 - cell, 2 - cell and zona - free 2 - cell mouse embryos in vitro. the purpose was to definite the committed step and the mechanism during in vitro fertilization ( ivf ) on which endotoxin affected, and to distinguish the sensitivities to endotoxin of three developmental systems of mouse embryos. all these data would provide the reference to clinical and laboratory quality control

    目的研究內毒素對小鼠、金黃地鼠及人精子的體外獲能和頂體反應、小鼠精卵結合及小鼠1 -細胞、 2 -細胞和去卵透明帶2 -細胞胚胎體外發育的影響，探討內毒素影響體外受精結局的環節及可能的機制，確定三個體外培養系統對內毒素的敏感性，以期為臨床和實驗室質量控制提供參考。
  4. One is the accumulation of the solute osmo - protectants, the other is the mechanisms of ion homeostasis including na + extrusion system and na + compartmentation into the vacuolar to reduce the toxic effects of this cation

    一方面增加細胞內可溶性物質，另一方面則通過na ~ +外排或na ~ +區隔化機制來維持胞質內較低的na ~ +濃度，以消除na ~ +的毒害。
  5. Functions : this product contains “ antifungal active protein ” with optimal and strong sterilization effect, which could strengthen the immunity of body fluid, activate the macrophage, strengthen the phagocytosis ability of macrophage, strengthen the body immunity, restrain the growth, pervasion and transfer of abnormal cells, the product has strong sterilization effect and could strengthen the disease resistance ; it is remarkably effective in improving the immunity and improving the infirm constitution of pets ; the amino acid content and composition in the product are moderate and rational, with the characteristics of strong palatability, nutrition balance and immune element abundance, etc

    功能：本產品中擁有極佳強烈殺菌作用的「抗菌活性蛋白」 ，能增強體液免疫功能，活化巨噬細胞，增強其吞噬能力，可增強機體免疫力，抑制非正常細胞生長、擴散和轉移，具有強烈的殺毒作用，增強抗病性；對提高寵物免疫力，改善虛弱體質有顯著效果；其中氨基酸含量適中、組成合理，具有適口性強、營養均衡和免疫物質豐富等特點。
  6. No, a first gas information molecule discovered in human being, is a typical endothelial - derived relaxant and mediates endothelium - dependent relaxation of blood vessels. in the pathogesis of endotoxin shock vec is one of the major target cells of lps and lps - induced proinflammatory cytokine such as tumor necrosis factor and interlukin 1 and activated. in vec inducible nitric oxide synthase ( inos ) is induced and lead to an increase in production of no, the while endothelial nitric oxide synthase ( enos ) is inhibited and elicit decrease in no formation, both of which are demonstrated to induce the

    在內毒素休克過程中vec是lps及其誘導機體產生的多種促炎細胞因子如tnf 、 il - 1作用的主要靶細胞， vec誘導型一氧化氮合酶（ induciblenitricoxidesynthase ， inos ）激活、 no大量誘生而內皮型一氧化氮合酶（ endothelialnitricoxidesynthase ， enos ）活性被抑制、 no生成障礙，是血管反應性異常變化、血管調節機制紊亂的重要發病環節。
  7. This finding may bring a hope to phytoremediate as - contaminated soils. the effects of as and ca on the growth of p. vittata l. and as uptake by p. vittata l. were studied under sand culture condition. the interaction of as and ca on concentration and distribution of metals including k, mg, mn, fe, zn and cu in it were also investigated

    通過砂培盆栽試驗，本論文從砷和鈣的作用對蜈蚣草生長和累積砷的影響及砷、鈣與其他必需金屬元素的關系等方面研究了蜈蚣草的營養特性，從砷、鈣、磷的亞細胞定位探討了蜈蚣草對砷的解毒機理及鈣的作用機制。
  8. The progress of study on cell mechanism of lead neurotoxicity

    鉛神經毒性細胞機制的研究進展
  9. The main mechanisms of earthworms ' resistance to metal pollutants are also elaborated : ( 1 ) its lipid antioxidative enzyme system helps relieve the stress of oxidation ; ( 2 ) compartment and immobilization of metals ; ( 3 ) process of chelating and detoxicification ; ( 4 ) lysosome and cellular plasmid are activated to restrain activity of heavy metals

    並闡述了蚯蚓對重金屬的主要耐性機制： （ 1 ）脂質過氧化保護酶系統緩解氧化脅迫； （ 2 ）分隔、固定作用； （ 3 ）螯合解毒作用； （ 4 ）溶酶體和細胞質粒抑制重金屬活性。
  10. Taken together, a possible mechanism for sa improving al - tolerance of c. tora is proposed : treatments with sa promoted the al - induced increase in citrate efflux, and this resulted in the decreased accumulation of al in root tips, thereby causing a limited oxidative stress and root growth inhibition

    Sa緩解c . tora鋁毒害的可能機制：增加檸檬酸的分泌，降低根尖細胞內al的含量，減輕鋁誘導的氧化損傷，從而增強c . tora對鋁毒的抗性。
  11. In order to understand the mechanism of mtx further and to investigate the genotoxic target organs, we studied the dna damage and the correlation with dose of mtx by using the alkaline single cell gel electrophoresis ( comet ) assay. liver, spleen, bone marrow, thymus, kidney, testicle, stomach and peripheral lymphocytes of mice were isolated at lh, 3h, 6h, 12h, 24h after 5mg / kg mtx intraperitoneal injection

    為了進一步了解甲氨蝶呤（ mtx ）的作用機制，探測其作用的遺傳毒性靶器官，為應用mtx治療過程中的臨床監測和副作用防治提供理論依據，我們以小鼠為研究對象，用單細胞凝膠電泳技術檢測了mtx腹腔注射染毒1h 、 3h 、 6h 、 12h 、 24h后對肝、脾、骨髓、胸腺、腎、睪丸、胃和外周血淋巴細胞的dna損傷作用及損傷程度與mtx劑量間的關系。
  12. Honokiol, as a major active constituent extracted from the bark of magnolia officianlis, can inhibit tumor growth through cytotoxic activity, inducing differentiation and apoptosis, inhibiting vessles, synthesis of enzyme, dna, rna and ( or ) protein

    和厚樸酚作為木蘭科植物厚樸的主要活性成分，通過細胞毒作用，抑制新生血管，誘導細胞分化，誘導細胞凋亡，抑制酶的合成以及抑制dna ， rna和（或）蛋白質的合成等機制發揮了抗腫瘤的作用。
  13. Cationic antimicrobial peptides ( cap ) are becoming the focus of people owing to its special cytotoxic mechanism

    正電性抗菌肽（ cationicantimicrobialpeptides ， cap ）由於其獨特的細胞毒機制而日益引起人們注意。
  14. Al toxicity - induced ultraweak luminescence ( ul ), sister - chromatid exchanges ( sces ), programmed cell death ( pcd ) in root tips of barley, al - resistant genetic engineering and the role of root border cells in al toxicity were studied respectively, in order to explore some new pathways for al toxicity, al - resistant mechanism and genetic improvement

    大麥是鋁毒極為敏感的作物，本實驗以大麥為材料，開展了鋁毒誘導的根尖細胞超微弱發光、姐妹染色單體交換（ sce ） 、細胞程序性死亡、耐鋁基因工程及邊緣細胞在鋁毒中的功能研究。試圖為植物鋁毒及耐鋁機制和遺傳改良研究探索新途徑。
  15. Because grow, factor is secrete by normal cell, do not have medicaments already kind noxiousness, also do not have immune reaction, because this is studying mechanism of its physiology action while, some already try out at clinical cure

    由於生長因子是由正常細胞分泌，既無藥物類毒性，也無免疫反應，因此在研究其生理作用機制的同時，有的已試用於臨床治療。
  16. The main cytotoxicity and mechanisms of fumonisin were also discussed

    探討了伏馬菌素主要的細胞毒性作用及機制。
  17. The mechanisms of fumonisin - induced toxicity have not been clarified, human and animal cell lines can been used extensively to study its toxicity

    摘要伏馬菌素毒性作用機制尚未研究清楚，目前採用了廣泛的細胞試驗進行研究。
  18. In addit ion, effect of so2 inhalation on the serum cytokine of peripheral blood was determined through the methods of molecular immunology. these studies further indicated that oxidative stress maybe plays an important role in the mechanisms of sulfur dioxide toxicity

    另外還從分子免疫學角度研究了二氧化硫吸入后小鼠血清中細胞因子的變化，上述研究結果進一步明確了氧化應激很可能是二氧化硫的重要毒性作用機制。
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