細胞的突變 的英文怎麼說
中文拼音 [xìbāodetūbiàn]
細胞的突變
英文
mutation of cells- 細 : 形容詞1 (條狀物橫剖面小) thin; slender 2 (顆粒小) in small particles; fine 3 (音量小) thin ...
- 胞 : Ⅰ名詞1 (胞衣) afterbirth2 (同一個國家或民族的人) fellow countryman; compatriot Ⅱ形容詞(同胞...
- 的 : 4次方是 The fourth power of 2 is direction
- 突 : Ⅰ動詞1 (猛沖) dash forward; shoot out 2 (高於周圍) protrude; bulgeⅡ副詞(突然) abruptly; sud...
- 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
- 突變 : 1 (突然急劇的變化) sudden change; change suddenly; transilience; accident; saltation; revulsion...
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The bacilliform cell penetrate into interior of the fibre to degrade the cellulose strongly and produced a mass of sticky polysaccharides. after cultured 48 hours, the bacilliform cell ' s surface of sporocytophaga have a great change. at this stage the bacilliform produce a lot of sticky polysaccharides. these sticky polysaccharides associated with the sites where the filter paper was decomposed intensively and form thorns on the surface of the bacillium. at the same time, the filter - paper weight loss is the greatest and decomposing rate is the fastest, so we think that the sticky polysaccharides are produced during the cellulose degradation
培養48小時,桿狀細胞的表面結構發生很大的變化,此時的菌體表面已產生大量的粘性多糖,這些粘性多糖因菌體在纖維素表面滑動而在菌體表面形成突起,即在纖維素被旺盛降解部位的菌體表面產生了大量突起;而產生突起的菌體深入到纖維素分子內部,纖維素表面可以清晰地看到由於菌體嵌入纖維素分子內部而留下的凹陷。So the researchers turned to mutant mice with cells that lacked cilia
因此,研究者轉向研究缺乏纖毛的突變異種老鼠的細胞。The dna damage caused by so : was paid more attention to in recent years. based on the early work, we studied the effects of so2 in the following aspects : the first part, damage effects of so2 derivatives injection on dna of cells in mice sulfite and hydrosulfite are two kinds of so : derivatives in vivo
近年來, so _ 2引起機體dna損傷而導致哺乳動物組織細胞dna突變日益引起了人們的關注,為此,我們在前人工作的基礎上,主要進行了以下幾方面的研究:一、 so _ 2衍生物對小鼠胃、肝、脾和腎細胞dna的損傷作用。Impatient and because these females are extremely easy generation insecurity angst, alone depression, sad sadness, dejected disappointment, angry wait for depressed mood, suffer undesirable mood for a long time to perverse stimulate, law of airframe life division happens disorder, function of nerve internal system is maladjusted, bring about core condition unbalance then, immune force drops, can make thymus is generated decrease with the thymus element that release, the monitoring capability to cell of the mutation inside body mixes cell of lymphocyte, huge bite gobble up ability to drop, incidental cancer is swollen
由於這些女性極易產生緊張焦慮、孤獨壓抑、悲哀憂傷、苦悶失望、急躁惱怒等抑鬱情緒,長期受不良情緒剌激,機體生命節律發生紊亂,神經內分泌系統功能失調,進而導致內環境失衡,免疫力下降,可使胸腺生成和釋放的胸腺素減少,淋巴細胞、巨噬細胞對體內突變細胞的監控能力和吞噬能力下降,輕易發生癌腫。What genetic change allowed multicellular animals to come into existence remains obscure
什麼樣的遺傳突變產生了多細胞的動物還不清楚。Whole cell c2d2 reduction by all four mutants comparing to wild type and ni / v mutant was also detected. the result showed that only single a - gln194 substitution did not perturb the stereospecificity of protonation of c2d2. the above comparing results indicate that in mofe protein ( 1 ) a a - gln190 site and its association with homocitrate are important for the transfer of electron / proton to femoco, while a - his194 site and the homocitrate are independent in h2 evolution
對四個突變株細胞的c _ 2d _ 2還原特性及還原產物中反式-順式- 1 , 2 -二氘代乙炔的比例進行了測定並與野生型及nifv突變株相比較,結果表明只有- gln ~ ( 194 )替換不影響c _ 2d _ 2還原產物中反式-順式- 1 , 2 -二氘代乙炔的比例,即未改變固氮酶還原c _ 2h _ 2加氫的立體構型的專一性。The seven sisters that dr de grey wishes to slaughter with sens are cell loss, apoptosis - resistance ( the tendency of cells to refuse to die when they are supposed to ), gene mutations in the cell nucleus, gene mutations in the mitochondria ( the cell ' s power - packs ), the accumulation of junk inside cells, the accumulation of junk outside cells and the accumulation of inappropriate chemical links in the material that supports cells
德?戈瑞博士想用這一策略對付的七個因素是:細胞丟失,細胞凋亡阻抑(細胞拒絕程序死亡的傾向) ,細胞核內的基因突變,線粒體(細胞的「動力倉」 )中的基因突變,細胞內的垃圾堆積,細胞外的垃圾堆積和細胞支持性物質中不當化學鍵堆積。Our previous work identified delayed mutation occurred at target supf trna gene in plasmid transfected into cells pretreated with low concentration of alkylating agent n - methyl - n ' - nitro - n - nitrosoguanidine ( mnng )
在收回的質粒上我們檢測到了延遲發生的、高於對照5倍以上的突變。由於w在細胞中的半壽期僅為1This is minimal change disease ( mcd ) which is characterized by effacement of the epithelial cell ( podocyte ) foot processes and loss of the normal charge barrier such that albumin selectively leaks out and proteinuria ensues
微小病變性腎小球腎炎,特點是:臟層上皮細胞(足細胞)的足突消失,正常的離子屏障喪失,表現為選擇性白蛋白漏出,接著出現蛋白尿。It has been reported that the eiav s2 is not essential and does not appear to affect virus infection and replication in vitro. thus, we introduced a his - tag into the s2 gene of an eiav infectious molecular clone recombinant plasmid ( pok8266 ) by using soe pcr method and obtained a new recombinant plasmid with his - tag, designated as eiav - pok8. 2 - his
本研究應用已構建好的eiav驢白細胞弱毒疫苗株的感染性分子克隆載體( pok8266 )為模板,通過soepcr方法在感染性分子克隆載體的s2基因獨特區內引入突變點,形成含有酶切位點( nspv )的突變體( p1p4 ) 。In our laboratory, a unique mutation detection system using a shuttle vector plasmid has been established to demonstrate that a low concentration of mnng ( 0. 2 m ) can induce nontargeted mutation in mammalian cells : the mammalian cells were exposed to 0. 2m mnng for 2. 5h, then a shuttle plasmid pz189 carrying supf trna gene was transfected into cells after 24h culture. we found a 5 - fold higher mutation frequency of the plasmid replicated in pretreated cells than the spontaneous mutation frequency of the plasmid replicated in control cells. this kind of mutation did not occur immediately after mnng exposure
我們實驗室曾用一特殊的突變檢測系統,直接證明dna損傷劑可在哺乳動物細胞誘發非定標性突變:首先用低濃度( 0 . 2 m )的短壽烷化劑mnng (半壽期為1 . 1hr )處理細胞2 . 5h后,繼續培養24h ,將重組有用作突變檢測的靶基因supftrna基因的穿梭質粒pz189轉入細胞復制,發現在未受致癌物直接攻擊的穿梭質粒中有較自發突變率高5倍以上的靶基因突變。Mouse bone marrow cell mutation induced by vechile exhaust
汽車尾氣對小鼠骨髓細胞的致突變性The mutant protein of huntington ' s attacks the railroad system of brain cells and impairs transport of essential materials required for neurons to function
突變的亨廷頓蛋白攻擊腦內細胞的轉運系統,繼而損害了神經元要發揮正常功能所必需的物質的轉運。According to antibody - antibody ab - ab affinity and antibody - antigen ab - ag affinity, the algorithm can allot adaptively the scales of memory unit and antibody population. it is proved theoretically that the csaim is convergent with probability 1
由於遺傳和免疫細胞在增殖中的基因突變,形成了免疫細胞的多樣性,這些細胞的不斷增殖形成無性繁殖系,無性繁殖稱為克隆。In recent years, studies indicated these molecular markers mainly involved : oncogenes and tumor - suppressor genes mutate or rearrange in some thyroid neoplasms ; the exceptional increasing on the activity of telomerase ; the special proteins which are related to cell cycle regulation or adhesion express excessively ; as well as some special matter which is discovered recently
近年研究表明這些分子標志物主要涉及癌基因或抑癌基因的突變或重排;端粒酶活性的異常增高;一些與細胞周期調控或與粘附有關的特殊蛋白的表達;以及一些新發現的特異物質。The sections were observed under microscope of olympus new vanox. ana preparation of samples for sem observation : anaesthesia and perfusion and fixation procedures were same as that mentioned a
3個6w ,突起長度540w 。 )室管膜細胞的加齡變化:在In the first large - scale screen of genetic changes in cancer cells, researchers have found that a typical breast or colorectal tumor results from mutations in about 90 genes, with different sets of mutations producing the same type of cancer
在首次癌細胞遺傳改變的大規模篩選中,研究者發現典型乳腺或者結直腸腫瘤源於約90種基因突變,不同的突變組合產生同種類型的癌癥。Metal masteries, for example hg, ag, pb, cd, as, cr, ni, are harmful to dna, rna, enzyme, protein and cell membrane and will cause the taking place of cancers and mutations of them. so the normal metabolize and split are damaged, the immunity function, the anti - cancer function will be weakened. it is very difficult to be cured once he or she is toxic
金屬汞、銀、鉛、鎘、砷、鉻、鎳等對dna 、 rna 、酶、蛋白質以及細胞膜的損傷非常大,從而引起它們的致癌、致突變作用,以破壞細胞的正常代謝和分裂;降低機體的免疫功能、防癌和抗癌能力,一旦中毒是很難治愈的。The sickle cell mutation apparently arose repeatedly in regions riddled with malaria in africa and the middle east
鐮狀細胞的突變在瘧疾肆虐的非洲和中東等區域,顯然曾重復發生過多次。However, dna damage has been ruled out as the prerequisite for alkylating agents - induced mutagenesis. for example, somatic hypermutation, which occurred around the variable region in the immunoglobin gene of b cells, is driven by antigen activation not by dna damage and is considered a kind of active mutation
但是dna損傷並不是引起突變的必要條件,一個典型的例子就是發生在b淋巴細胞的免疫球蛋白( ig )可變區基因上的「體細胞超突變( somatichyermutation ) 」 ,是由表面抗原受體而不是dna損傷驅動的主動突變。分享友人