缺氧時間 的英文怎麼說

中文拼音 [quēyǎngshíjiān]
缺氧時間 英文
hypoxia time
  • 缺 : Ⅰ動詞1 （缺乏; 短少） be short of; lack 2 （殘缺） be missing; be incomplete 3 （該到而未到） be ...
  • 氧 : 名詞[化學] （氣體元素） oxygen （o）
  • 時 : shí］Ⅰ名1 （比較長的一段時間）time; times; days:當時at that time; in those days; 古時 ancient tim...
  • 間 : 間Ⅰ名詞1 （中間） between; among 2 （一定的空間或時間里） with a definite time or space 3 （一間...
  • 缺氧 : anoxia; hypoxia; oxygen deficit; oxygen lack缺氧代謝 anaerobic metabolism; 缺氧環境 oxygen defici...
  • 時間 : time; hour; 北京時間十九點整19 hours beijing time; 上課時間school hours; 時間與空間 time and spac...
  1. The annihilation of the octahedron voids at the tips of fpds was divided two processes : ( 1 ) the oxide on the void was removed by the out - diffusion of oi in the shallow region, especially the oi aroud the void and by the entry of the interstitial si atomics. ( 2 ) the void without oxide shrinked by emitting vacances and the migration of silicon atoms from edge to the bottom of void

    Fpds端部八面體空洞的消失分為兩個階段： （一）覆蓋在空洞各個內壁上的氧化膜由於高溫下矽片表面區域的間隙氧原子，尤其是空洞型缺陷周圍的間隙氧原子的外擴散及自間隙硅原子的進入，而逐漸變薄直至最終消失。 （二）無氧化膜的空洞，在高溫下發出一個個空位，同時八面體空洞周圍的自間隙硅原子不斷的從空洞的邊緣遷移至空洞的底部，使空洞逐漸變淺直至最後消失。
  2. The dependence of oxygen precipitation and induced - defects in heavily as - doped silicon on heat treatment process was studied by annealing and ig process, chemical etching, scanning electron micrograph ( sem ) and transmission electron microscopy ( tem ). a developed ig technique was suggested and the mechanism of the influence of as on oxygen precipitation formation in heavily as - doped silicon was discussed

    本文通過化學腐蝕、光學顯微鏡、掃描電鏡（ sem ） 、透射電境（ tem ）等分析技術，對重摻砷硅單晶在單步退火工藝和內吸雜退火工藝中氧沉澱及誘生缺陷的形態，形核與熱處理溫度、時間的關系等進行了研究。
  3. The result showed that crystal defect of nano - cuo prepared by pressure - hydrothermal method is less, structure is more stable, the process of charge and discharge is more steady, and duration is longer

    結果表明，壓力-熱液法制備的納米氧化銅晶格缺陷少，結構穩定，充放電過程平穩，且持續時間長。
  4. Method : dividing 90 kunming mice to 3 groups, observing the index of weight loading swimming test, bearing hypoxia at normal pressure, activity of lactic dehydrogenase

    方法: 90隻雄性昆明種小鼠分為3組，觀察艾灸后小鼠的負重遊泳試驗、常壓耐缺氧時間、乳酸脫氫酶活力等指標。
  5. Result : moxibustion had the function of reinforcing weight loading swimming ability and activity of lactic dehydrogenase and lengthening the time of bearing hypoxia at normal pressure

    結果:艾灸能有效地提高小鼠的負重遊泳能力和常壓耐缺氧的時間，並可增強乳酸脫氫酶的活力。
  6. Lying in an intensive - care ward is a world away from climbing everest, but a connection will be drawn this spring when 45 scientists and 208 volunteers tackle the mountain to bring back information about oxygen deprivation

    躺在搶救室里和攀登珠穆朗瑪峰原本毫不相干，但今年春天，它們之間的聯系將會在45位科學家和208位志願者為了獲取關于缺氧的信息而挑戰這座高峰的時候建立起來。
  7. After the coke being puted into the coke - quenching device, the lid of the device should be shut at once. while the suction pump, refrigeration compressor, high pressured water circuling pump are started, the subpressure will be produced by the burning coke. then, in the vacuum, the coke will be extinguished in a flash. the burning of the coke instantly, still, produces a large quantity of heat which can be taken away by circulating water in refriger and the interlayer, after that, the temperature will fall to 60oc, even lower. in this condition, the coke will be pushed out to be stored, thus, the coke won ' t rekindle

    當焦炭推上攔焦車進入熄焦器后，立即關閉罐蓋，同時啟動真空泵、冷凍機、高壓循環水泵，這時，燃燒著的焦炭就會因真空泵把熄焦器內抽成負壓，由於缺氧，使焦炭熄滅，雖然煤炭在瞬間被熄滅，但由於焦炭生成過程中所產生的大量的熱量還存在，這時，冷凍機和夾層中的循環水的工作就會把焦炭中的熱量帶走，使之在短時間內將溫度降到60以下，在這種狀態下把焦炭從熄焦器內推出入庫，焦炭就不會復燃。
  8. Some of these problems, similar to those of living in outer space, are pressure, lack of oxygen and weightlessness.

    其中有一些問題就象在外層空間生活時會遇到的問題一樣，是壓力、缺氧和失重。
  9. Ethanol is a product of anaerobic respiration in plants, and waterlogged roots suffer from a shortage of oxygen

    乙醇是植物無氧呼吸的產物，水淹的植物根系受到短時間的缺氧。
  10. Effect of hyperlipoproteinemia on time of useful consciousness during hypoxia in pilots

    高脂蛋白血癥對飛行人員缺氧有效意識時間的影響
  11. Using this system, we have studied matrine - inhibittory effect and trifluoperation - neuroprotection effect in hippocampal slices, also discussed the mechanism of long - term potentiation using anesthetic rats. the experiment results showed that matrine can inhibit the hyperactivity induced by penicillin sodium in dosage by changing the relative parameters of field potential ; trifluoperation can alter ps change with the time, enhance the degree and the ratio of ps recovery, then minis the hypoxic injury ; high frequency stimulate can increase ps amplitude and epsp slope for long time, buildup the in / out function of nerve cells, and enhance synaptic plasticity

    結果表明，苦參堿能夠劑量依賴性地抑制青霉素誘導的神經元順向信號傳導激活過程，使細胞外記錄到的場電位各個參數發生相應改變；三氟拉嗪可以改變ps的時相變化，提高ps的恢復程度和恢復率，減小了神經元因缺氧引起的不可逆損傷；高頻刺激（ highfrequencystimulate ， hfs ）可以長時間的增強ps的幅度和epsp的斜率，進而增強神經元的輸入輸出功能，增加了突觸的可塑性。
  12. The second part of the thesis includes a derivation of reduced order models for the activated sludge process. firstly, the paper introduces the derivation of time - varying linear state - space models for the anoxic part and the aerobic part. secondly, by integrating the anoxic part model with the aerobic part model, we can get the resulting models - a time - varying bilinear state - space model for the activated sludge prenitrification system

    為了控制應用的需要，本文首先分別建立了缺氧池和好氧池的時變線性狀態空間模型，然後把好氧池和缺氧池的時變線性狀態空間模型結合起來得到由缺氧和好氧兩部份構成的前硝化活性污泥系統的時變雙線性模型，最後簡單描述了理想狀態下沉澱池的模型。
  13. After annealing at 600, because of formation of multi - vacancy - type defects that have long positron lifetime, positron annihilation average lifetime increased. when the average positron lifetime increased to maximum value ( 360ps ), the interstitial oxygen concentration decreased to minimum value ( 4 1017atoms / cm3 ). this result suggested that oxygen was involved in the formation of multi - vacancy - type defects

    正電子湮沒技術測試證明，快中子輻照直拉硅中在大約600退火時產生的多空位缺陷具有較長正電子壽命，可以使正電子平均壽命增加，當樣品的正電子平均壽命達到最大時（ 360ps ） ，其間隙氧含量降到一個極小值（ 4 10 ~ （ 17 ） atoms / cm ~ 3 ） ，這說明氧參與了這些缺陷的形成。
  14. We found that with the time prolongation of ogd, ttc staining of the hippocampal slices was reduced and ldh release rate was increased consistently. the percent decrease of ttc staining was significantly correlated ( r = - 0. 933 ) to the percent increase of ldh release due to ogd insults

    結果發現，隨著缺氧缺糖時間的延長腦片hc染色逐漸下降， ldh釋放率也逐漸增高， ttc染色下降百分率和ldh釋放百分率呈顯著負相關（ r 。
  15. Optimal time window selection in hyperbaric oxygen treatment for rat transient global cerebral ischemia on the basis of neuron - specific enolase bioactivity changes

    高壓氧治療大鼠短暫全腦缺血的最佳時間窗選擇以神經元特異性烯醇化酶活性變化為標準
  16. To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation

    為了驗證上述假設，本文分別在整體和離體水平，採用大腦中動脈缺血（ middlecerebralarteryocclusion ， mcao ）和體外氧?糖剝奪（ hypoxiaandglucose - deprivation ， hgd ）缺血模型，首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況，觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化，然後通過外源性給予vegf蛋白，在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響，最後利用vegf反義脫氧寡核苷酸（ oligodeoxynucleotide ， odn ）特異阻斷內源性vegf蛋白的表達，觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用，以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。
  17. Channel gating, ion selectivity, or ionic conductance and regulatory mechanisms of the ion channels on cell membranes. besides, we pay attention to the effect of intermittent and sustained hypoxia to the movement of ions. we also try to simulate an inflammatory environment to survey the changes in infection and trauma

    其中生理部分，在於研究細胞膜表面各種離子孔道的特性如離子孔道啟閉離子選擇性或離子通透度等和調控機制，同時並著重於間歇性與持續性缺氧的影響至於病理部分，則模擬感染或外傷的環境來觀察對離子運輸的改變。
  18. Of the 29 red tide incidents recorded this year, all but one were formed by non - toxic species. as all these red tides receded within a short time, they had not caused any serious oxygen depletion in seawater and thus should not have caused significant impact on the marine resources and ecology

    （二）在今年錄得的二十九宗紅潮中，除了一宗是由有毒品種組成外，其餘均為無毒品種，而所有紅潮亦在短時間內消退，並沒有引致海水出現嚴重缺氧的情況，相信不會對海洋資源及環境生態造成大的影響。
  19. Abstract : the saturation behavior of stress current is studied. the three types of precursor sites for trap generation are also introduced by fitting method based on first order rate equation. a further investigation by statistics experiments shows that there are definite relationships among time constant of trap generation, the time - to - breakdown, and stress voltage. it also means that the time constant of trap generation can be used to predict oxide lifetime. this method is faster for tddb study compared with usual breakdown experiments

    文摘:基於一階速率方程，討論了恆定電壓應力下應力電流的飽和行為.通過對應力電流的擬合，發現存在三類缺陷產生的前身.更進一步的統計實驗顯示，在缺陷產生時間常數、擊穿時間以及應力電壓之間存在著明確的關系.這意味著缺陷產生時間常數能夠被用於有效預測氧化層的壽命.與常規的氧化層擊穿實驗相比，基於缺陷產生時間常數的預測更快、更有效
  20. Thirdly, this paper compared the different responses of plants to these two stresses, the change of the defensive enzyme activity and the rate of losing water of leaves was different at two different stresses. the rate of losing water of leaves at drought stress was more rapid than that at salt stress, the leaves would wither and the change of activity of enzymes notably at drought stress, these results showed that the damage to plants was result from the oxidation at drought stress. at salt stress, the content of relative water of leaves changed slightly as well as did the activity of enzymes, these results showed that the damage to plants was caused by poison of ion and lack of nutrition

    三、通過研究兩種冰草植物對兩種脅迫的反應指出乾旱脅迫和鹽脅迫通過不同的途徑對植物進行傷害，主要表現在葉片的失水速率和保護酶活性的變化，乾旱脅迫短時間內葉片失水速率很快，葉片萎蔫，引起保護酶活性顯著變化，表明乾旱脅迫主要是通過氧化脅迫對植株造成傷害，而鹽脅迫下葉片相對含水量幾乎保持不變，表明鹽脅迫對植物的傷害主要通過離子毒害和營養虧缺造成。
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