肝組織結構 的英文怎麼說

中文拼音 [gānzǔzhījiēgòu]
肝組織結構 英文
liver structure
  • 肝 : 名詞（人和高等動物的消化器官之一） liver
  • 組 : Ⅰ名詞1 （由不多的人員組成的單位） group 2 （姓氏） a surname Ⅱ動詞（組織） organize; form Ⅲ量詞（...
  • 織 : 動詞（編織） knit; weave
  • 結 : 結動詞（長出果實或種子） bear （fruit）; form （seed）
  • 構 : Ⅰ動詞1 （構造; 組合） construct; form; compose 2 （結成） fabricate; make up 3 （建造; 架屋） bui...
  • 組織 : 1 （組織系統） organization; organized system 2 （組成） organize; form 3 [紡織] weave 4 [醫學] [...
  • 結構 : 1 （各組成部分的搭配形式） structure; composition; construction; formation; constitution; fabric;...
  1. The pathology characteristic that liver fiber changes is collect there are many fibre hyperplasia and deposit inside abbacy and liver flocculus, but have not form the interval inside flocculus, liver cirrhosis has false flocculus to form, central vein area and collect abbacy occurrence interval, the normal structure of liver is destroyed, liver fiber changes further progress to be liver cirrhosis namely

    肝纖維化的病理特點為匯管區和肝小葉內有大量纖維組織增生和沉積，但尚未形成小葉內間隔，肝硬化則有假小葉形成，中心靜脈區和匯管區出現間隔，肝的正常結構遭到破壞，肝纖維化進一步發展即為肝硬化。
  2. Using cd, cu, zn and cr as experimental toxicants and crucian as experimental creature, we employed the environmental biotechniques ( flame atomic absorption spectrophotometry, the method of improved pyrogallol autoxidation and electron microscopy ) to study the acute toxicity and secure concentrations of heavy metals to crucian ; the accumulation and distribution of mixed heavy metals to fingling crucian ; the activities of superoxide dismutase ( sod ) of gill and liver tissues and the hispathological and ultrastructural change of superfine structure of liver and kidney of crucian after cadmium exposure. through which, we intended to fully and systemically study the toxic influence of heavy metals to fish, and preliminarily discuss the influence of heavy metals pollution to the diversified level of fish such as individual, organ, tissue, cell and molecule. thus we tried to provide scientific evidence to establish preventative management measures, avoid and relieve the harm of heavy metals pollution to aquicolous ecosystem in time

    隨后以這幾種重金屬作為實驗毒物，以鯽魚為實驗動物，應用環境生物技術、火焰原子吸收分光光度法、鄰苯三酚自氧化法、透射電鏡等技術研究了重金屬cu 、 zn 、 cd和cr對鯽魚的急性毒性及其安全濃度評價、混合重金屬在鯽魚幼體組織內的積累和分佈、 cd對鯽魚鰓和肝臟中sod活性的影響、 cd對鯽魚肝細胞和腎細胞超微結構的影響等，全面和系統地研究了水環境中重金屬暴露對魚類的毒性影響，初步探討了重金屬污染對魚類的個體? ?器官? ?組織? ?細胞? ?分子等各水平層次的影響，為制定漁業生產上預防性的管理措施提供科學依據，及時避免或減輕重金屬污染對水生生態系統造成的損害。
  3. We found nuclear deformation lymphocytes with increased heterochromatin and impaired thymus epithelium cells with increased lysosomes and deformation of mitochondrias. ( 4 ) effects on mouse liver of so2 challenge : so2 can cause significant liver injury. he staining showed several kinds of necrosis of liver including spot necrosis, focal necrosis and submassive necrosis infiltrated with lymphocytes, monocytes, few neutrophils and eosinophils ; tem observation showed fatty degeneration with dispersion of fatty droplets and dilation of rough endoplasmic reticulums, acid degeneration with significant hyperplasia of mitochondrias, necrosis of hepatocytes with karyorrhexis and other organelles losing their normal structure

    （ 4 ）二氧化硫染毒對小鼠肝臟的組織學結構有明顯影響，可引起肝臟點狀壞死、灶狀壞死甚至片狀壞死，伴隨不同程度的炎性細胞浸潤；透射電鏡觀察發現二氧化硫可引起肝細胞脂肪變性、嗜酸性顆粒變和壞死，脂肪變肝細胞中可見大小不等的脂滴存在，嗜酸性顆粒變肝細胞中可見線粒體明顯增生，壞死肝細胞可見細胞核結構破壞，細胞器減少，細胞膜不完整。
  4. The main study in this paper included as follows : the content and distribution of heavy metals in sediments and benthic organisms from the sewage stream in guangzhou city ; the acute toxicity and joint toxicity of mercury and selenium to swordtail fish ( xiphophorus helleri ) ; the damage of mercury to the indexes of antioxidant system in the gills and livers in swordtail ( including the measurement of the activities of total antioxidative capacity [ t ~ aoc ], superoxide dismutase [ sod ], glutathione peroxidase [ gsh - px ] and the concentration of malondiald - ehyde [ mda ] ) and the relief effects of selenium on it, as well as the physiological damage of mercury on the tissues, namely : the antagonistic effect of na + - k + ~ atpase activity on the tissues between selenite and mercury, and the ultrastructural damage under the exposure of mercury

    研究內容主要有：廣州市河涌沉積物及底棲生物體內重金屬含量及評價；汞和硒對劍尾魚的急性毒性和聯合毒性及安全濃度的評價；汞對劍尾魚鰓和肝臟中抗氧化系統的毒性，包括對總抗氧化能力、超氧化物歧化酶、谷胱甘肽過氧化物酶活力及丙二醛含量的測定及硒對其保護作用；汞對劍尾魚組織生理毒性即：汞對na ~ + - k ~ + atpase活力的影響及硒的保護作用和汞和對劍尾魚組織超微結構的損傷等。以高氯酸?硝酸消化法和火焰原子吸收分光光度法測定了廣州市河涌沉積物和底棲生物中重金屬含量。
  5. Investigation on histopathology and ultrastructure damage of rat liver and kidney induced by mini - dose t - 2 toxin

    2毒素致大鼠肝腎損傷的組織病理及超微結構觀察
  6. Results in the 260 slices of liver biopsy, there were 20 ones of acute viral hepatitis, 78 ones of chronic hepatitis, 79 ones of hepatocirrhosis, 28 ones of primary liver cell cancer, 27 ones of liver karyon heterogeneity, 20 ones of liver metastatic adenocarcinoma, 12 ones of fatty liver, five ones of alcohol liver, three ones of liver cyst, two ones of congenital bile duct atresia, six ones of illegible structure, 20 ones without liver cell or with few scattered liver cell

    結果260例肝穿活檢組織病例中，急性（病毒性）肝炎20例；慢性肝炎78例；肝硬化39例；原發性肝細胞癌28例；肝細胞核異質27例；肝轉移性腺癌20例；脂肪肝12例；乙醇肝5例；肝囊腫3例；先天性膽管閉鎖2例；穿刺組織結構模糊6例；未穿到或僅穿到少量肝細胞20例。
  7. The hepatic adenoma is on the right and is composed of cells that closely resemble normal hepatocytes, but the neoplastic liver tissue is disorganized hepatocyte cords and does not contain a normal lobular architecture

    右側為肝臟腺瘤，腫瘤細胞類似於正常肝細胞，但腫瘤組織中肝細胞條索排列紊亂，無正常肝小葉結構。
  8. Hla - g1, which is a newly defined non - classical hla class i molecule, plays an important role in mediating immunotolerance and protecting embryo and even some kinds of tumors from nk cells attacking. the full - length coding sequences containing cdna of hla - g1 were cloned from placenta, monocytes and liver cancer tissue of chinese donors. sequence analysis reveals that it is a highly conserved human gene with only two amino acid mutation sites compared to foreign nationality. its truncated form was overexpressed in

    從中國人外周血單個核細胞胎盤組織和肝癌組織等樣品中克隆了包含完整hla - g1讀框的cdna與國外同行獲得的該基因及其蛋白質序列比較分析表明，該基因雖然有著細微的種族特異性，但高度保守並獲得了它的截斷型重組蛋白，根據蛋白一級結構和同源比較方法，模建了它及其與特異性受體kir2dl4形成復合體的空間結構模擬，預測了它們之間相互作用的特徵。
  9. Ferrannini, lanfranchi a, rohner jeanrenaud f, et al. influcence of longterm diabetes on liver glycogen metabclism in the rat [ j ]. metabolism. 1990, 39 ( 10 ) : 1082

    賈雪梅，齊易祥，王惠珠，等.實驗性糖尿病對小鼠肝臟酶組織化學和超微結構的影響[ j ] .解剖學雜志， 1995 ， 18 （ 5 ） : 451
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