肺泡上皮細胞 的英文怎麼說

中文拼音 [fèipāoshàngpíxìbāo]
肺泡上皮細胞 英文
alveolar epithelial cells
  • 肺 : 名詞（人和高等動物的呼吸器官） lungs
  • 泡 : 泡Ⅰ名詞1 （氣泡） bubble 2 （像泡一樣的東西） sth shaped like a bubble Ⅱ動詞1 （較長時間地放在液...
  • 上 : 上名詞[語言學] （指上聲） falling-rising tone
  • 皮 : Ⅰ名詞1 （人或物體表面的一層組織） skin 2 （皮革） leather; hide 3 （毛皮） fur 4 （包在外面的一層...
  • 細 : 形容詞1 （條狀物橫剖面小） thin; slender 2 （顆粒小） in small particles; fine 3 （音量小） thin ...
  • 胞 : Ⅰ名詞1 （胞衣） afterbirth2 （同一個國家或民族的人） fellow countryman; compatriot Ⅱ形容詞（同胞...
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  1. 1 sassler cn, bloomfield gl, fowler aa. current concepts of sepsis and acute lung injury. clinics in chest medicine 1996 ； 17 ( 2 ) : 213 - 235

    2潘芳，李文志.肺泡型上皮細胞的體外培養及其在麻醉學研究領域的應用.國外醫學麻醉學與復甦分冊2000 ； 21 （ 6 ） 368 - 371
  2. 3. lh positive cells were appeared in 18 - day fetuses and rest in the epithelium of bronchioles and the primitive alveoli

    Lh陽性細胞最早在胚18日胎肺組織內出現，位於肺內支氣管上皮和原始肺泡壁內。
  3. Results the major morphologic changes were as follows : histologically, alveolar inflammation and interstitial fibrosis were observed. electron microscopic findings were : 1. alveolar type i cells were degenerated 、 broken - down and desquamated, endothelial cells were swelled, with inter cellular tight junction shortened, alveolar type ii cells hyperplastic, basement membrane thinned and deformed ; 2. alveolar macrophages and interstitial macrophages were hyperplastic ; 3. mast cells were infiltrated and degranulated ; 4. electron - dense deposits were present at alveolar wall ; 5. myofibroblasts 、 fibroblasts 、 collagen and basement membrane like material were hyperplastic

    電鏡觀察可見： （ 1 ） i型肺泡上皮細胞變性、崩解和脫落，內皮細胞腫脹，細胞間緊密連接短小， ii型肺泡上皮細胞增生，基底膜變薄和破壞； （ 2 ）肺泡巨噬細胞、間質巨噬細胞增多； （ 3 ）肥大細胞浸潤並見脫顆粒現象； （ 4 ）肺泡壁電子緻密物沉積； （ 5 ）肌纖維母細胞、纖維母細胞、膠原原纖維及基底膜樣物質增生。
  4. We also investigated the pathological changes of mouse liver, thymus and cerebrum cortex challenged by so2 inhalation by in vivo tests. we studied the apoptotic induction on mouse spleen cells and cytotoxicity of human embryo lung fibroblasts of so2 derivatives by in vitro tests. in vivo tests of sulfur dioxide inhalation showed : ( 1 ) effects on mouse lung of so2 challenge : we found no significant apoptotic changes induced by so2 inhalation but obvious pathological changes of lung with vacuolating of osmiophilic multilamellar bodies which maybe related with the decrease of surfacant and decrease of microvillus of type ii alveolar cells ; we also found thickening of part of basement lamina between type i alveolar cells and capillary endothelium cells which may inhibit the dispersion of oxygen and contribute to lung dysfunction

    二氧化硫熏氣染毒的體內實驗結果表明，在本次實驗的濃度范圍內（ 56mg m ~ 3 、 112mg m ~ 3 、 168mg m ~ 3低、中、高三個濃度） ： （ 1 ）通過透射電鏡、 dna凝膠電泳分析和流式細胞分析發現二氧化硫吸入染毒一周對小鼠肺臟沒有明顯的凋亡誘導作用，但通過透射電鏡觀察發現二氧化硫可引起肺臟明顯的超微結構改變，引起型肺泡上皮細胞板層體空泡化，微絨毛減少，線粒體緻密化或腫脹變性；肺泡血管內皮細胞和型肺泡上皮細胞之間基膜增厚，使氧氣彌散功能出現障礙，從而降低肺功能。
  5. Mechanism of glutamate - induced inhibition of pulmonary surfactant lipid synthesis ? mda receptor ( nmdar ) antagonist mk - 801 could reverse glutamate - induced inhibition of [ 3h ] - choline incorporation in lung explants ; ? he expression of nmdar1 subtype existed in alveolar type ii epithelial cells ; ? he effect of glu on pc synthesis required l - arginine in cultured lung explants, nos activity and no production were increased significantly in lung explants with glu treatment ; and mk - 801 and nos inhibitor l - nna could block the increase of nos activity and no production induced by glu ; l - nna also could reverse the inhibition of [ 3h ] - choline incorporation induced by glu ; ( 4 ) mda content increased and sod level decreased in cultured lung explants of glu treament groups, mk - 801 and l - nna could reverse these effects ; ( 3 ) mk - 801, l - nna could block the release of ldh induced by glu

    谷氨酸對肺表面活性物質脂質合成抑制的機制研究nmda受體拮抗劑mk - 801能逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應；肺泡上皮細胞有nmda受體nmdar1亞單位的表達； glu對ps脂質合成的影響依賴l -精氨酸的存在， glu可使肺組織勻漿中nos活性、 no合成量升高； mk - 801 、 nos的抑制劑l - nna均可阻斷glu所致的這些效應； l - nna可逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應； glu可使肺組織勻漿中mda碩士學位論文中文摘要含量增高、 sod水平下降。 mk七0 、 l nna能阻斷q所致的這些效應； mk80 、 l nna還能阻斷gill誘導的ldh釋放增多的效應。
  6. Conclusions electron microscopic obser vations show that the alveolar epithelial cells, endothelial cells and basement membrane are all injured, interstitial macrophages hyperpplasia, mast cells infiltration and degranulation, electron - dense deposits at alveolar wall are present

    結論電鏡觀察可見特發性肺纖維化時肺泡上皮細胞、血管內皮細胞及基底膜均有損傷，並可見間質巨噬細胞增生，肥大細胞浸潤及脫顆粒，肺泡壁電子緻密物沉積。
  7. This is normal lung microscopically. the alveolar walls are thin and delicate. the alveoli are well - aerated and contain only an occasional pulmonary macrophage ( type ii pneumonocyte )

    正常肺顯微鏡顯示肺泡壁比較薄弱。肺泡內充滿空氣，有肺巨噬細胞（ ii型上皮細胞） 。
  8. Downregulation of gap junctional intercellular communication induced by silicon dioxide in the pulmonary alveolar epithelial cen

    2刺激肺泡上皮細胞間隙連接功能下調的研究
  9. Methods : in cultured lung explants without serum, the lipid component synthesis of pulmonary surfactant was evaluated in [ 3h ] - choline incorporation ; mrna content of phosphocholine cytidylyltransferase ( cct ) in lung explants was investigated in rt - pcr ; the changes of the ultrastructure of the at ii cells were observed with electron microscope ; the expression of nmdar1 subtype was observed in immunohistochemistry staining ; nitric oxide synthase ( nos ) activity, nitric oxide ( no ) content, superoxide dismutase ( sod ) level, malondialdehyde ( mda ) content and lactae dehydroase ( ldh ) level were determined by biochemistry methods. results : 1. influence of glutamate on synthesis of the lipid component of pulmonary surfactant ? with l - arginine, glu inhibited [ 3h ] - choline incorporation with good dose - dependence and time - dependence ; ( 2 ) mrna content of cct of the glu treatment groups was decreased ; ( 3 ) glu increases the release of ldh in cultured lung explants ; ( dwith electron microscope histochemistry, glu induced the changes of the ultrastruture of at ii iv cells

    方法：採用成年大鼠肺組織無血清培養，運用[ ~ 3h ] -膽堿摻入法測定ps主要脂質磷脂酰膽堿（ pc ）合成量； rt - pcr擴增檢測肺組織中pc合成限速酶磷酸膽堿二胞苷酰基轉移酶（ cct ） mrna含量；透射電子顯微鏡法觀察肺泡型上皮細胞和ps系統超微結構的變化；免疫組織化學染色檢測glu的受體nmdar1亞單位的表達；生化測定肺組織乳酸脫氫酶（ ldh ）釋放量和肺組織勻漿中一氧化氮合酶（ nos ）活性、一氧化氮（ no ）生成量、超氧化物歧化酶（ sod ）水平以及丙二醛（ mda ）含量。
  10. Scientists at the imm used the in itro method to create lung epithelial cells known as aleolar epithelial type ii

    Imm的科學家以這種方法在體外製出了肺上皮細胞?又名ii型肺泡上皮細胞。
  11. Most of the cgrp positive cells in 16 - day fetuses were round or oval in shape, and usually rest on the basement membrane of epithelium and the primitive alveoli

    胚16日組陽性細胞呈單個球形或卵圓形，分佈中文摘要于肺內支氣管上皮基部和原始肺泡壁內。
  12. Like certain other bacterial pathogens, chlamydiae induce epithelial cells ? in this case, those lining genital tracts, eyelids or lungs ? to absorb them within a membrane - bound sac, or vacuole

    一如某些病原菌，披衣菌會誘使上皮細胞利用由細胞膜包圍而成的囊泡，將自己納入細胞內；對披衣菌而言，主要的感染對象是生殖道、眼瞼或肺臟的表皮細胞。
  13. Transplantable aleolar epithelial type ii cells may also one day be helpful in the treatment of other lung diseases including chronic obstructie pulmonary disease ( copd ), the fourth leading cause of death in the united states, claiming the lies of 122, 283 americans in 2003, and asthma, wetsel said

    可移植ii型肺泡上皮細胞有一天可能會有助於治療其它肺部疾病包括慢性阻塞性肺病（ copd ）及哮喘，慢性阻塞性肺病是美國第四大致死疾病，僅在2003年就奪去了122 ， 283人的生命。
  14. It was firstly observed that cgrp plays a protective role on lung injury and attenuates glutamate - induced inhibition of pulmonary surfactant lipid synthesis. the protective mechanisms of cgrp are not mediated by blocking no production

    並進一步觀察到rp具有抗iu所致肺泡上皮細胞損傷的保護作用，可減輕o對ps脂質合成的抑制，但cgrp的作用並不是通過阻斷o激活nos ，使no生成減少而實現的。
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