腎素瘤 的英文怎麼說

中文拼音 [shènliú]
腎素瘤 英文
reninoma renin-secreting tumor
  • : 名詞[生理學] (腎臟) kidney
  • : Ⅰ形容詞1 (本色; 白色) white 2 (顏色單純) plain; simple; quiet 3 (本來的; 原有的) native Ⅱ名...
  • : 名詞(瘤子) tumour
  1. Of the 41 functional adrenal tumors, 20 were primary aldosteronism, 10 were cushing ' s syndrome, 10 were pheochromocytoma, and one was an androgen - producing tumor

    在本研究41位功能性上腺腫中, 20位為上腺留鹽激過多癥, 10位為庫辛氏徵候群, 10位為嗜鉻細胞腫, 1位為雄性分泌腫
  2. However, citrinin, produced in the growth process of monascus, is a mycotoxin and will induce teratogenicity, carcinogenicity and mutagencity

    桔黴是一種真菌毒,作用的靶器官是臟,可以致畸、誘發腫、突變等。
  3. Clinical characteristics and relational factors analysis of multiple myeloma with renal involvement

    多發性骨髓損害臨床特徵及有關因分析
  4. Objective to report 16 cases of intracranial aneurysms with calcified vascular wall and study the relationship between vascular calcification and pathogenic mechanism of intracranial aneurysms. methods spiral ct scan, clinical data and serum calcium, phosphate, glucose, lipid, renal function of the 16 patients were studied in retrospect. all aneurysms were clipped, in which 8 were resected after clipping. results the location of aneurysms with aneurismal wall calcification was, on posterior communicating artery in 3, on middle cerebral artery in 2, on anterior communicating artery in 2, on basilar artery in 5 and on basilar artery in 4. all patients showed normal serum calcium, phosphate, glucose and normal renal function. serum lipid of patients had been elevated. all patients recovered with no significant neurological deficits. conclusion intracranial vascular calcification is an active and regulated process in close relation to atherosclerosis, serum calcium, phosphate, lipid, thyroid, parathyroid function and other factors. intracranial vascular calcification has close relationship with the pathogenesis and management of intracranial aneurysms

    目的報告16例壁有鈣化的顱內動脈病例的治療,分析顱內血管鈣化與顱內動脈病理發生機制的關系.方法回顧性分析16例壁有鈣化的顱內動脈病例的影像學、臨床資料及手術治療.結果壁有鈣化的顱內動脈的位置是: 3例位於后交通動脈, 2例位於大腦中動脈, 2例位於前交通動脈, 5例位於椎動脈, 4例位於基底動脈.所有患者血鈣、磷酸鹽、血糖、功能正常. 6例患者血脂增高. 16例患者均行動脈夾閉術,其中8例術中切除動脈,治療效果良好.結論動脈壁鈣化與其病理發生機制有關,常見于巨大或大動脈壁血管鈣化是與動脈粥樣硬化、血鈣、磷酸鹽、血脂、甲狀腺、甲狀旁腺等因有關的主動耗能,多因調控的病理過程
  5. Other mechanisms become involved when hypertension due to an identifiable cause ( eg, catecholamine release from a pheochromocytoma, renin and angiotensin from renal artery stenosis, aldosterone from an adrenal cortical adenoma ) has existed for some time

    當因某些易於確定的原因所引起的高血壓存在一段時間后,如嗜鉻細胞釋放的兒茶酚胺、動脈狹窄所產生的和血管緊張上腺皮質腺分泌的醛固酮等,其他機制也會參與高血壓病的形成。
  6. 1 x 10 m l - dopa can induce the settlement and metamorphosis of s. canopus larvae. but, adrenaline and noradrenaline do n ' t show any significant effects on the settlement and metamorphosis of ascidi an larvae

    對于l - dopa和兒茶酚胺類神經遞質, 1 10 ~ ( - 6 ) m的l - dopa顯示出對冠海鞘幼體的附著變態有誘導作用,而上腺和去甲上腺的誘導作用均不顯著。
  7. Indication : treatment of acute or chronic viral hepatitis ( type b, c ), herpes zoster, pointed condyloma, viral meningitis ( influenza virus, mumps virus, adenovirus ), hairy cell leukemia, chronic granulocyte leukemia, renal cell carcinoma, kaposi ' s sarcoma, lymphoma, malignant melanoma concerned with aids and other virus and tumor diseases

    :用於急慢性病毒性肝炎(乙肝、丙肝等) 、帶狀皰疹、尖銳濕疣、病毒性腦膜炎(流感病毒、腮腺炎病毒、腺病毒) 、毛細胞白血病、慢性粒細胞白血病、細胞癌、愛滋病相關性卡波濟氏肉、淋巴、惡性黑色等病毒性及腫性疾病。
  8. Sleep / waking cycle is a complex network modulation and many factors such as interleukin - 1 ( il - 1 ), tumor necrosis factor ( tnf ), growth hormone releasing hormone ( ghrh ), vasoactive intestina polypeptide ( vip ) and many conventional neurotransmitters such as serotonin ( 5 - ht ), acetylcholine ( ach ), norepinephrine ( ne ), dopamine ( da ) and gamma - aminobutyric acid ( gaba ) were involved in it. recent evidence has shown that no synthesized in neurons in several areas of the brain can induce the release of neurotransmitters. in the rat central nervous system, the anatomical distribution of nos - containing neurons is now well established, and it was reported that nos is co - localized with neurotransmitters well known for their involvement in sleep mechanisms, i. e. 5 - ht, ach, da and gaba

    鄭州大學2003屆碩士畢業論文gaba受體激動劑消除no合成酶抑制劑對大鼠睡/醒周期的影響睡/醒周期的形成是一個復雜的網路調控的結果,體內許多因子都參與了這一調控網路,這些因子如白介一1 ( il一1 ) 、腫壞死因子( tnf ) 、生長激釋放激( ghrh ) 、血管活性腸膚( vip )以及經典的神經遞質如5一輕色氨( 5一ht ) 、乙酞膽堿( ach ) 、去甲上腺( ne ) 、多巴胺( da )和卜氨基丁酸( gaba )等,它們在睡眠的發生和調節中也發揮著重要作用。
  9. In mice, for instance, delivery of melatonin increases the risk of tumor development, and the overproduction of growth hormone leads to kidney problems, premature heart and lung failure, and an increased probability of early death

    譬如在小白鼠身上,給予褪黑激會增加腫發生的風險,而生長激過量,也會造成臟問題、心臟與肺臟發育不全,以及增加早夭的機率。
  10. Survivors were at increased risk for stomach cancer, colorectal cancer, lung cancer, soft tissue sarcoma, melanoma, non - melanoma skin cancer, endometrial cancer, ovarian cancer, renal cancer, thyroid cancer, and leukemia

    她們患上胃癌直腸癌肺癌軟組織肉惡性黑色及其它種類的皮膚癌子宮內膜癌卵巢癌癌甲狀腺癌以及白血病的可能性均會有所增加。
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