膽脂堿酸 的英文怎麼說
中文拼音 [dǎnzhījiǎnsuān]
膽脂堿酸
英文
nopalinic acid-
The fish lil liquid hard capsule selects the pure fish oil and the import lecithin which the non - pollution deep sea fish refinement becomes is the primary data, including the rich unsaturated fatty acid ( dha + epa ) and phosphor - lipin acid radical choline ( pc ), is the young people and the child ce - rebrum grows the important nutrition ingredient
魚油液體硬膠囊選用無污深海魚提煉而成的精純魚和進口卵磷脂為主要原料，含豐富的不飽和脂肪酸（ dha + epa ）和磷脂酰膽堿（ pc ） ，是青少年及兒童大腦成長中重要的營養成分。By this mechanism, new phosphatides containing the fatty acids of phosphatidyl choline may be prepared.
根據這個原理，可以制備含有磷脂膽堿脂肪酸的新磷脂。Effects of sodium hyaluronate preparation containing phosphatidylcholine on osteoarthritis in rabbit
含磷脂酰膽堿的玻璃酸鈉制劑對兔骨關節炎的影響Mechanism of glutamate - induced inhibition of pulmonary surfactant lipid synthesis ? mda receptor ( nmdar ) antagonist mk - 801 could reverse glutamate - induced inhibition of [ 3h ] - choline incorporation in lung explants ; ? he expression of nmdar1 subtype existed in alveolar type ii epithelial cells ; ? he effect of glu on pc synthesis required l - arginine in cultured lung explants, nos activity and no production were increased significantly in lung explants with glu treatment ; and mk - 801 and nos inhibitor l - nna could block the increase of nos activity and no production induced by glu ; l - nna also could reverse the inhibition of [ 3h ] - choline incorporation induced by glu ; ( 4 ) mda content increased and sod level decreased in cultured lung explants of glu treament groups, mk - 801 and l - nna could reverse these effects ; ( 3 ) mk - 801, l - nna could block the release of ldh induced by glu
谷氨酸對肺表面活性物質脂質合成抑制的機制研究nmda受體拮抗劑mk - 801能逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應；肺泡上皮細胞有nmda受體nmdar1亞單位的表達； glu對ps脂質合成的影響依賴l -精氨酸的存在， glu可使肺組織勻漿中nos活性、 no合成量升高； mk - 801 、 nos的抑制劑l - nna均可阻斷glu所致的這些效應； l - nna可逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應； glu可使肺組織勻漿中mda碩士學位論文中文摘要含量增高、 sod水平下降。 mk七0 、 l nna能阻斷q所致的這些效應； mk80 、 l nna還能阻斷gill誘導的ldh釋放增多的效應。3. calcitonin gene - related peptide attenuates glutamate - induced inhibition of pulmonary surfactant lipid synthesis ( d cgrp could reverse the decrease of [ 3h ] - choline incorporation induced by glu with dose - dependence ; ( 2 ) cgrp could block the decrease of the content of cct a mrna induced by glu in lung tissue ; ( 3 } cgrp could reduce the impairment of the ultrastructure of at ii cells induced by glu ; ( 4 ) cgrp could reverse the increase of mda content and decrease of sod level induced by glu in cultured lung explants, respectively ; ? grp had no significant effect on nos activity and increase of no production induced by glu
降鈣素基因相關肽減輕谷氨酸所致肺表面活性物質脂質合成抑制的保護作用降鈣素基因相關肽grp ）可顯著減輕0所致肺組織h一膽堿摻入pc量的降低，並且呈劑量依賴保護效應； cgrp可逆轉gill所致cctqinrna含量的降低； cgrp可減輕o所致肺11上皮細胞超微結構的損傷； cgrp可逆轉o所致肺組織勻漿中mda含量增多、 sod水平降低的效應，並可逆轉q所致肺組織ldh釋放增多的效應； cg販對gill引起的nos活性和no含量的升高均沒有顯著性影響。Assessment of cholinesterase ( che ) inhibition is widely used as a specific biomarker for evaluating the exposure of non - target organisms to the pollutants, organophosphorus pesticide and carbamate
摘要應用水生生物膽堿脂酶作為生物標志物對水環境中有機磷和氨基甲酸鹽殺蟲劑的暴露和效應進行評價日益受到重視。Methods : in cultured lung explants without serum, the lipid component synthesis of pulmonary surfactant was evaluated in [ 3h ] - choline incorporation ; mrna content of phosphocholine cytidylyltransferase ( cct ) in lung explants was investigated in rt - pcr ; the changes of the ultrastructure of the at ii cells were observed with electron microscope ; the expression of nmdar1 subtype was observed in immunohistochemistry staining ; nitric oxide synthase ( nos ) activity, nitric oxide ( no ) content, superoxide dismutase ( sod ) level, malondialdehyde ( mda ) content and lactae dehydroase ( ldh ) level were determined by biochemistry methods. results : 1. influence of glutamate on synthesis of the lipid component of pulmonary surfactant ? with l - arginine, glu inhibited [ 3h ] - choline incorporation with good dose - dependence and time - dependence ; ( 2 ) mrna content of cct of the glu treatment groups was decreased ; ( 3 ) glu increases the release of ldh in cultured lung explants ; ( dwith electron microscope histochemistry, glu induced the changes of the ultrastruture of at ii iv cells
方法：採用成年大鼠肺組織無血清培養，運用[ ~ 3h ] -膽堿摻入法測定ps主要脂質磷脂酰膽堿（ pc ）合成量； rt - pcr擴增檢測肺組織中pc合成限速酶磷酸膽堿二胞苷酰基轉移酶（ cct ） mrna含量；透射電子顯微鏡法觀察肺泡型上皮細胞和ps系統超微結構的變化；免疫組織化學染色檢測glu的受體nmdar1亞單位的表達；生化測定肺組織乳酸脫氫酶（ ldh ）釋放量和肺組織勻漿中一氧化氮合酶（ nos ）活性、一氧化氮（ no ）生成量、超氧化物歧化酶（ sod ）水平以及丙二醛（ mda ）含量。Promotive action of lysophosphatidic acid on proliferation of rat embryonic neural stem cells and their differentiation to cholinergic neurons in vitro
溶血磷脂酸促進離體大鼠胚胎神經幹細胞的增殖及其向膽堿能神經元分化Lecithin ( phosphatidyl choline ) one of a group of phospholipids that contain glycerol, fatty acid, phosphoric acid, and choline that are found widely in higher plants and animals, particularly as a component of cell membranes
卵磷脂（磷脂酰膽堿） ：磷脂的一種，由甘油、脂肪酸、磷酸和膽堿組成，在高等動植物中廣泛存在，是細胞膜的重要組分。A mechanism that interprets this phenomenon was proposed as follows : the adsorbents can interact with phospholipid of ldl through calcium ion cross - linking of the phospholipid phosphate groups of ldl and the carboxyl groups of sde ligands. on the other hand, the electrostatic interaction between the positive charge of the phospholipid choline nitrogen of ldl and the negative charge of carboxyl groups of sde ligands is enhanced by the calcium cross - linking of the phosphate groups of neighboring phospholipids in ldl
其可能的原因是：在吸附體系中如存在大量的cay ，一方面ca可以作為橋梁連接ldl所含磷脂極性頭部的磷酸基團和吸附微珠配體上的孩基；另一方面又可交聯相鄰磷脂間的磷酸基團，從而促進ldl所含磷脂極性頭部的膽堿n 」與配體竣基的相互作用。分享友人