致病變型 的英文怎麼說

中文拼音 [zhìbìngbiànxíng]
致病變型 英文
pathovar
  • 致 : Ⅰ動詞1 （給與;向對方表示禮節、情意等） deliver; send; extend 2 （集中於某個方面） devote （one s ...
  • 病 : Ⅰ名詞1 （疾病; 失去健康的狀態） illness; sickness; disease; malum; nosema; malady; morbus; vitium...
  • 致病 : pathopoiesia; pathopoiesis致病菌 disease germ; pathogenic bacteria; 致病因素 pathogenic factor
  1. The results of biological tests have demonstrated that allantoic fluid of the first passage virus did n ' t produce macroscopic pathogenic role to chicken embryos and after passaged for four times, gross lesions were observed in chicken embryo. the virus showed typical coronavirus under electron - microscope and it could n ' t form plaque in cef cells and could hemagglutinates chicken red blood cells after treatment with 1 % trypsin. to surprise, the virus replicated in cef cells also showed hemagglutination activity to chicken red blood cells. in addition, the spf chickens which inoculated with the virus isolated from the chicken damaged tissue showed clinical sign and grow lesion, but it ' s gross lesion did n ' t resemble to those of field outbreaks

    生物學特性：雞胚尿囊液經離心、磷鎢酸負染后，電鏡觀察該病毒為典型的冠狀病毒；該毒株的第一代尿囊液對雞胚無肉眼可見的致病作用，當繼代到第5代后，胚體嚴重病變；病毒在雞胚中隨著接種時間的延長，其效價增高， 96h可達到48h的2倍；該毒株可在cef上生長，但不能形成明顯的蝕斑；經1胰酶處理后可凝集雞紅細胞；雞胚的第四代尿囊液病毒回歸動物體，病死雞腎臟呈典型的花斑腎，腺胃則未見肉眼可見的病變。
  2. The arteriolar sclerosis that results from chronic hypertension leads to small lacunar infarcts, or " lacunes ", one of which is seen here in the pons

    緩進型高血壓小動脈硬化導致小范圍的腔隙性梗死灶，腦橋上可見一此種病變。
  3. Gpv ( goose parvovrius ) has been classified by its morphological, biochemical, and clut - ure characteristics as a member of the parvovirus genus of the parvoviridae. lt is a highly fatal disease of gos1ings and muscovy ducklings, the typical pathological lesions was found in digest tract, specially in small intestine

    Gpv （ gooseparvovirus ）依據其形態、生化及培養特性被劃歸為細小病毒科細小病毒屬成員，引起雛鵝和雛番鴨高度致死性疾病，主要以消化道，尤其是小腸部位的典型病變為特徵。
  4. In this study, sd rats were used to establish the animal model of brain injury induced by repeated + gz exposure and suppression subtractive hybridization technique was adopted to screen the differentially expressed genes in rat brains of + gz exposure group. the aim of the study was to obtain preliminary experimental data for the molecular mechanisms of the brain injury

    本研究利用大鼠重復+ 10gz暴露引發腦損傷的動物模型，觀察腦的病理學改變；應用抑制性消減雜交技術篩選+ gz重復暴露大鼠腦的差異表達基因，旨在初步探討+ gz重復暴露致腦損傷的分子機制。
  5. Bovine viral diarrhea virus were propagated by mdbk cells. after five to six continuously and rapidly passing, the cells emerged evident cytopathic effect. then the infected cells and its solution were harvested

    Bvdvnadl株屬于致細胞病變型毒株，用mdbk細胞培養增殖bvdv ，經5 - 6次連續傳代至出現明顯的細胞病變。
  6. During follow - up, the two grade 1 ulcers which infected with strains carrying virulence genes rapidly deteriored : the array technology showed unchanged genotype profiles

    在隨訪中，感染攜帶致病基因的兩名1級病人病程急劇惡化：晶元方法顯示基因型未發生改變。
  7. In 1996, there was global concern when scientists found that eating beef products from infected cows may cause a similar fatal brain disease in humans - a variant of creutzfeldt - jakob disease

    1996年，科學家發現食用感染的牛肉產品可能導致人類患類似的致命腦病，即新變異型克雅氏病，這引發了全球性的關注。
  8. This autoimmune disease leads to synovial proliferation and joint destruction, typically in a symmetrical pattern involving small joints of hands and feet, followed by wrists, ankles, elbows, and knees

    這種自身免疫病導致滑膜增生與關節破壞，典型病變為對稱性畸形，出現于手與足的小關節，其次是腕關節、踝關節、肘關節、膝關節。
  9. The rest mutations in pp38 and pp24 are at random. sequence analysis also shows the first 195 nuclear acids of pp38 and pp24 are the same except for the 81 site ( g / c ), but this mutation does not cause the change of amino acid. we regard this as a genetic marker connecting with geography in the evolution of mdv but not related to isolated time and pathotype of different strain of mdv i

    對pp24基因和pp38基因進行同源性比較分析的結果表明，絕大多數毒株二者的前195個核苷酸完全一致，不同毒株間的第81位核苷酸的差異（ g / c ）並不引起編碼的氨基酸變化，僅僅與地域分佈有關，這很可能是mdv在長期病毒衍化過程中形成的地域性遺傳標志，而與病毒的分離年代及mdv的致病型等因素無關。
  10. The comparative analysis of sequences indicated that the sequences of meq in different pathotypes are relatively conserved and the homology of the amino acid sequences is very high. the significant differences include two mutations in both mdv - 1 vaccine strains cvi988 / rispens and 814 strain : the deletion of a proline ( no. l93aa ), and this mutation is just exactly located in a 15 - amino - acid ( eelcaqlcstppppi ) repeat sequence within the c - terminal transactivation domain of meq protein ; and a point mutation with a shift from alanine ( a ) of all virulent strains to serine ( s ) was occurred on the no. 71 aa

    結果發現， mdv不同致病型的meq基因序列相對比較保守，它們相互間核苷酸和氨基酸序列的同源性均很高；但是，與所有七個致瘤性的mdv毒株相比，二個型弱毒疫苗cv1988 rispens株和814株均出現了兩個特徵性的突變：即第194位的p缺失性突變和第71位氨基酸由a變成了s的位點突變；缺失性突變恰恰位於meq基因中轉錄激活域內的一個多脯氨酸的重復序列（ pppp ）之中。
  11. Immunologic effect on the model rat of spleen - qi deficiency induced by complex factors

    復合病因造模法致脾虛證大鼠模型在免疫系統方面的變化
  12. Aim : 1. to establish a lidocaine subarachnoid anesthesia death model with dogs, observe the vital sign and pathology of dogs after a lethal dose of lidocaine being injected into subarachnoid space. 2

    目的1 、本研究選擇狗建立利多卡因蛛網膜下腔麻醉致死的動物模型，觀察狗蛛網膜下腔麻醉致死過程的生命體征變化和死後組織臟器的病理改變。
  13. The paper build a plane model of the whole bridge based on the fem, and analyze the internal force and structural displacement changes in every damage circumstance. the data reflect that the damages in arch rib in the tip will make internal force of some sections increased, accordingly the stability and the durability of the structure reduce ; the damages in the arch foot will change the structure system ; and the displacements of abutments will develop great accessional internal force, and deform the main arch and even crack, accordingly change the system of the structure

    論文建立了全橋的平面有限元模型，分析了在各種病害情況下全橋的內力及結構位移的變化，計算數據反映了，拱頂拱肋損傷會造成某些截面的內力增大，結構的穩定性和耐久性降低；拱腳損傷可能引起結構體系改變；墩臺位移會使上部結構產生較大的附加內力，使拱圈變形甚至於開裂，從而導致結構的受力體系發生改變等。
  14. Three times in the last century, the influenza a viruses have undergone major genetic changes mainly in their h - component, resulting in global pandemics and large tolls

    甲型流感病毒在上一世紀經歷了三次重大基因變異，變異部分以h組成部分為主，造成全球大流行，並引致重大傷亡。
  15. In this chapter, we point out that the armijo - type line search will make the step k very small even close to zero in some cases, which leads to the pathological problem

    在本章中，我們指出採用armijo型的線搜索，在某些情況下易使步長變的很小，甚至趨于零，這將在計算過程中導致病態現象。
  16. A quantifying model of vascular proliferation in oxygen - induced retinopathy

    可定量的氧致血管增生性視網膜病變小鼠模型
  17. Laboratory research on l - form variation and pathogenicity of vibrio cholerae o

    型變異和誘導實驗的觀察及致病性的探討
  18. Hemorheological changes in blood flow can lead to endothelial dysfunction, resulting in real - time microvascular abnormalities in vascular diseases, including type - 2 diabetes mellitus t2dm and alzheimer s disease

    血液流變可產生血管功能障礙，引致即時微血管異常等血管疾病，包括2型糖尿病及老人癡呆癥等。然而即時微血管異常與血液流變的直接關系仍有待實驗證明。
  19. As qzkvjd biotechnology becomes more sophisticated and powerful, and as the genetic code smt of more organisms is identified, biologiosts will learn how to mix genes uof different microbes to create unnatural strains that cana e turned i xntoteadly, effective 762720265 weapons

    隨著生物技術變得越來越復雜和強大，隨著越來越多的有機體的基因密碼被識別出來，生物學家們將會研究出如何混合各種不同類型的微生物以製造出非天然的病菌，可以將它們製成致命而威力強大的武器。
  20. In 2001, wilson and his colleagues cloned another two members - human wnk1 gene and wnk4 gene which were located on chromosome 12 and 17, respectively. the two genes are disease - causing genes responsible for a mendelian hypertension. the disease - causing mutations are the large deletions in the first intron in wnk1 gene, causing increased expression in leukocytes

    2001年， wilson等克隆了此家族的另兩個成員? ?人的wnk1和wnk4基因，分別位於12號和17號染色體，這兩個基因是一種孟德爾遺傳型高血壓病的致病基因，致病突變分別是wnk1基因第一內含子的大片段缺失，導致患者白細胞的基因表達量提高5倍左右。
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