藥物性肌病 的英文怎麼說

中文拼音 [yàowùxìngjībìng]
藥物性肌病 英文
drug myopathy
  • 藥 : Ⅰ名詞1 （藥物） medicine; drug; remedy 2 （某些有化學作用的物質） certain chemicals Ⅱ動詞1 [書面...
  • 物 : 名詞1 （東西） thing; matter; object 2 （指自己以外的人或與己相對的環境） other people; the outsi...
  • 性 : Ⅰ名詞1 （性格） nature; character; disposition 2 （性能; 性質） property; quality 3 （性別） sex ...
  • 肌 : 名詞1. （肌肉） muscle; flesh 2. [書面語] （皮膚） skin
  • 病 : Ⅰ名詞1 （疾病; 失去健康的狀態） illness; sickness; disease; malum; nosema; malady; morbus; vitium...
  • 藥物 : medicinal; medicine; medicant; medication; drug; pharmaceuticals; medicaments
  1. Magical fire ( eye doctor ) looks at well - being cream of sticking drawing tradition traditional chinese medicine formula famous and precious in tsinghua, is tied in wedlock modern up - to - date medicine result of scientific research, the various chinese medicinal crop famous and precious being carefully chosen, adopt the modern nano - technology and target to poison a technology to being given to, let various active material, tiny molecule, nutrition factor glutathione etc. guide medicine it is all right for to go ahead, the brute force passes through blood eye parclose, make pesticide effect reach nidus directly location, prompt the nutrition replenishing an eye with the part ( include ciliary muscle, retina, crystalline lens, optic nerve ), active eye part cell, improve eye part immunocompetence and oxidation resistance, boost an eye part organizing an assimilation of the new and excretion of the old, microcirculation improving and restoring an eye part, thereby reach eliminate look at strain, purpose improving and improving sight

    清華神火視康貼汲取傳統中藥名貴配方之精華，結合現代醫藥最新科研成果，精選多種名貴中藥材，採用現代納米技術和靶向給藥技術，讓多種活性物質、微分子、營養因子谷胱甘肽等引藥上行，強力穿透血眼屏障，使藥效直達病灶部位，迅速補充眼部（包括睫狀肌、視網膜、晶狀體、視神經）的營養，激活眼部細胞，提高眼部免疫能力和抗氧化能力，促進眼部組織新陳代謝，改善和恢復眼部微循環，從而達到消除視疲勞，改善和提高視力的目的。
  2. If pathological changes has restored, period of treatment was finished, recrudescent possibility is dinky ; tuberculosis has infection of n / med tuberculosis bacterium to cause, the cold has a fever is not main reason, it is inducement likely ; after tuberculosis is cured, the meeting inside body is put in dormancy bacterium, dormancy bacterium won ' t come on commonly, do not infect, it is to rely on human body resistance to keep clear of, remedy is not effective to dormancy bacterium

    假如病變恢復好，療程完成了，復發的可能性極小；結核病是有結核菌感染引起的，感冒發燒不是主要原因，有可能是誘因；結核病治愈之後，體內會存在休眠菌，休眠菌一般不會發病，不傳染，是依靠肌體反抗力來清除，藥物對休眠菌不起作用。
  3. Corticosteroids : corticosteroids most commonly have been reported to cause drug - induced myopathy

    皮質類固醇激素是目前報道較多的能引起藥物誘導性肌病的藥物。
  4. Mutations in ion channels are likely to be inoled in epilepsy, chronic pain, neurodegeneratie diseases and muscular diseases, and ion channels are the target of many drugs

    癲癇，慢性疼痛，神經變性疾病，和肌肉疾病可能都涉及到離子通道的突變，並且離子通道是許多藥物的靶點。
  5. In the third experiment, we first constructed a test model of differentiation from mouse es cells to cardiomyocytes. then series of dilutions of three chemicals were screened on this platform. the combination of all trans retinoic acid and dmso is employed as positive control

    實驗三首先建立了一個小鼠胚胎幹細胞定向分化成心肌細胞的實驗平臺，然後採用了3種心肌疾病相關的藥物：黃芑生脈飲、 1 ， 6二磷酸果糖和hg （代號） ，在此定向分化的實驗平臺上進行了一系列濃度的藥物篩選，實驗採用視黃酸和dmso的混合誘導劑作為陽性對照。
  6. Emg analysis of peripheral nerve lesion resulting from drug toxicity

    藥物中毒性周圍神經病變肌電圖分析
  7. Neuroleptic malignant syndrome presenting features include : fever, altered level of consciousness, and " lead - pipe " skeletal muscle rigidity causing rhabdomyolysis

    抗精神病藥物惡性癥候群會引起意識改變、高燒、鉛管樣肌肉僵直並橫紋肌溶解癥。
  8. But the patient because of constitutionally distinctive, produced drug allergy feeling to die ( the person that be like some case disease has uterine flesh tumor, lumbar hemp applies in the operation, shock of sensibility of patient happening drug allergy is sudden ), should consider vest in accident to this kind of death like that

    但是病人由於體質上的特異，發生了藥物過敏感死亡（如某一案例病者患子宮肌瘤，手術中施行腰麻，病人發生藥物過敏感性休克忽然） ，然對于這類死亡應考慮歸屬于意外事件。
  9. Emerging knowledge about the key mechanisms, such as the impairment of glucose - stimulated insulin secretion and lipotoxicity and the role of muscle resistance to insulin secretion has become the focus of drug therapy

    隨著分子生物學研究的開展及對2型糖尿病發病關鍵機制的認識加深：葡萄糖依賴的胰島素分泌功能受損、脂毒性、肌肉組織的胰島素抵抗逐漸成為藥物治療的靶點。
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