誘導抑制性 的英文怎麼說
中文拼音 [yòudǎoyìzhìxìng]
誘導抑制性
英文
suppressor inducer t cell- 誘 : 動詞1. (誘導) guide; lead; induce 2. (使用手段引人隨從自己的意願) lure; seduce; entice
- 導 : 動詞1. (引導) lead; guide 2. (傳導) transmit; conduct 3. (開導) instruct; teach; give guidance to
- 抑 : Ⅰ動詞(向下按; 壓制) restrain; repress; curb Ⅱ連詞[書面語]1 (表示抉擇) or 2 (表示轉折) but3 ...
- 制 : Ⅰ動詞1 (製造) make; manufacture 2 (擬訂; 規定) draw up; establish 3 (用強力約束; 限定; 管束...
- 性 : Ⅰ名詞1 (性格) nature; character; disposition 2 (性能; 性質) property; quality 3 (性別) sex ...
- 誘導 : guide; lead; induce; guidance; induction
- 抑制 : 1 (控制) restrain; control; check; hold up; curb; stop; repress; bridle; choke; prehension; sup...
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Ketamine inhibits the excitability induced by formalin in spinal dorsal horn neurons of rats
氯胺酮對甲醛致痛誘導大鼠脊髓背角神經元興奮性的抑制In addition, axud1 has a high homologue with transforming growth facotr beta ( tgf - ) induced apoptosis protein 2 and 12 using blast exploration at genebank database. these suggest that axudl may have a tumor - suppressor function in these organs and may play an important role in tgf - - induced apoptosis
通過genkbank上blast的搜索結果表明, axud1與tgf -誘導的凋亡蛋白2 、 12 ( taip - 2 、 taip - 12有較高的同源性,提示axud1可能是一個具有腫瘤抑制功能的基因,並可能在tgf -誘導的細胞凋亡中起著重要作用。Inhibition of steroidogenesis and induction of apoptosis in rat luteal cells by cell - permeable ceramide in vitro
細胞滲透性神經酰胺可抑制大鼠離體黃體細胞甾體激素生成並誘導細胞凋亡It is found that the noise color can suppresses the noise - induce transport of brownian particles both in periodic and in quasi - periodic systems
結果發現在周期性和非周期性系統中,噪聲的有色性抑制了噪聲誘導的布朗粒子的輸運現象。Based on the extensive studies of subtilisin - like protease ( prl ) of metarhizium anisopliae, extracellullar serine protease is suggested to be a key enzyme involved in the fimgal penetration to invertebrates. the investigation of serine protease in the nematode infected by owvtl may help to understand the mechanism of nematophagous fimgi as biological control agents. a 3l kda serine protease was isolated and purified from the liquid culture of h rhossiliensis owvtl challenged with nematode panagrellus redivivus
本研究利用線蟲誘導下owvt - 1菌株液體發酵,通過粗分級分離、離子交換層析和凝膠過濾層析分離提純了一個分子量為31kda的絲氨酸蛋白酶,生物學測定表明其對大豆胞囊線蟲二齡幼蟲具有致死作用,同時測定了該酶理化特性,酶活力在75附近酶活力最高,隨著ph的增加酶的穩定性升高,與膽堿酯酶具有相似的ph曲線,對特異性底物aape ( suc - ala - ala - pro - glu - pna )具有作用, ssi和ci - 2抑制該酶的活性。The deleted mutant pap gene was also cloned into yeast secreted expression ppic9k vector to form ppic9k ~ 3, then the vector was transferred into pachia pastoris gs115 strain. the specific expression protein was secreted into the medium after inducing with methanol and the protein amount reached about 50 - 60 u g per millilitre measured by uv - absorbed methods in the supernatant of the medium via high density fermentation. sds - page results showed that there was one protein band in the gel which molecular weight was about 34ku
將缺失型pap基因克隆于酵母分泌型表達載體ppicgk構成重組載體,然後導入畢赤酵母( p8chianastoris )菌株gslls細胞中,在甲醇的誘導下,經過酵母高密度發酵進行pap的表達,經sds page分析,結果表明,在培養基上清液中含有一明顯的特異性蛋臼條帶,大小為34ku ,經western blotting分析,該蛋白與法國pap抗血清有特異性反應,體外活性檢測表明該蛋白對tmv的侵染性具有高度的抑制性,說明該pap基因在畢赤酵母gs中也得到了正確表達。Curcumin has been shown to induce a wide variety of tumor cells by several mechanisms, mainly including modulation of the expression of oncogenes and cancer suppressor genes, down - regulation the activation of transcription factors, via many signal transduction pathways, and the modulation of cell cycle. provides an overview of domestic and international apoptosis mechanisms of malign tumor cells induced by curcumin in order to better explore and open up new avenues of cancer treatment
姜黃素能夠誘導多種腫瘤細胞系凋亡,其機制主要是調控癌基因和抑癌基因,下調多個轉錄因子活性,通過多種信號轉導途徑,誘發細胞周期停滯而誘導細胞凋亡.對近年來姜黃素誘導細胞凋亡的機制進行綜述,以便更好地探求和開辟治療惡性腫瘤的新途徑。The carbofuran - degrading experiment of cds - 1 was carried out in lab scale, the results showed that the highest degrading efficiency was obtained with ph, temperature being 7. 0, 30c respectively ; the change of aeration had no influece on degrading rate ; the increasing inocula could accelerate carbofuran degrading progress ; the degrading capability of cds - 1 was n ' t inhibited by high carbofuran concentration ; the addition of low concentration of nutrients had no distinct effect on the degrading rate while high concentration had inhibiting effect the distribution of degrading enzyme was also primarily studied, the results showed that degrading - related enzyme was endocellular and degrading progress was not cometabolism
Cds - 1的降解酶(系)是誘導酶(系) ,存在明顯的誘導期;胞內、胞外酶實驗表明呋喃丹降解酶(系)存在於細胞內。添加低濃度外源營養物質對cds - 1的降解性能無明顯影響,說明cds - 1降解呋喃丹的過程不屬于共代謝過程,可以在無外源營養物質存在的條件下降解呋喃丹。添加高濃度外源營養物質會對該菌降解性能產生抑制。No, a first gas information molecule discovered in human being, is a typical endothelial - derived relaxant and mediates endothelium - dependent relaxation of blood vessels. in the pathogesis of endotoxin shock vec is one of the major target cells of lps and lps - induced proinflammatory cytokine such as tumor necrosis factor and interlukin 1 and activated. in vec inducible nitric oxide synthase ( inos ) is induced and lead to an increase in production of no, the while endothelial nitric oxide synthase ( enos ) is inhibited and elicit decrease in no formation, both of which are demonstrated to induce the
在內毒素休克過程中vec是lps及其誘導機體產生的多種促炎細胞因子如tnf 、 il - 1作用的主要靶細胞, vec誘導型一氧化氮合酶( induciblenitricoxidesynthase , inos )激活、 no大量誘生而內皮型一氧化氮合酶( endothelialnitricoxidesynthase , enos )活性被抑制、 no生成障礙,是血管反應性異常變化、血管調節機制紊亂的重要發病環節。In our experiments, however, the inhibition of flavonoid on root system development is related to high nitrate, and quercetin, one kind of flavonoid, does not inhibit but promotes the gravitropism instead
但我們在實驗中發現,類黃酮對根系發生的抑制是與高n相聯系的。對于向地性,類黃酮中的一種? ? quercetin不但不抑制,反而促進向地性? ?誘導超向地性。The protein level of p21 increased lightly from 1 to3 h, then decreased at 6 - 9 h, reached maximum at 24 h after 2gy, it increased from 3 to 24 h exposed tology, and the protein level of p21 associated with cdk2 increased, which did not inhibit the cyclin e / cdk2 kinase activity, because of upregulation of cyclin e and deregulation of p27kipl after irradiation. in contrast to p21, p27kipl may be a key regulator of cyclin e / cdk2 activity in dna damage responses. hi the ubiquitin - proteasome pathway invovled in regulation of p27kiplexpression in response to ir the ubiquitin - proteasome pathway regulated selective and time - controlled elimination of p27 kipl
與p27ki 」 ,表達水平不同, pzi蛋白表達水平在zgy照射后略有增加, 6一gh略有下降,在照后24h達到最大; 10gy照后1一3h開始增加,持續到24h ,並且cycline / cdkz復合物中pzi蛋白結合增多,但cycline / cdkz激酶活性並未受到抑制,除了因ir誘導cydine蛋白表達水平增加外,另一重要的原因就是與pzi同一家族的p27kip ,蛋白表達水平下降,失去了對cycline / cdkz的抑制活性。The effects of single - species " films may be the function of the character of the bacterium or the function of their exopolymers
單種菌株的菌膜所表現出的誘導或抑制作用,可能是細菌本身的特性的表現,也可能是它所分泌的胞外產物的作用。The animal study was designed to determine whether ph - egf fusion protein was protective to ethanol induced injury of gastric mucosa in rats. thirty - two sd rats weighting 200 - 250g starved for 24hour were divided into four groups which received pupae infected with recombinant bmbacph - egf virus, normal pupae, ranitidine, 0. 9 % saline respectively for 3days before received absolute ethanol. the results showed that both pupae infected with bmbacph - egf virus and normal pupae protect the gastric mucosa against ethanol induced damage
本研究還建立了動物模型,大鼠乙醇灌胃后的胃粘膜經病理學觀察和病理組織學檢查,發現重組病毒感染蠶蛹5天的樣品灌胃后對乙醇誘導潰瘍的預防性抑制率達50左右,但正常蠶蛹樣品也有一定的預防乙醇誘導損傷、保護胃粘膜作用。For the acute exposure duration, there expressed the dose - thresholds and time - thresholds in the effects of cu2 + on the activities of cat, pod and sod in two macrophytes. when the test species were exposed at low dose and for a short period, cu2 + can stimulate activities of antioxidant enzymes. once exceeding the thresholds, cu2 + may inhibit enzymes activities
Cu ~ ( 2 + )低濃度和短時間內對抗氧化酶有誘導作用,超過一定的閾限會產生抑制作用;金魚藻cat 、 pod 、 sod活性在7天內的最大抑制率與暴露cu ~ ( 2 + )濃度呈顯著正相關。The activity of sod of the gill was restrained more and more by cd2 + concentration from 0 - 1. 50mg / l and from time 1 - 7 day, and the activity of sod of the liver was also restrained by 1. 50mg / l cd2 + concentration, but increased by the lower cd2 + concentration, in the hard water group the activity of sod increased especially in the higher cd2 + concentration group
鎘對草魚鰓sod活性抑制隨鎘作用時間延長和濃度增高而加深,而對肝臟sod作用在低濃度時能起一定誘導作用,高濃度時則隨時間延長而受到明顯抑制,提高水環境中的ca ~ ( 2 + )濃度能使sod活性增強,高鎘離子濃度時增強尤為顯著。Three kinds of works were described in this thesis with bacillus thuringiensis as major material. 1. autoinducer inactivity protein aii in b. thuringiensis strains and its effect on pathogenicity of plant - pathogen bacteria autoinducer, especially ahl molecule, is a kind of cell - to - cell communication molecule that has extensive distribution in gram - negative plant pathogen bacteria
蘇雲金芽胞桿菌自身誘導物抑制蛋白aii及其對植物病原菌致病性的影響自身誘導物( autoinducer ,以下簡稱ai )分子是一種廣泛存在於革蘭氏陰性植物病原菌的細胞間信號分子,與病原菌的致病性有著直接或問接的關系。Increased citrate efflux due to the sa treatment was associated with decreased inhibition of root growth and al content in root tips, suggesting that exogenous sa could confer al tolerance by increasing citrate efflux
由於sa增加al誘導的檸檬酸的分泌,因此明顯降低了根尖內al的含量,改善了鋁對根生長的抑制,表明sa可以通過增加檸檬酸的分泌來提高c . tora對鋁的抗性。Mitogen - activated protein ( map ) kinase signal transduction cascades are routes through which eukaryotic cells deliver extracellular messages to the cytosol and nucleus, and the increasing evidences showed that mapks are involved in aba -, sa - or h2o2 - signaling respectively. in addition, plant guard cells have been a well - developed model system for understanding how components interact within a signaling network in a single cell
本實驗在表皮生物分析的基礎上,主要利用顯微注射技術、膜片鉗技術和激光共聚焦顯微技術,運用專一性蛋白激酶抑制劑處理,探索蛋白激酶對蠶豆( viciafabal . )氣孔保衛細胞中aba和sa誘導的h _ 2o _ 2產生及其信號轉導影響機理,結果如下: 1As a proteasome inhibitor, bortezomib has shown inhibition on cell proliferation, inducing cell apoptosis, inhibition of cell adhension and inhibition of tumor angiogenesis for multiple myeloma cells
摘要硼替佐米作為一種蛋白酶抑制劑,對多發性骨髓瘤細胞表現出抑制細胞生長、誘導腫瘤細胞凋亡、抑制細胞粘附、抑制腫瘤血管生成等作用,同時對血液系統其他惡性腫瘤具有顯著的作用。When the body suffered from malignancy, on the one hand, tumor restrained cell immunity function, antibody ' s production and the body ' s immunity effect through inducing restraining t cell, so it accelerated tumor ' s growth, on the other hand, tumor induced body fluid restrained gene, which prevented t cell and nk cell from killing tumor and restrained lymphocyte to mitotic multiplication reaction, indeed it accelerated tumor ' s growth
當機體一旦患上惡性腫瘤后,一方面腫瘤通過誘導抑制性t細胞,抑制細胞免疫功能及抗體的產生,抑制機體對腫瘤的免疫效應,從而使腫瘤逃逸機體的免疫監視,促進腫瘤的生長。另一方面,腫瘤通過誘導體液抑制因子阻止t細胞和nk細胞的等對腫瘤的殺傷作用,抑制淋巴細胞對有絲分裂原的增殖反應,甚至促進腫瘤的生長。分享友人