酪氨酸轉氨酸 的英文怎麼說

中文拼音 [làoānsuānzhuǎnānsuān]
酪氨酸轉氨酸 英文
tyrosine transaminase
  • 酪 : 1 （用牛、羊、馬的乳汁製成的半凝固的食品） junket 2 （用果子做的糊狀食品） thick fruit juice; fru...
  • 氨 : 名詞[化學] （氮和氫的化合物） ammonia; hydrogen nitride
  • 酸 : 酸構詞成分。
  • 轉 : 轉構詞成分。
  1. There was less nuclear translocation of tyrosine - phosphorylated stat3 in the luminal epithelium of implantation sites on day 5 of pregnancy

    第5天胚胎著床以後，著床位點處子宮腔上皮中酪氨酸磷酸化stat3的核轉位減弱。
  2. By observing the effect of acidity on the uv difference spectra of ehpg or hbed, the spectral characteristics of apoovotransferrin binding to metals and anions at ph 7. 4 show that the reaction of apoovotransferrin with metals is related to deprotonation of the phenolic group of tyrosine of the metal binding sites, while the binding of apoovotransferrin to anions is associated with the protonation of the phenolic group of tyrosine of the anion binding sites

    摘要通過觀察酸度對模擬轉鐵蛋白小分子ehpg和hbed紫外差光譜的影響，結合脫鐵伴清蛋白與金屬離子、陰離子結合的光譜特性，推斷脫鐵伴清蛋白與金屬離子的結合導致金屬離子結合位點處酪氨酸酚羥基的去質子化，而與陰離子的結合導致陰離子結合位點處酪氨酸酚羥基的質子化。
  3. Nuclear translocation of tyrosine - phosphorylated stat3 was also not detected in the uteri on day 4 of pseudopregnancy and under delayed implantation

    在假孕第4天的子宮和延遲著床的子宮中未發現酪氨酸磷酸化stat3的核轉位現象。
  4. Pka, receptor tyrosine kinase ( trk ) and classical nuclear receptor of gc were not involved in the gc " s activation of mapks the second part studied the nuclear translocation of gc activated mapks, mainly p38 and jnk, with laser confocal microscopy. the results showed that : 1

    Gc激活的mapks的激活不需要pka酪氨酸激酶受體trk及經典gc核受體的參與第二部分是研究gc激活的mapks的核轉位，主要是p38和jnk ，用激光共聚焦顯微鏡觀察到以下結果： 1
  5. Studies revealed that p - catenin dissociated from ccc and translocated into free catenin pool in cytosol after it has been phosphorylated at tyrosine or serine residues, and in this situation, the ccc has been disrupted and cell adhesion function disturbed. a large amount of the free p - catenins in the cytosol can be degraded by the tumor suppressor apc, and the remains translocate into nucleus and bind to transcriptional factor tcf / lef in the nucleus and then promote cell proliferation related gene or anti - apoptosis gene transcription

    當-連環蛋白酪氨酸或絲氨酸殘基磷酸化后，就與ajs發生解離而游離到細胞漿中，此時細胞的粘附功能也發生障礙，游離到胞漿中的-連環蛋白，一部分被抑癌因子apc降解，一部分則轉移到細胞核內，與核內的轉錄因子tcf lef結合，啟動與細胞增殖有關的基因轉錄。
  6. There were strong stat3 immunostaining in the glandular epithelium and weak nuclear translocation of tyrosine - phosphorylated stat3 in the luminal epithelium containing a blastocyst in the day 4 morning

    第4天上午子宮腺上皮中的stat3免疫染色較強，且在胚泡所在處的的子宮腔上皮中有弱的酪氨酸磷酸化stat3蛋白的核轉位。
  7. These results indicate that the alteration of cell proliferation and dna synthesis caused by different gnt - v cdna transfection may at least partly result from the modification of n - glycan structure and function of egfr. it seems that the increased 1, 6 glcnac branch on the n - glycans of egfr may benefit to its binding with egf and the resulting tyrosine auto - phosphorylatio n, while the decrease of this branch may prevent these processes

    用特異性抗體結合westemblot結果發現，正義或反義gnt一vcdna的轉染並不引起pkb 、 p44 / 42mapk和mek蛋白質表達的變化，而gntv一s / h 」 21細胞pkbt308 、 5473位點磷酸化和免疫沉澱pkb的酪氨酸磷酸化以及以gsk召a /日磷酸化為指標的pkb的活性都較mock細胞增加， gntv一as / h7721細胞中這些指標的變化則相反。
  8. Exercise also decreased striatal da transporter and tyrosine hydroxylase immunoreactivity in mptp - lesioned mice

    運動也可以減少紋狀體多巴胺轉運體，還能抑制酪氨酸羥化酶的免疫反應性。
  9. There is a relationship between catenin phosphorylation, translocation and tumoregenesis, further more, in this relationship, cell signaling cascade and mitogen and their receptors are involved. base on these evidences, people are trying to disturb the key molecules in this signaling cascade for anti - tumor purpose

    表皮生長因子（ egf ）能使p120 ~ （ ctn ）酪氨酸磷酸化，磷酸化使bel - 7404細胞的粘附能力降低而遷移行為增強； p120 ~ （ ctn ）在細胞內的分佈出現明顯的核內轉位， -連環蛋白也出現相似的變化。
  10. In the day 4 afternoon and midnight, stat3 expression and nuclear translocation of tyrosine - phosphorylated stat3 became stronger in the glandular and luminal epithelium of the potential implantation sites

    第4天下午及午夜，胚胎將要著床處子宮的腔上皮和腺上皮中stat3表達繼續增強，酪氨酸磷酸化stat3的核轉位明顯增強。
  11. Gfral is an extracellular protein that is attached to the outer cell membrane by gpi - anchor and could not transducted signalling directly. according to the original model, a gdnf dimer first binds to either monomeric or dimeric gfral, and the gdnf - gfral complex then interacts with two ret molecules, thereby inducing their homodimerization and tyrosine autophosphorylation

    傳統觀點認為： gdnf首先與gfr 1結合形成gdnf - gfr 1復合物，此復合物再與ret結合形成三聚體復合物，引起ret的二聚化，並引起酪氨酸殘基自磷酸化，從而引起下游的信號轉導。
  12. Ngf binding to trka receptors results in receptor dimerization and kinase activation. recent evidence supports that not all extracellular subdomains are responsible for receptor activation. structure based drug design for neurotrophic agonists with small molecular weight relies on knowledge of the interaction of neurotrophin with their receptors

    Trka在ngf作用下發生二聚化，使胞內域中酪氨酸激酶區激活，從而使trka中酪氨酸自磷酸化，並進一步激活胞內信號轉導通路，從而實現神經營養信號傳遞。
  13. P120ctn is a more newly found catenin, studies revealed that it is also an important molecule in e - cadherin mediated cell - cell adhesion, and phosphorylated tyrosine in p120ctn were evidenced. translocation of p120ctn occurred in cancer cells and tissues. evidences showed that there were a key relationship between p120ctn translocation and tumoregenesis

    P120 ~ （ ctn ）是發現得較晚的一種連環蛋白，研究發現它也參與上皮鈣粘蛋白介導的細胞粘附過程並且不可缺少，同時p120 ~ （ ctn ）也和-連環蛋白一樣易於酪氨酸殘基磷酸化，磷酸化后的p120 ~ （ ctn ）也發生轉位，並與腫瘤的發生發展有密切關系。
  14. C - kit is a receptor tyrosine protein kinase on the cellular membrane, while c - myc is a transcription regulatory factor in the nucleus

    Kit是位於細胞膜上的受體酪氨酸蛋白激酶， myc是位於細胞核內的轉錄調節因子。
  15. The fluorescence spectrum ( fls ) of lra excited at 280nm and 295nm showed a maximum peak at 338nm. the characteristic peak of tyr did not exist, and it showed that the fluorescence energy of tyr was transformed to trp and strength the fluorescence of trp. when lra was excited at 295nm, the fls showed a maximum peak at 338nm, the max of fluorescence emission spectrum blue - shifted more than 10nm compared with the max of free tyr ( 348nm )

    Lra的熒光光譜研究表明在激發光波長為280nm時，其最大熒光發射峰在338nm處，熒光光譜未見有酪氨酸（ tyr ）殘基的發射峰，表明tyr殘基的熒光基本上通過能量轉移到trp上，使熒光強度增強，在激發光譜為295nm時，其最大熒光發射峰338nm ，比游離trp的最大熒光發射峰（ 348lun ）藍移了近10nln ，說明trp周圍的極性較弱，處于疏水的微環境。
  16. Ebv gp350 and c3d bind selectively to the same site on the cr2, epitopes on scr - 1 and scr - 2 are necessary for the binding. as soon as c3d binds to scr - 1 and scr - 2, cr2 may cross - link - 8 - to memberane ig and form a " co - capping " structure, moreover, activate b lymphocyte through cd19, a component of cd19 / cd21 / cd81 / leul3 complex, signaling and / or nucleolin tyrosine phosphorylation independent on cd 19 signaling pathway

    配體與scr l和scr 2結合后， crz與細胞膜免疫球蛋白（ inlg ）形成「共帽」 kofthofcccptor ）結構，並經過d19c 21兒81肚u13復合體中d19的信號轉導以及不依賴于cd途徑的核仁素（ nucleolin ）酪氨酸磷酸化激活b細胞。
  17. Sh2 domain can specifically recognize phosphated tyrosine residue, and mediate receptor tyrosine protein kinase signal transduction

    Shz結構域可以特異性識別磷酸化酪氨酸短序列，介導受體酪氨酸蛋白激酶信號轉? 2 ?導途徑。
  18. The members of src family are non - receptor tyrosine kinases. their make an important function in a complex network of intracellular signals, such as cell growth, proliferation, transformation, apoptosis, and cell adhesion, migration. the fact that src was discovered as an oncogene suggests that src could closely involvement in cancer

    而今， src已發展成為包括九個成員在內的激酶家族，隸屬于非受體酪氨酸激酶，是細胞信號轉導途徑中一類重要的信號分子，調控細胞的生長，增殖，凋亡，細胞黏附與遷移等一系列功能。
  19. Protein tyrosine kinases play an important role in signal transduction pathways in regulating a number of cellular functions

    蛋白質酪氨酸在一系列細胞活動的信號轉導途徑中起著重要的作用。
  20. The expression, tyrosine - phosphorylation and nuclear translocation of stat3 were increased in the luminal and glandular epithelium of immature mice by pmsg treatments. hcg treatment also stimulated the tyrosine - phosphorylation of stat3 in the nuclei of luminal epithelium

    用pmsg處理性未成熟的小鼠，可刺激子宮腔上皮和腺上皮中stat3的表達，並促進子宮腔上皮和腺上皮中的stat3發生酪氨酸磷酸化及核轉位。
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