鉀外流 的英文怎麼說

中文拼音 [jiǎwàiliú]
鉀外流 英文
potassium efflux
  • 鉀 : 名詞[化學] potassium （19號元素, 符號 k）
  • 外 : Ⅰ名詞1 （外面） outside; external side 2 （外國） foreign country 3 （以外） besides; beyond; in ...
  • 流 : Ⅰ動1 （液體移動; 流動） flow 2 （移動不定） drift; move; wander 3 （流傳; 傳播） spread 4 （向壞...
  1. When examined using some inhibitors with respect to orll receptor, the nociceptin - induced current was depressed in amplitude by nocistatin ( a putative nociceptive precursor product ) and also by compb ( a non - peptidyl orll receptor antagonist ) to a different extent without a change in holding currents of sg neurons

    Noosoo h口對傳向脊髓背角sg神經元的興奮性和抑制性突觸傳遞的影響在i己錄電極內液中加入gdp書s 、艷離子和tea （主要用於阻斷突觸后的g蛋白和鉀通道的作用）的狀況下， nociceptin誘致的外向電流可以完全被阻斷。
  2. After pretreated with nifedipine to block the influx of extracellular calcium, the vessels did not contract when they were stimulated by 60 mmol / l kc1 solution ; on the other hand, after pretreated with thapsigargin to deplete the intracellular calcium stores, the vessel remained the ability to contract when stimulated by 60 mmol / l kcl solution

    使用ifedipine預處理血管，阻斷細胞外鈣離子內流，將血管置於含60mmol lkci的高鉀環境中，血管不發生收縮。使用細胞內鈣耗竭劑thapisin呷in預處理血管，耗竭細胞肌漿網內儲存的鈣離子。
  3. Effects of microenvironmental change on transient outward potassium currents

    心臟微環境的改變對瞬時外向鉀電流的影響
  4. The results showed that they had no evident effect of outword delay - rectified potassium current, while they could inhibit ttx - s fast sodium current

    結果表明，三種蜘蛛粗毒對外向延遲整流鉀電流沒有明顯作用，但對ttx - s的快鈉電流表現出較強的抑制效應。
  5. Previous studies indicate that vascular endothelial growth factor ( vegf ) has direct neurotrophic action and prevents ischemic neuronal injury ; it also can inhibit the outward delayed - rectifier potassium currents ( / k ) in the neurons

    以往的研究結果顯示血管內皮生長因子（ vascularendothelialgrowthfactor ， vegf ）具有直接的神經營養及神經細胞保護效應，並能抑制神經元的外向延遲整流鉀電流（ outwarddelayed - rectifierpotassiumcurrent ， i _ k ） 。
  6. It has been demonstrated that kvl. 2, an isoform of delayed - rectifier potassium channel, is up - regulated in the ischemic neurons. besides, the activity of kvl. 2 is tyrosine phosphorylation dependent, phosphorylation of the tyrosine residue in the amino terminus of the channel by tyrosine kinases suppresses the channel current. therefore, we hypothesize that vegf could enhance the phosphorylation of kvl. 2, which results in an inhibition of / k

    在介導i _ k的鉀通道家族中， kv1 . 2是其中的一個亞型，研究報道它在缺血神經元中的表達是增高的；此外，它的活性與其酪氨酸磷酸化水平相關，在其氨基末端的酪氨酸殘基經酪氨酸激酶磷酸化后可顯著降低其介導的電流的幅度。
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