鼠大腦中動脈 的英文怎麼說
中文拼音 [shǔdànǎozhōngdòngmài]
鼠大腦中動脈
英文
mcao-
Permanent middle cerebral artery occlusion, pmcao
方法採用大鼠永久性大腦中動脈阻塞Effect of the antibody of brain - derived neurotrophic factor on the rat with ischemic injury from obstruction of middle cerebral artery
腦源性神經營養因子抗體對大腦中動脈阻塞大鼠缺血損傷的影響Time course of regional cerebral blood flow in middle cerebral artery occlusion model in rats
大鼠大腦中動脈局灶性腦缺血局部腦血流動態觀察Establishment and evaluation of the model of middle cerebral artery occlusion reperfusion in mice
小鼠大腦中動脈閉塞再灌注模型的建立和評價To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation
為了驗證上述假設，本文分別在整體和離體水平，採用大腦中動脈缺血（ middlecerebralarteryocclusion ， mcao ）和體外氧?糖剝奪（ hypoxiaandglucose - deprivation ， hgd ）缺血模型，首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況，觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化，然後通過外源性給予vegf蛋白，在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響，最後利用vegf反義脫氧寡核苷酸（ oligodeoxynucleotide ， odn ）特異阻斷內源性vegf蛋白的表達，觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用，以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。The present study determined if the potent and nonaromatizable androgen, dihydrotestosterone ( dht ), exacerbates ischemic damage in the male rat and alters postischemic gene expression after middle cerebral artery occlusion
新近研究對有效的未芳香化的雄激素，雙氫睪酮，是否會加劇雄性大鼠的缺血損傷並改變大腦中動脈閉塞后的缺血后基因表達進行了研究。Membrane potential properties in the smooth muscle cells of middle cerebral artery in stroke - prone spontaneously hypertensive rats
易卒中型自發性高血壓大鼠大腦中動脈血管平滑肌細胞膜電位的特性分享友人