caspase 中文意思是什麼

caspase 解釋
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  1. A study of caspase - 8 expression in bronchus asthma rat ' s myocardium

    8在支氣管哮喘大鼠心肌細胞中的表達
  2. Expression of caspase - 3 in myocardial cell of carbon disulfide poisoned mice

    3在二硫化碳染毒小鼠心肌細胞中的表達
  3. Smyth mj, krasovskis e, sutton vr, et al. the drug efflux pro tein, p - glyoprotein, additiionally protects drug - resistant tumor cells from multiple forms of caspase - dependenl apoptosis [ j ]. proc natl acad sci usa, 1998, 95 : 7024

    闞兵，羅鋒，雷松，等.用流式細胞術檢測腫瘤組織細胞多藥耐藥基因表達產物[ j ] .華西醫科大學學報， 1998 ， 29 （ 3 ） : 269
  4. Furthermore, k + ionophore valinomycin and k + channel opener diazoxide alone induced neuronal apoptosis and caspase - 3 activation. besides, katp channel blockers tolbutamide and glybenclamide also showed neuroprotective effects and decreased caspase - 3 activation after hypoxia / reoxygenation. while the role of katp channels in neuronal apoptosis is not clear

    為進一步證實鉀通道在缺氧復氧誘導的神經元凋亡中的作用，我們採用了鉀通道開通劑100pmdiazoxide及鉀離子載體50nmvallnomycln 。
  5. However, a - type channel blocker 4 - ap and channel blocker apamin displayed no effect on cell death and percentage of tunel positive cells and caspase - 3 activation

    通道阻斷劑叩amin門卜m ）對缺氧復氧后神經元死亡、 tunel陽性細胞比例以及caspascs激活均無顯著影響，表明bkc 。
  6. Adjustment of bushentongluo recipe to the caspase - 3 of the chondrocyte in osteoarthritis rats

    補腎通絡方對大鼠膝關節炎軟骨中半胱氨酸天冬氨酸特異性蛋白酶3的調節
  7. These data suggested that following hypoxia / reoxygenation, over - activity of bkca channels may mediate cultured hippocampal neuronal apoptosis induced by hypoxia / reoxygenation via activation of caspase - 3

    X現diazoxide或valinomycin均可誘導正常培養的神經元凋亡及激活caspase上。上述結果表明缺氧復氧可能引起bkc 。
  8. Tumor necrosis factor -, caspase - 3 expression and hepatocyte apoptosis in fulminanting hepatic failure

    3表達與暴發性肝衰竭細胞凋亡
  9. Labeling tunel method. the cell ultrastructural changes were similar to apoptosis in animal cells : the apical meristemetic cells underwent the programmed cell death. this was first detected in the apex cells of apical meristem, while peripheral cells differentiated gradually into different parts of a floral bud. but all the cells in the floral bud were subjected to the pcd process before it developed into a complete flower. 140bp dna fragment was found to deposit in apical bud during the plant development. the most important role of caspase - 8 was detected by western blot, and the expression of the procaspase - 8 was time - related with the dna frgmentation and the transformation from vegetative to the reproductive growth. these results suggested that pcd was an active process during the differentiation of apical meristem, and the senescence observed in the apical bud was due to the pcd process

    顯微超微結構研究表明，短日照條件下豌豆頂芽的衰老過程是從營養生長錐向花芽的轉化，而用dna原位末端標記tunel caspase - 8 western blot和140 bp dna片斷積累的試驗結果證明，轉化為花芽的整個生長錐細胞發生了編程性死亡pcd ，而且其最頂端部分細胞首先發生pcd ，而頂端周圍的分生組織細胞逐漸分化出花芽的各部分，但頂芽最後並沒有發育成為完整的花，所有細胞就都發生pcd ，從而頂芽衰老。
  10. Progress in research on roles of caspase inhibitor in prevention and treatment of hypoxic - ischemic brain damage

    半胱胺酸蛋白酶抑制劑防治缺血性腦損傷研究
  11. Several genes and cytokines take part in the accommodation of these changes and influence the severity of brain injury, such as tumor necrosis factor a, bel - 2 gene family and ced gene family ( cystein - dependent aspartate - specific protease, caspase ). they are all has relationship with cell death after tbi and control the different cascade of cell death. there are two kinds of cell death after traumatic brain injury ( tbi ) that is necrosis and apoptosis

    腦外傷后腦組織發生的這些改變由許多基因、細胞因子參與調節並影響著損傷程度，除腦水腫外，腦損傷程度主要取決于細胞因子，這些因子包括腫瘤壞死因子- （ tumornecrosisfactor - ， tnf - ） ， b細胞淋巴瘤白血病- 2 （ b - celllymphoma leukemia - 2 ， bcl - 2 ）基因中的bcl - 2 、 bax ， ced基因家族中的半胱天冬酶（ cystein - dependentaspartate - specificprotease ， caspase ）等，它們都與腦損傷后的神經細胞死亡有關，控制著引起細胞死亡的不同層面。
  12. Tissue sections from every animal were double - labeled with the antibodies of map - 2, cox - 2, gdnf, caspase - 3 and either the neuron - specific antibody neuronal nuclear protein ( neun ) or the astroglial - specific marker glial fibrillary acidic protein ( gfap ). we carried out a series of research to explore the effects and mechanism of map - 2, cox - 2, gdnf, caspase - 3 during tbi and trie d to provide some useful theory basis for both the treatment of tbi in the practice and forensic medicine

    並通過上述指標分別與神經元特異性標志物神經元核蛋白（ neuronalnuclearprotein ， neun ）和星形膠質細胞特異標志物膠質纖維酸性蛋白（ glialfibrillaryacidicprotein ， gfap ）進行免疫組織化學雙染色，探討腦損傷后神經元及神經膠質細胞反應性變化情況及其分子生物學機制，以期為腦損傷研究提供有益的數據材料，也為以上指標在法醫學實際檢案的應用提供必需理論依據。
  13. Caspase - 3 mrna expression in cerebral cortex and hippocampus of rats with lymphostatic encephalopathy

    在淋巴滯留性腦病大鼠腦皮質及海馬的表達
  14. Influence of ciliary neurotrophic factor on spinal cord caspase - 3 expression in rats of different age following sciatic nerve damage

    不同年齡大鼠坐骨神經損傷后睫狀神經營養因子對脊髓組織中天冬氨酸特異酶切的半胱氨酸蛋白酶3表達的影響
  15. Functions of caspase - 3 in repair of acute cerebral and spinal injuries

    半胱氨酸天冬氨酸蛋白酶3在急性腦及脊髓損傷修復中的作用
  16. Effects of electro - acupuncture on expressions of fas and caspase - 3 in rats with acute spinal cord injury

    和半胱氨酸天冬氨酸蛋白酶3表達的影響
  17. Influence of caspase - 3 on hippocampal neuronal apoptosis following ischemia - reperfusional injury in rats

    半胱氨酸天冬氨酸蛋白酶3對缺血再灌注大鼠腦海馬神經元凋亡的影響
  18. Neuronal apoptosis and caspase 3 gene expression of in vitro cultured rat hippocampal neurons of epilepsy models

    體外培養大鼠海馬神經元癲癇模型中神經元凋亡及半胱氨酸天冬氨酸蛋白酶3基因的表達
  19. Tea treatment also reduced the number of tunel positive cells and caspase - 3 activation

    500pmtea還可減少tunel陽性細胞及caspase工激活。
  20. Then several kinds of selective k + channel blockers were applied to further clarify the specific type of k + channels involved in apoptosis. both bkca channel blockers, ibtx and chtx ( at the concentration of 100 nm ), significantly reduced neuronal death and number of positive tunel - staining cells and inhibited caspase - 3 activation

    通道阻斷劑100nmibtx或100nmchtx均有顯著的神經元保護作用，可減少tunel陽性細胞比例及抑制caspase上激活；然而， a型鉀通道阻斷劑4apo0qm ）或skc 。
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