endotoxin shock 中文意思是什麼
endotoxin shock
解釋
內毒素休克-
Endotoxic shock ( es ), resulting from cascade reaction of inflammatory mediators induced by excessive endotoxin, leads to uncontrolled disturbance of organisms, multiple organ failure and lethality
內毒素休克（ endotoxicshock ， es ）時由於大量內毒素（ endotoxin ）誘導產生多種炎癥介質的級聯反應，使機體處於一種自身難以控制的紊亂狀態，可發生多器官功能障礙綜合征而最終導致死亡。 -
Endotoxin shock is a common and severe pathological process with high mortality, and has been one of the most important topics in biomedicine
內毒素休克是臨床常見的休克類型之一，死亡率極高，一直是醫學研究的重點課題。 -
The disorders of vascular regulations is characteristic of endotoxin shock with the manifestations of marked reduction in systemic mean arterial pressure whereas pulmonary arterial hypertension, as well as abnormal changes of vascular reactivities of systemic and pulmonary arteries at its initial stage
血管調節機制紊亂是內毒素休克特徵性病理改變之一，主要表現為內毒素休克發生早期主動脈壓下降、肺動脈壓升高以及離體肺動脈及主動脈血管反應異常變化。 -
No, a first gas information molecule discovered in human being, is a typical endothelial - derived relaxant and mediates endothelium - dependent relaxation of blood vessels. in the pathogesis of endotoxin shock vec is one of the major target cells of lps and lps - induced proinflammatory cytokine such as tumor necrosis factor and interlukin 1 and activated. in vec inducible nitric oxide synthase ( inos ) is induced and lead to an increase in production of no, the while endothelial nitric oxide synthase ( enos ) is inhibited and elicit decrease in no formation, both of which are demonstrated to induce the
在內毒素休克過程中vec是lps及其誘導機體產生的多種促炎細胞因子如tnf 、 il - 1作用的主要靶細胞， vec誘導型一氧化氮合酶（ induciblenitricoxidesynthase ， inos ）激活、 no大量誘生而內皮型一氧化氮合酶（ endothelialnitricoxidesynthase ， enos ）活性被抑制、 no生成障礙，是血管反應性異常變化、血管調節機制紊亂的重要發病環節。
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