inos 中文意思是什麼
inos
解釋
伊諾什-
The data we obtained show that in resting macrophages, the basal levels of inos activity and no production are relatively low ; nevertheless, inos activity and no production can be significantly induced in response to oligochitosan stimulation. oligochitosan ( 80 ( ug / ml ) significantly induced the release of no 12 h after incubation, and the amount of no increased with time
試驗結果表明:用80林留ml劑量的殼寡糖作用於raw264 . 7細胞（ 4xl護個細胞/ m1 ） 12h便可以明顯誘導no的生成，隨著刺激時間的延長， no的產量不斷增加，但增加的幅度逐漸變小。 -
No, a first gas information molecule discovered in human being, is a typical endothelial - derived relaxant and mediates endothelium - dependent relaxation of blood vessels. in the pathogesis of endotoxin shock vec is one of the major target cells of lps and lps - induced proinflammatory cytokine such as tumor necrosis factor and interlukin 1 and activated. in vec inducible nitric oxide synthase ( inos ) is induced and lead to an increase in production of no, the while endothelial nitric oxide synthase ( enos ) is inhibited and elicit decrease in no formation, both of which are demonstrated to induce the
在內毒素休克過程中vec是lps及其誘導機體產生的多種促炎細胞因子如tnf 、 il - 1作用的主要靶細胞， vec誘導型一氧化氮合酶（ induciblenitricoxidesynthase ， inos ）激活、 no大量誘生而內皮型一氧化氮合酶（ endothelialnitricoxidesynthase ， enos ）活性被抑制、 no生成障礙，是血管反應性異常變化、血管調節機制紊亂的重要發病環節。 -
The activity of inducible nitric oxide synthase ( inos ) was determined using inos assay kit
用誘導型一氧化氮合酶（ inos ）檢測試劑盒測定胞質內inos的活性。 -
Interaction between ho - 1 co and inos no in acute lung injury induced by intestinal ischemia - reperfusion in rats
白細胞介素11預防大鼠腸道缺血性損傷的機制探討 -
This article reviews the development on studies of the structure, function and catalytic pathway of nitric oxide synthase ( nos ). the structure - function relationship and oxygenation mechanism of the inos were discussed
摘要綜述了一氧化氮合酶的結構、功能和催化路徑，討論了誘導型一氧化氮合酶的結構和功能的關系以及加氧氧化的機理。 -
Many antibodies against pro - inflammatory cytokines and inhibitors of inducible no synthase ( inos ) have been produced, but most of them are agents targeting one factor and ca n ' t efficiently block the development of systemic inflammation
盡管已經研製出許多抗促炎細胞因子抗體及誘生型no合酶（ induciblenosynthase ， inos ）抑制劑等，但多為單因素抗炎制劑，不能有效地阻斷全身性炎癥的發展。
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