insulin secretion 中文意思是什麼
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Effects of repeated intake of large amounts of sucrose or maltose on insulin secretion in healthy adults
重復大量攝入蔗糖與麥芽糖對健康成人胰島功能的影響 -
Physiological functions of insulin ; regulation of adenohypophysis hormones secretion ; mechanisms of hormone action ; regulation of thyroid hormones secretion
胰島素的主要生理作用;腺垂體激素分泌的調節;激素作用的機制；甲狀腺激素及糖皮質激素分泌的調節。 -
Shengyuan maintenance blood - sugar capsule is chooses the cactus, the balsam pear, the black bee bee glue, the kudzu root four pelts the sugar ingredient reasonably to blend, purifies the highly effective multi - skill health product by the modern biotechnology extract which but becomes, has the bidirectional adjustment, the control blood sugar, the activation and the nutrition island of langerhans beta cell, the promotion endogenous secretions insulin secretion, enhances the human body sugar metabolism and the fat metabolism ability, strengthens the pancreas function the function, at the same time also has the repair diabetes liver, the kidney damage, the prevention and the improvement diabetes illness complication function
聖源維糖軟膠囊是選擇仙人掌、苦瓜、黑蜂蜂膠、葛根四大降糖成份合理配伍，以現代生物科技萃取精製而成的高效復合型健康產品，具有雙向調節、控制血糖，激活和營養胰島細胞，促進內源性胰島素分泌，提高人體糖代謝和脂肪代謝能力，增強胰臟功能的作用，同時又具有修復糖尿病肝、腎損傷，預防和改善糖尿病並發癥的作用。 -
Purposes : enhancement of insulin secretion from the islet b cells is a principal goal for treatment of patients with type 2 diabetes. some gastrointestinal hormones are associated with insulin secretion
目的：胰高血糖素樣肽- 1 （ glucagon - likepeptide - 1 ， glp - 1 ）是一種組織特異性的胃腸道激素，主要由回腸末端l細胞、胰腺內分泌部a細胞以及神經細胞合成和分泌。 -
The effects of insulin on expression and secretion of tumor necrosis factor - protein of human peripheral blood mononuclear cells and the intervention effects of pravastatin
蛋白表達及分泌的影響以及普伐他汀的干預作用 -
The effects of glp - 1 ( 7 - 36 ) nh2 on insulin secretion glp - 1 ( 7 - 36 ) nh2 with the concentration of 2. 5nmol / l, 5. 0nmol / l, l0. 0nmol / l, 20. 0nmol / l, 40. 0nmol / l respectively were added to the medium as different experimental groups, 24 hours later, insulin amount are 68. 76 ? 1. 71 72. 30 ? 3. 13 104. 16 ? 5. 57 110. 98 ?. 29 111. 58 ? 0. 65miu / l respectively, and the insulin account is 55. 53 ?. 63miu / lin the control group. there was no significant difference between the groups with 2. 5nmol / land 5. 0nmol / l glp - 1 ( 7 - 36 ) nh2 respectively ; and there was not significant difference among the groups with lo. onmol / l, 20. 0nmol / l and 40. 0nmol / l glp - 1 ( 7 - 36 ) nh2 respectively. but the difference is significant between experimental groups and control group ( p < 0. 05 ). the data show that with the rising concentration of glp - 1 ( 7 - 36 ) nh2, there is an increasing amount of insulin
對照組培養液中不含g廿一1 （ 7一36 ） nhz ，實驗組培養液中含有20nmol / lglp一1 （ 7一36 ） nhz ，培養24h后，用0 . 25 %胰蛋白酶消化胰島分散細胞，塗片后利用針對胰島素mrna的寡核甘酸探針進行細胞原位雜交， dab顯色，高清晰度病理圖文分析系統（ highpathologiealimageanalysissystem ， hp認s ）對細胞著色的平均光密度（ mean即tiealdensity ， mod ）量化分析，觀察實驗組和對照組胰島素mrna的表達情況。 -
This is due to excessive growth of their pancreatic islet cells that results in uncontrolled insulin secretion
潛在之原因是胰島細胞過度增生導致胰島素過量分泌從而引發低血糖。 -
Conclusions : we conclude that two years of treatment of elderly men with testosterone does not improve carbohydrate tolerance nor does it alter insulin secretion, insulin action, glucose effectiveness, hepatic insulin clearance or the pattern of postprandial glucose metabolism
結論： 2年睪酮激素替代治療不能改善老年男性的糖耐量，也不能改善胰島素分泌胰島素功能、葡萄糖自身代謝效能、胰島素在肝內清除及餐后血糖代謝。 -
The following research areas will be involved in the project : ( 1 ) mechanism of hereditic factors and its function in chinese type 2 diabetes ; ( 2 ) metabolomics on the cell stress and its nutrition intervention in the type 2 diabetes ; ( 3 ) mechanisms of metabolic nuclear receptors in the process of insulin resistance and type 2 diabetes onset ; ( 4 ) roles of signal transduction molecules, lipid metabolic disorder to insulin resistance ; ( 5 ) type 2 diabetes development and role of pathological changes of blood vessels ; ( 6 ) abnormal insulin secretion, apoptosis and type 2 diabetes ; ( 7 ) function of molecular network of type 2 diabetes progression
本項目擬開展以下研究工作： （ 1 ）中國人2型糖尿病發生過程中遺傳因素的發現及其作用機制； （ 2 ）細胞應激在中國人2型糖尿病發生過程中的作用機制及其營養干預的代謝組學； （ 3 ）代謝性核受體在胰島素抵抗和2型糖尿病發病中的作用機制； （ 4 ）細胞信號轉導分子、脂代謝紊亂與胰島素抵抗的關系； （ 5 ） 2型糖尿病發展過程與血管病變； （ 6 ）胰島素分泌異常、細胞凋亡與2型糖尿病； （ 7 ） 2型糖尿病發生過程中的分子網路與作用機制。 -
Scientists found that glucose levels were lower and the secretion of insulin better in the group that had taken anakinra
科學家發現服用阿那白滯素的實驗組患者葡萄糖水平降低，胰島素分泌增多。 -
The effect of ca2 channel bloker on insulin secretion in rat pancreatic islet cells
鈣通道拮抗劑對大鼠胰島細胞胰島素分泌的影響 -
Reducing glycemic load may be especially important to achieve weight loss among individuals with high insulin secretion
在高胰島素分泌的個體中，減低的糖負荷可能對減輕體重特別重要。 -
Regardless of insulin secretion, a low ? glycemic load diet has beneficial effects on high - density lipoprotein cholesterol and triglyceride concentrations but not on low - density lipoprotein cholesterol concentration
而不顧胰島素分泌，低糖飲食對高密度脂蛋白膽固醇和甘油三酯濃度有有利的效應，但是對低密度脂蛋白膽固醇濃度則無。 -
New york ( reuters health ) aug 09 - osteocalcin, a protein produced by bone cells, influences beta - cell proliferation, insulin secretion, and insulin sensitivity in mice, according to a report in the august 10th issue of cell
紐約（路透社健康新聞） 8月9日報道? ? 《細胞》雜志8月10日刊中有論文稱，骨細胞產生的一種蛋白骨鈣素可影響小鼠細胞增生、胰島素分泌及胰島素敏感性。 -
In type i or insulin - dependent diabetes iddm, little or no insulin is produced. in type ii or non - insulin - dependent diabetes niddm, there is reduced insulin secretion and the body cells do not react to insulin
在糖尿病i型或胰島素依賴型糖尿病中，只有少量或完全沒有胰島素分泌在糖尿病ii型或非胰島素依賴型糖尿病中，胰島素的分泌會減少，而身體的細胞亦減低對胰島素的反應。 -
Emerging knowledge about the key mechanisms, such as the impairment of glucose - stimulated insulin secretion and lipotoxicity and the role of muscle resistance to insulin secretion has become the focus of drug therapy
隨著分子生物學研究的開展及對2型糖尿病發病關鍵機制的認識加深：葡萄糖依賴的胰島素分泌功能受損、脂毒性、肌肉組織的胰島素抵抗逐漸成為藥物治療的靶點。 -
Diabetes is the chronic disease due to insufficiency of pancreatic insulin secretion or poor tolerance of insulin, which would cause hyperglycemia or urine sugar via the decreasing of utilization ratio of glucose within food
摘要糖尿病是由於胰臟分泌的胰島素量不足或作用不好，使體內新陳代謝發生障礙而引起的慢性疾病，由於病人對食物中醣類的利用率減低，而造成血糖過高或尿中有糖的現象。 -
Reductions in the amount of those by - products would presumably limit insulin secretion and thereby minimize insulin ' s unwanted actions in the body
減少葡萄糖代謝產物的產生，想當然耳會限制胰島素的分泌，因而將胰島素對身體的不良作用降到最低。 -
Adipokines, the bioactive factors derived mainly from adipocytes, regulate pancreatic - cell function including insulin secretion, gene expression and apoptosis. in this review, we propose that adipokines influence - cell function through three interdependent pathways. the first is through regulating lipid and glucose metabolism in - cells. the second implicates the change of ion channel opening and closing in - cells. the third pathway is via the modification of insulin sensitivity of - cells. the endocrine function of adipocytes is dynamic, and the secretion of various adipokines changes under different metabolic conditions. during the progression from the normal state to obesity and to type 2 diabetes, adipokines contribute to the occurrence and development of - cell dysfunction in type 2 diabetes
脂肪因子包括脂肪細胞分泌的多種活性因子，它們通過內分泌方式調節胰島細胞的胰島素分泌、基因表達以及細胞凋亡等多方面的功能.本文提出脂肪因子影響胰島細胞功能主要通過三條相互聯系的途徑而實現.第一是調節細胞內葡萄糖和脂肪的代謝;第二是影響細胞離子通道的活性;第三是改變細胞本身的胰島素敏感性.脂肪細胞的內分泌功能是一個動態過程，在不同的代謝狀態下，各脂肪因子的分泌發生不同變化.從正常代謝狀態發展到肥胖以及2型糖尿病的過程中，脂肪因子參與了胰島細胞功能障礙的發生與發展 -
Abstract : adipokines, the bioactive factors derived mainly from adipocytes, regulate pancreatic - cell function including insulin secretion, gene expression and apoptosis. in this review, we propose that adipokines influence - cell function through three interdependent pathways. the first is through regulating lipid and glucose metabolism in - cells. the second implicates the change of ion channel opening and closing in - cells. the third pathway is via the modification of insulin sensitivity of - cells. the endocrine function of adipocytes is dynamic, and the secretion of various adipokines changes under different metabolic conditions. during the progression from the normal state to obesity and to type 2 diabetes, adipokines contribute to the occurrence and development of - cell dysfunction in type 2 diabetes
文摘:脂肪因子包括脂肪細胞分泌的多種活性因子，它們通過內分泌方式調節胰島細胞的胰島素分泌、基因表達以及細胞凋亡等多方面的功能.本文提出脂肪因子影響胰島細胞功能主要通過三條相互聯系的途徑而實現.第一是調節細胞內葡萄糖和脂肪的代謝;第二是影響細胞離子通道的活性;第三是改變細胞本身的胰島素敏感性.脂肪細胞的內分泌功能是一個動態過程，在不同的代謝狀態下，各脂肪因子的分泌發生不同變化.從正常代謝狀態發展到肥胖以及2型糖尿病的過程中，脂肪因子參與了胰島細胞功能障礙的發生與發展
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