jnk 中文意思是什麼

jnk 解釋
c-jun氨基端激酶
  1. A study on apoptosis and activity of sapk jnk in adrenocortical cells induced by cadmium chloride

    氯化鎘誘發腎上腺皮質細胞凋亡與應激活化蛋白激酶活性的研究
  2. Comprehensive cellular responses was found in human amnion fl cells following exposure to low concentration of mnng, such as the lowering of dna replication fidelity resulted from alteration of dna polymerase profile ; activation of a lot of transcription factors, such as api, creb, nf - kb etc ; clustering of egfr ( epidermal growth factor receptor ) and tnfr ( tumor necrosis factor receptor ) and activation of camp - pka - creb and jnk / sapk signal pathways

    我們發現，低劑量mnng處理后的人羊膜fl細胞有廣泛的細胞反應，並有多個信號轉導通路的激活和基因表達的改變。例如dna復制保真度下降， dna聚合酶譜發生改變，應用報告基因技術和底物磷酸化檢出技術證明細胞一系列轉錄因子如ap1 、 creb 、 nf b等被激活，細胞表面受體如表皮生長因子受體、腫瘤壞死因子受體發生聚簇，細胞信號轉導通路camp - pka - creb和jnk sapk被激活。
  3. For example, we have found the clustering of egfr ( epidermic growth factor receptor ) and tnfr ( tumor necrosis factor receptor ) and the activation of camp - pka - creb and jnk / sapk pathways after mnng treatment

    例如細胞表面受體如表皮生長因子受體、腫瘤壞死因子受體發生聚簇，細胞信號轉導通路camp kacgyb和jnk sapk被激活。
  4. 2. the proapoptotic effects result from the reduction of erk, activation of jnk during treatment of bfgf and p38 also has some role in this process

    Bfgf作用於u2 os細胞引起的erk活性抑制和jnk長時程激活共同介導了u2 os細胞凋亡的發生， p38也在一定程度上參與了這個過程。
  5. They are pkc, pka, mek1 in erk pathway, mekk1 in sapk / jnk, p38 in p38 pathway, the three latter pathways all belong to the map kiiiase pathway. the inhibition of pkc decreases the proliferation after the il - 16 treatments, which hint its role in proliferation signal transduction

    結果表明，加入pkc的抑制劑，強烈抑制了所有濃度16處理下細胞的增殖，暗示pkc在u一16與cd4結合后，可能傳遞了增殖相關信號，它的存在是jllrk盯細胞的生長增殖必須的。
  6. Pka, receptor tyrosine kinase ( trk ) and classical nuclear receptor of gc were not involved in the gc " s activation of mapks the second part studied the nuclear translocation of gc activated mapks, mainly p38 and jnk, with laser confocal microscopy. the results showed that : 1

    Gc激活的mapks的激活不需要pka酪氨酸激酶受體trk及經典gc核受體的參與第二部分是研究gc激活的mapks的核轉位，主要是p38和jnk ，用激光共聚焦顯微鏡觀察到以下結果： 1
  7. The activated p44 / 42 decreased to basal level rapidly while the phosphor - p38 and jnk sustained at high level until 4hr. 3

    P44 42在快速被激活后很快降到基礎水平，而p38和jnk則持續激活可達4小時3
  8. Western blot : the expression of jnk was obviously stronger in psd group than in the two control groups. the jnk activity had increased 75 % than hc group and 62. 5 % than lc group respectively

    C jun免疫組化觀察：免疫組化c jun廠性結果為胞核著色，呈黃色或棕黃色顆粒，細胞漿內未見著色。
  9. Effects of sodium tanshinone a sulfonate on angiotensin - induced cardiomyocyte hypertrophy and expressions of phosphorylated jnk and mitogen - activated protein kinase phosphatase

    和絲裂原活化蛋白激酶磷酸酶1的表達
  10. Induction of apoptosis and change of the activity of sapk jnk in rat cortical neurons treated by aluminum chloride

    鋁致大鼠皮層神經元凋亡及應激活化蛋白激酶活性的變化
  11. Therefore, this project intend to explore mechanism of apoptosis mediated by protein tyrosine kinase ( ptk ) pathway by study of the role of erk1 / 2, jnk and p38 in apoptosis of u 2 - os cell line induced by bfgf

    本課題欲通過研究erk1 2 、 jnk 、 p38在bfgf誘導u2 - os細胞凋亡中的作用來探討酪氨酸蛋白激酶途徑參與細胞凋亡調控的作用機制。
  12. Protective effect of jnk inhibitor cep - 11004 on the alcl3 - induced apoptosis in cortical neurons of rat pups

    11004對鋁誘導的大鼠皮層神經元凋亡的保護作用
  13. The effects of jnk rna interference on 293 cell apoptosis induced by cadmium

    對鎘致293細胞凋亡的影響
  14. The p38 and jnk tanslocated into nucleus at 30min after 15min of b stimulation 2

    在b15分鐘的作用下，於30分鐘后p38和jnk轉位入核2
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