ldh-l 中文意思是什麼

ldh-l 解釋
乳酸脫氫酶l
  • ldh : 乳酸脫氫酶
  • l : L-beam, L-iron 不等邊角鋼。L,￡＝ 〈拉丁語〉 libra （＝ pound）。
  1. Coal ldh ( ai : co = l : 2 ) as electrode has a stable structure and good cyclic performance. specific capacitance of its single electrode attains 400f / g

    Ai雙氫氧化物結構較穩定，循環性能較好，單電極比容量達到400fig 。
  2. Mechanism of glutamate - induced inhibition of pulmonary surfactant lipid synthesis ? mda receptor ( nmdar ) antagonist mk - 801 could reverse glutamate - induced inhibition of [ 3h ] - choline incorporation in lung explants ; ? he expression of nmdar1 subtype existed in alveolar type ii epithelial cells ; ? he effect of glu on pc synthesis required l - arginine in cultured lung explants, nos activity and no production were increased significantly in lung explants with glu treatment ; and mk - 801 and nos inhibitor l - nna could block the increase of nos activity and no production induced by glu ; l - nna also could reverse the inhibition of [ 3h ] - choline incorporation induced by glu ; ( 4 ) mda content increased and sod level decreased in cultured lung explants of glu treament groups, mk - 801 and l - nna could reverse these effects ; ( 3 ) mk - 801, l - nna could block the release of ldh induced by glu

    谷氨酸對肺表面活性物質脂質合成抑制的機制研究nmda受體拮抗劑mk - 801能逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應；肺泡上皮細胞有nmda受體nmdar1亞單位的表達； glu對ps脂質合成的影響依賴l -精氨酸的存在， glu可使肺組織勻漿中nos活性、 no合成量升高； mk - 801 、 nos的抑制劑l - nna均可阻斷glu所致的這些效應； l - nna可逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應； glu可使肺組織勻漿中mda碩士學位論文中文摘要含量增高、 sod水平下降。 mk七0 、 l nna能阻斷q所致的這些效應； mk80 、 l nna還能阻斷gill誘導的ldh釋放增多的效應。
  3. ( 3 ) there was only ldh5 in the livers of al l groups of microtus fortis. the ldh4 and ldh5 were absent in the kidneys of the small voles from heilongjiang, but five kinds of ldh isozymes were all found in the kidneys of the other three groups

    （ 3 ）四類東方田鼠的肝臟中都只含ldh5 ；腎臟中，除黑龍江小體型東方田鼠缺乏ldh4和ldh5外，其餘三類東方田鼠都含五種ldh同工酶。
  4. Methods : in cultured lung explants without serum, the lipid component synthesis of pulmonary surfactant was evaluated in [ 3h ] - choline incorporation ; mrna content of phosphocholine cytidylyltransferase ( cct ) in lung explants was investigated in rt - pcr ; the changes of the ultrastructure of the at ii cells were observed with electron microscope ; the expression of nmdar1 subtype was observed in immunohistochemistry staining ; nitric oxide synthase ( nos ) activity, nitric oxide ( no ) content, superoxide dismutase ( sod ) level, malondialdehyde ( mda ) content and lactae dehydroase ( ldh ) level were determined by biochemistry methods. results : 1. influence of glutamate on synthesis of the lipid component of pulmonary surfactant ? with l - arginine, glu inhibited [ 3h ] - choline incorporation with good dose - dependence and time - dependence ; ( 2 ) mrna content of cct of the glu treatment groups was decreased ; ( 3 ) glu increases the release of ldh in cultured lung explants ; ( dwith electron microscope histochemistry, glu induced the changes of the ultrastruture of at ii iv cells

    方法：採用成年大鼠肺組織無血清培養，運用[ ~ 3h ] -膽堿摻入法測定ps主要脂質磷脂酰膽堿（ pc ）合成量； rt - pcr擴增檢測肺組織中pc合成限速酶磷酸膽堿二胞苷酰基轉移酶（ cct ） mrna含量；透射電子顯微鏡法觀察肺泡型上皮細胞和ps系統超微結構的變化；免疫組織化學染色檢測glu的受體nmdar1亞單位的表達；生化測定肺組織乳酸脫氫酶（ ldh ）釋放量和肺組織勻漿中一氧化氮合酶（ nos ）活性、一氧化氮（ no ）生成量、超氧化物歧化酶（ sod ）水平以及丙二醛（ mda ）含量。
分享友人
分享到 Line

分享到臉書