middle cerebral 中文意思是什麼

middle cerebral 解釋
大腦中動脈
  • middle : n 1 中央,正中;中間,中部;中途。2 人體的中部,腰部。3 中間物,媒介物;中人,中間人,調解人;中...
  • cerebral : adj. 1. 大腦的,腦的。2. (文藝等)觸動理智的;理智方面的;非感情方面的。
  1. Thromboemboli can lodge in cerebral arteries, particularly in the distribution of the middle cerebral, and peripherally toward branch points

    血栓栓子可以栓塞于腦動脈,尤其是在大腦中動脈及其分支。
  2. Permanent middle cerebral artery occlusion, pmcao

    方法採用大鼠永久性大腦中動脈阻塞
  3. Of these, 6 underwent surgical revascularization, including superficial temporal artery to middle cerebral artery anastomosis and / or pial synangiosis

    其中, 6例行外科血管成形術,包括淺表顳動脈到中腦動脈吻合和或頸部吻合。
  4. Objective to report 16 cases of intracranial aneurysms with calcified vascular wall and study the relationship between vascular calcification and pathogenic mechanism of intracranial aneurysms. methods spiral ct scan, clinical data and serum calcium, phosphate, glucose, lipid, renal function of the 16 patients were studied in retrospect. all aneurysms were clipped, in which 8 were resected after clipping. results the location of aneurysms with aneurismal wall calcification was, on posterior communicating artery in 3, on middle cerebral artery in 2, on anterior communicating artery in 2, on basilar artery in 5 and on basilar artery in 4. all patients showed normal serum calcium, phosphate, glucose and normal renal function. serum lipid of patients had been elevated. all patients recovered with no significant neurological deficits. conclusion intracranial vascular calcification is an active and regulated process in close relation to atherosclerosis, serum calcium, phosphate, lipid, thyroid, parathyroid function and other factors. intracranial vascular calcification has close relationship with the pathogenesis and management of intracranial aneurysms

    目的報告16例瘤壁有鈣化的顱內動脈瘤病例的治療,分析顱內血管鈣化與顱內動脈瘤病理發生機制的關系.方法回顧性分析16例瘤壁有鈣化的顱內動脈瘤病例的影像學、臨床資料及手術治療.結果瘤壁有鈣化的顱內動脈瘤的位置是: 3例位於后交通動脈, 2例位於大腦中動脈, 2例位於前交通動脈, 5例位於椎動脈, 4例位於基底動脈.所有患者血鈣、磷酸鹽、血糖、腎功能正常. 6例患者血脂增高. 16例患者均行動脈瘤夾閉術,其中8例術中切除動脈瘤,治療效果良好.結論動脈瘤瘤壁鈣化與其病理發生機制有關,常見于巨大或大動脈瘤,瘤壁血管鈣化是與動脈粥樣硬化、血鈣、磷酸鹽、血脂、甲狀腺、甲狀旁腺等因素有關的主動耗能,多因素調控的病理過程
  5. Effect of the antibody of brain - derived neurotrophic factor on the rat with ischemic injury from obstruction of middle cerebral artery

    腦源性神經營養因子抗體對大腦中動脈阻塞大鼠缺血損傷的影響
  6. We describe a 57 - year - old man who presented with simultaneous large middle cerebral artery territory infarction and posterior cerebral artery territory infarction confirmed by dwi

    我們報告一大范圍的中大腦動脈及后大腦動脈同時發生腦梗塞,並經由擴散磁振造影確認的病例。
  7. In this slice, the lesion extends from near the occipital pole to the sylvian fissure, conforming to the territory of the posterior branch of the middle cerebral artery

    在這一層,損害區從靠近枕骨粗隆延伸至至大腦外側裂,與大腦中動脈后支支配的范圍相吻合。
  8. We sought to determine the accuracy of serial national institutes of health stroke scale ( nihss ) scores to detect complete early recanalization of the middle cerebral artery

    我們設法確定一系列美國國立衛生研究院的中風尺度( nihss )的準確度,以便發現大腦中動脈的早期再通情況。
  9. Time course of regional cerebral blood flow in middle cerebral artery occlusion model in rats

    大鼠大腦中動脈局灶性腦缺血局部腦血流動態觀察
  10. Establishment and evaluation of the model of middle cerebral artery occlusion reperfusion in mice

    小鼠大腦中動脈閉塞再灌注模型的建立和評價
  11. Correlation between degree of middle cerebral artery stenosis and cerebral blood flow with 64 - slice spiral ct

    分析癥狀性大腦中動脈狹窄分級與腦血流灌注的相關性
  12. Among asian patients with vascular risk factors, middle cerebral artery ( mca ) stenosis is frequently present, even in the absence of a history of stroke or transient ischemic attack, investigators report in the june issue of neurology

    有血管危險因素的亞洲病人中,經常存在大腦中動脈狹窄,即使在那些沒有卒中史或短暫腦缺血發作史的病人也是如此,這是研究人員在神經學雜志六月刊中報道的
  13. Thomas gn, lin jw, lam wwm, et al. increasing severity of cardiovascular risk factors with increasing middle cerebral artery stenotic involvement in type 2 diabetic chinese patients with asymptomatic cerebrovascular disease j. diabetes care, 2004, 27 : 1121

    王玉祥,李志群,孫向東. 2型糖尿病合併腦梗死患者頸動脈、血脂及血漿纖維蛋白原的變化j .臨床神經病學雜志, 2005 , 18 : 263
  14. Surgical chance of operation in decompressive craniectomy in malignant middle cerebral artery syndrome

    惡性大腦中動脈綜合征的手術治療時機
  15. Comparative proteomic analysis of cerebral cortex between middle cerebral artery occlusion rats and normal controls

    正常與腦缺血大鼠的腦皮質蛋白質差異分析鑒定
  16. Experiment about single - strand dna breaks in permanent middle cerebral artery occlusion of rats

    單鏈斷裂損傷的實驗研究
  17. To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation

    為了驗證上述假設,本文分別在整體和離體水平,採用大腦中動脈缺血( middlecerebralarteryocclusion , mcao )和體外氧?糖剝奪( hypoxiaandglucose - deprivation , hgd )缺血模型,首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況,觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化,然後通過外源性給予vegf蛋白,在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響,最後利用vegf反義脫氧寡核苷酸( oligodeoxynucleotide , odn )特異阻斷內源性vegf蛋白的表達,觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用,以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。
  18. Middle cerebral artery occlusion, mcao

    大腦中動脈閉塞
  19. Effect of electroacupuncture on bdnf expression at ischemia cortex and infarct volume after middle cerebral artery occlusion in rat

    累加電針對腦缺血大鼠皮層腦源性神經營養因子表達及腦梗塞體積的影響
  20. The present study determined if the potent and nonaromatizable androgen, dihydrotestosterone ( dht ), exacerbates ischemic damage in the male rat and alters postischemic gene expression after middle cerebral artery occlusion

    新近研究對有效的未芳香化的雄激素,雙氫睪酮,是否會加劇雄性大鼠的缺血損傷並改變大腦中動脈閉塞后的缺血后基因表達進行了研究。
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