neuroprotection 中文意思是什麼

The neuroprotection of ketamine and midazolam in a rat model of transient forebrain cerebral ischemia

氯胺酮和咪唑安定對全腦缺血大鼠的腦保護作用

Neuroprotection of tea on neuronal damage in ca1 region of hippocampus after global ischemia. neurosci lett

有關，為細胞凋亡參與神經元遲發性死亡提供了新的機制。

His research interests centre on neural plasticity. recent projects focus on developmental establishment of space - centered coordinates in relation to auditory - vestibular cues as well as on strategies to stimulate neuroprotection and repair of the adult nervous system

在學術研究方面，對中樞神經細胞處理從內耳傳入的空間定向信息進行了廣泛的研究工作，並進一步探討該類神經細胞在動物幼年發育臨界期前後可塑性的變化。

Using this system, we have studied matrine - inhibittory effect and trifluoperation - neuroprotection effect in hippocampal slices, also discussed the mechanism of long - term potentiation using anesthetic rats. the experiment results showed that matrine can inhibit the hyperactivity induced by penicillin sodium in dosage by changing the relative parameters of field potential ; trifluoperation can alter ps change with the time, enhance the degree and the ratio of ps recovery, then minis the hypoxic injury ; high frequency stimulate can increase ps amplitude and epsp slope for long time, buildup the in / out function of nerve cells, and enhance synaptic plasticity

結果表明，苦參堿能夠劑量依賴性地抑制青霉素誘導的神經元順向信號傳導激活過程，使細胞外記錄到的場電位各個參數發生相應改變；三氟拉嗪可以改變ps的時相變化，提高ps的恢復程度和恢復率，減小了神經元因缺氧引起的不可逆損傷；高頻刺激（ highfrequencystimulate ， hfs ）可以長時間的增強ps的幅度和epsp的斜率，進而增強神經元的輸入輸出功能，增加了突觸的可塑性。

An experimental study on the neuroprotection of magnesium sulfate

硫酸鎂神經保護作用的實驗研究

The assessments and the discussions on the mechanissm on neuroprotection of tcm in ischemic stroke

中醫藥對缺血性中風神經保護作用的機理探討及評價

This suggests that melatonin possesses an ability of neuroprotection

誘導的神經細胞增殖抑制。以上結果提示，

A study of the neuroprotection effect of 21 aminosteroid u 74389g post traumatic brain injury

對腦外傷后記憶功能的影響

Her research interest is study on the cellular and molecular mechanisms of neuronal injury and neuroprotection

等雜志編委。目前，她主要從事神經細胞的損傷和保護機制研究。

Pretreament with repeated electroacupuncture induced neuroprotection against spinal cord ischemiareperfusion injury in rabbits

重復電針預處理對兔脊髓缺血再灌注損傷的保護作用

The neuroprotection of induced hypothermia has been investigated intensively and confirmed in animal models and it has been used clinically in many fields since the finding of sub - hypothermia can also reduce cerebral injury

摘要低溫對腦損傷的保護作用已被眾多動物實驗所證實，在臨床方面，尤其是發現輕度和中度低溫的保護作用之後，應用逐漸增多。

Background and purpose : in preclinical stroke models, high - dose human albumin confers robust neuroprotection. we investigated the safety and tolerability of this therapy in patients with acute ischemic stroke

背景及目的：在臨床前的動物中風模型中，高劑量的人血清白蛋白顯示出明顯的神經保護作用。本文研究了人血清白蛋白治療急性缺血性中風的耐受性及安全性。

To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation

為了驗證上述假設，本文分別在整體和離體水平，採用大腦中動脈缺血（ middlecerebralarteryocclusion ， mcao ）和體外氧?糖剝奪（ hypoxiaandglucose - deprivation ， hgd ）缺血模型，首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況，觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化，然後通過外源性給予vegf蛋白，在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響，最後利用vegf反義脫氧寡核苷酸（ oligodeoxynucleotide ， odn ）特異阻斷內源性vegf蛋白的表達，觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用，以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。