代謝性還原 的英文怎麼說

中文拼音 [dàixièxìngháiyuán]
代謝性還原 英文
metabolic reduction
  • 代 : Ⅰ動詞1 （代替） take the place of; be in place of 2 （代理） act on behalf of; acting Ⅱ名詞1 （歷...
  • 謝 : Ⅰ動詞1 （感謝） thank 2 （認錯; 道歉） make an apology; excuse oneself 3 （辭去; 拒絕） decline 4...
  • 性 : Ⅰ名詞1 （性格） nature; character; disposition 2 （性能; 性質） property; quality 3 （性別） sex ...
  • 還 : 還副詞1 （表示現象繼續存在或動作繼續進行; 仍舊） still; yet 2 （表示在某種程度之上有所增加） even...
  • 原 : Ⅰ形容詞1 （最初的; 原來的） primary; original; former 2 （沒有加工的） unprocessed; raw Ⅱ動詞（原...
  • 代謝 : 1. （交替; 更替） supersession 2. [生物學] （新陳代謝的簡稱） metabolize
  • 還原 : 1 （恢復原狀） return to the original condition or shape; restore2 [化學] （還原作用） reduction;...
  1. Inhibitors of cyp51, such as azalanstat ( rs - 2i607 ) and rs - 2i745, could inhibit the synthesis of ff - mas to decrease the accumulation of ff - mas. inhibitor of a14 - reductase, such as ay9944 - a - 7, could inhibit the metabolism of ff - mas to increase the accumulation of ff - mas ; and some other reagents, such as nystatin, could combine with the downstream intermediate in the cholesterol biosynthesis pathway to accumulate mas. in this study, we investigated the role of mas by using these reagents to change the level of endogenous ff - mas

    抑制cyp51酶的抑制劑，如azalanstat （ rs - 21607 ）和rs - 21745等，均能抑制mas的合成，降低mas的積累；而抑制14 -還原酶的抑制劑，如ay9944 - a - 7等，能抑制ff - mas向t - mas的轉化而造成ff - mas的積累；還有一些物質，如制黴菌素，能阻止mas向下游的代謝而造成其積累，本文主要通過應用這些物質降低、或增加組織細胞中內源性mas的積累，來研究mas的作用。
  2. The metabolism of these extreme microbes during the production of maotai liquor would further produce multiple enzymes of thermal stability such as amylase, protease, saccharifying enzyme, cellulose, glucase, xylanase, and each kind of dehydrase involved in redox reaction, and dna polyase etc

    茅臺酒釀造過程中極端釀酒微生物代謝產生多種熱穩定性的酶，如澱粉酶、蛋白酶、糖化酶、纖維素酶、葡萄糖甘酶、木聚糖酶、參與氧化還原反應的各種脫氮酶、磷酸烯醇丙酮酸激酶及dna聚合酶等。
  3. This paper describes several latest industrial microbial technologies in detail, which are the synthesis of the chiral diols by epoxide hydrolase from microbie, cofactors regeneration for redox with fdh, production of nano / micro wire by the phage display, metabolic network rebuilding for conventional fermentation and the application of the organic solvent tolerance and the metagenomics technology

    本文綜述了幾項最新的工業微生物技術，主要包括：微生物環氧化水解酶催化合成手性二醇、微生物甲酸脫氫酶用於再生氧化還原反應的輔因子、通過噬菌體展示技術得到納米級金屬絲、代謝網路改造和重建用於傳統發酵生產以及有機溶劑耐受菌和宏基因組技術的應用。
  4. The effects of meor include as follows : being favorable to oil - gas percolation, changing wettability of rocks, improving hydrocarbon migration, reducing surface tension of emulsified oil, decreasing crude viscosity and improving sweep efficiency, etc

    採油微生物具有自身的優勢，有利於油氣滲流及提高採收率，尤其是微生物能改變巖石潤濕性、改善油氣運移、乳化原油降低表面張力，微生物及代謝產物還對原油具有降粘作用，對地層水產生有利影響，提高波及系數。
  5. When inadequate n was added, the chloroplast structure in mesophyll cell was damaged in ear leaf, the amount of carbohydrate decreased in mestome sheath, and excessive n - redistribution and n - transportation to grain from vegetative mass appeared, which resulted in earlier leaf senescence. excessive n application led to too high activity of nitrate reductase, excessive vigorous nitrogen metabolism and too much exhaustion of carbohydrate in ear leaf, which resulted in the lack of enough carbohydrate in the lower leaf, meanwhile, the expansive chloroplast grana lamellae in leaf mesophyll cell and starch grain without nuclear in mestome sheath cell was observed, which led to the decrease of chlorophyll content and photosythetic capacity in maize leaf, then the early senescence occured

    氮肥用量不足導致穗葉葉肉細胞葉綠體結構性差，維管束鞘細胞碳水化合物累積減少，營養體氮素再分配率大而引起葉片早衰；而過量供氮則導致生長後期硝酸還原酶活性過高，氮素代謝過旺，消耗了大量碳水化合物，以致下位葉不能得到充足的碳水化合物供應而提早脫落，同時葉肉細胞葉綠體片層結構膨脹，呈「肉汁化」特徵，維管束鞘細胞澱粉粒大量消耗，無核澱粉粒出現，從而葉片葉綠素含量下降，光合能力降低而出現早衰。
  6. The co - expression of l l6 - hsd1 and gr in the saxne chorionic trophoblast suggests possible intrcrine achons ofglucoconicoid generated by l l6 - hsdl within the cells. 2. ewe radiomctric conversion assay showed that trctrient of the cells with the synthetic glucocorticoid - - dexarnethasone ( l0 - ' m ) for 24h increased the conversion of conisone to cortisol, and thes increase was blocked by the gr antagonist ru486

    雙標免疫組織化學染色結果顯示11p hsdi和gr共存於同一個滋養層細胞，提示在體內無活性的gc代謝產物?一17羥d脫氫皮質酮經11p hsdi還原活化后可以直接與同一個細胞內的gr結合，即以內在分泌（ intracrine ）形式發揮作用
  7. L l6 - hsd2 is localized to the sppytiotrophoblast of the placenty providing a fimctional barrier protecting the fetus from matemal glucocorticoids. a sequence resembling glucocorticoid response element ( gre ) has been identified in the promoter region of the human l l0 - hsdl gene. glucocorticoids have been shown to induce the expression of 11p - hsdl in the hippomus in vitro " whereas controversial results were obtained in the hepatocyte

    體內至少存在兩型11 - hsd ，在細胞完整的狀態下， 11 - hsd1主要為還原酶，它活化gc的代謝產物17 -羥- 11 -脫氫皮質酮（嚙齒類為脫氫皮質酮）為有活性的皮質醇（嚙齒類為皮質酮） ，而11 - hsd2為氧化酶，它催化皮質醇為無活性的17 -羥- 11 -脫氫皮質酮，因此11 - hsd1加強gc的作用，而11 - hsd2減弱gc的作用。
  8. Alfred marshall justifies the existence of medium and small enterprises by using some of the biological theories, such as the theory of species involution, the theory of metabolism and so on. among them the most noted ones are the law of tress growth in the forest, which is used to describe the development of enterprises ; chamberlain ' s imperfect competition theory, which illustrates the causes for the existence and development of medium and small enterprises ; and alfred marshall ' s " best scale " theory

    馬歇爾根據生物學中的物種進化、循序漸進、新陳代謝原理來分析中小企業存在的依據，其中比較著名的是用森林中樹木生長規律來闡述企業發展原理；張伯倫等人的不完全競爭理論用「產品差別性」說明大企業和中小企業都存在產品差別，產品差別使中小企業得以存在和發展；還有羅賓遜等人的「最佳規模」理論等等。
  9. 1, 2. 3, 4, 5, 6 respectively. with the increase of lead level, the changing trend of spad and soluble sugar content and the activity of nitric acid reverting enzyme ( nr ) had appeared the same direction, which ascended first and then fell

    2植物對pb污染的生理生態效應為了揭示pb污染對富集pb植物的生理代謝活動的影響，測定了葉片葉綠素含量、可溶性糖含量、硝酸還原酶的活性和脫落酸含量等生理指標。
  10. The fact that stevioside is glycosylated at position c13 could explain the absence of mutagenicity. the active metabolite of steviol responsible for its mutagenic activity is not known

    甜菊糖?的c13位置被糖基化，是沒有引起突變的原因。目前，還沒有找出引發突變能力的甜菊醇活性代謝物。
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