未受抑制的 的英文怎麼說

中文拼音 [wèishòuyìzhìde]
未受抑制的 英文
unchecked
  • 未 : Ⅰ副詞1 （沒） did not; have not 2 （不） not Ⅱ名詞1 （地支的第八位） the eighth of the twelve ear...
  • 抑 : Ⅰ動詞（向下按; 壓制） restrain; repress; curb Ⅱ連詞[書面語]1 （表示抉擇） or 2 （表示轉折） but3 ...
  • 制 : Ⅰ動詞1 （製造） make; manufacture 2 （擬訂; 規定） draw up; establish 3 （用強力約束; 限定; 管束...
  • 的 : 4次方是 The fourth power of 2 is direction
  • 抑制 : 1 （控制） restrain; control; check; hold up; curb; stop; repress; bridle; choke; prehension; sup...
  1. Under mixed culturing conditions, it was observed that bacterial number rapidly incre ; ised soon after the lysing of host cells. on the contrary, while the non - host cyanobacterium ( i. e. anabaena flos - aquae ) was incubated in the mix culturing system, the breeding of the bacteria could be inhibited. it may be suggested from the result above that cyanophage could bring about the changes in microorganism populations

    調查結果顯示， 19個採集的水樣中有6個含有裂解織線藻的噬藻體，而且水樣未經濃縮即能裂解宿主，說明噬藻體在淡水中分佈較廣泛，裂解性也較強；混合培養條件下的研究結果表明，噬藻體裂解宿主后，細菌數量快速增加，而當培養系統中有非宿主藻類存在時，細菌的增殖則受到非宿主藻的抑制，說明噬藻體可以顯著改變系統中微生物的種群結構。
  2. The degradation of i k b and dissociation from nf - k b complex are essential process for activation of nf - k b. there is no report about whether cck - 8 inhibits nf - k b activity through inhibiting lps - induced degradation of i k b. the present study was designed to observe the regulatory effect of cck - 8 on changes of immune functions induced by lps, the receptor mechanism and signal transduction mechanism involving mapk, stat3 and i k b at different levels including in vivo and in vitro, in order to study its immunomodulatory effect on reversing es and its molecular mechanisms

    Ikb的降解、並從w兒b復合物上解離是w4b激活的必需過程。 ccks是否通過抑制lps誘導的ikb降解，從而抑制nf幾b激活尚未見報道。本研究在整體、細胞及分子水平，觀察了ccke對lps引起的免疫功能改變的調節作用，研究了其受體機制及mapk 、 stat3和ikb的信號轉導機制，以探討cck七抗es時的免疫調節作用及其分子機制。
  3. It is most difficult to remember that the increase of every creature is constantly being checked by unperceived hostile agencies.

    有一點常常使人最不容易記起的，就是各種生物的增殖都是不斷地受著未被覺察的敵對作用的抑制。
  4. To search proteins that associate with the mouse mint protein and regulate notch signaling in nuclei, and to study the function and mechanism of mint - mediated transcription repression, yeast two - hybrid assay was used to screen proteins that interact with a fragment of mint ( f5, amino acids 2226 - 2959 ). from 4x106 yeast clones transformed with the bait plasmid and a cdna library of 9 dpc mouse embryo, fifty - one were positive for nutritional screening and p - galactosidase assay. restriction digestion identified 10 independent positive clones and these were analyzed by dna sequencing these clones represent 3 correctly fused cdna fragments, which are mint, alpha a - crystallin - binding protein i ( alphaa - crybpl ), and nuclear receptor co - repressor 1 ( n - corl ), respectively

    鑒于mwt基因編碼區較長，共有10799個堿基，故此我們將mint分為六段，分別命名為fi一f6 ，本研究以其中的f5 （ 222e959 ）和f6 （ 296s576 ）片段為研究對象，將h者分別插入pgb盯7載體中，結果顯示： mintfs可與核受體輔助抑制因子1階cori ） ， o晶狀體蛋白結合蛋白1hlphatcrybp入及mw加互作用，而f6可與igm的重鏈恆定區、泛素結合酶2l6和一個未知功能的新的蛋白基因進行結合。
  5. The protein level of p21 increased lightly from 1 to3 h, then decreased at 6 - 9 h, reached maximum at 24 h after 2gy, it increased from 3 to 24 h exposed tology, and the protein level of p21 associated with cdk2 increased, which did not inhibit the cyclin e / cdk2 kinase activity, because of upregulation of cyclin e and deregulation of p27kipl after irradiation. in contrast to p21, p27kipl may be a key regulator of cyclin e / cdk2 activity in dna damage responses. hi the ubiquitin - proteasome pathway invovled in regulation of p27kiplexpression in response to ir the ubiquitin - proteasome pathway regulated selective and time - controlled elimination of p27 kipl

    與p27ki 」 ，表達水平不同， pzi蛋白表達水平在zgy照射后略有增加， 6一gh略有下降，在照后24h達到最大; 10gy照后1一3h開始增加，持續到24h ，並且cycline / cdkz復合物中pzi蛋白結合增多，但cycline / cdkz激酶活性並未受到抑制，除了因ir誘導cydine蛋白表達水平增加外，另一重要的原因就是與pzi同一家族的p27kip ，蛋白表達水平下降，失去了對cycline / cdkz的抑制活性。
  6. The second approach is based on the use of protocols of stimulation that suppress cortical excitability, targeting the intact hemisphere in order to counteract the imbalance due to the increased interhemispheric inhibition onto the lesioned cortex, and reducing the potential negative interference of the intact hemisphere with the function of the affected one

    第二種方法是建立在抑制皮質的興奮性這樣的刺激設計應用，目標是未受累側半球，來阻礙由於受損皮質半球內抑制而增強的不平衡，減少未受累半球對受累側功能潛在的負干擾。
  7. In terms of regulatory t cell, cd4 + cd25 + t cell is representitive. ln naive individuals, cd4 + cd25 + t cells function as self tolerance, in recipients with long term survival allograft. they play an important role in suppression of allograft rejection. immune regulation potent cd4 + cd25 + t cells can be recovered from recipients with long term survival allografts. such regulatory t cells can suppress t cell mediated rejection in vitro and in vivo

    其中， cd4 + cd25 + t細胞是具有代表性的調節性t細胞。在未經處理的機體內， cd4 + cd25 + t細胞對保持自身耐受起著關鍵的作用。在同種異體移植物長期存活的受體內， cd4 + cd25 + t細胞對抑制同種異體移植物排斥反應也起到很重要的作用。
  8. Exactly why nsaids cause hypertension is not clear because they inhibit the generation of several soluble factors ( known as prostaglandins ) that can affect blood pressure and the effects of inhibiting the individual receptors for prostaglandins are not known

    確切地說，非甾體抗炎藥引發高血壓的機理還不清楚，它們可以抑制一些能夠影響血壓的可溶性因子的產生（稱作前列腺素） ，但是抑制前列腺素的單獨受體的影響作用還未揭曉。
  9. Result 1, human antisense cd40 rna eukaryotic expression vector was constructed successfully. 2, in the presence of cd40 / pcdna3, cd40 expression was significantly decreased, cell proliferation and antibodies generation were significantly restrained, compared to that of the controls ( p < 0. 01 )

    2 、與轉染pcdna3空載體組或未轉染質粒組相比較，轉染cd40 pcdna3的健康人及sle患者b細胞系cd40的表達均明顯減少，增殖能力明顯下降， ig的分泌受到明顯抑制。
  10. The result shows why mitochondrial suppression is so important to tumours : when they are unsuppressed, the tumour they are in stops growing

    為何線粒體抑制對腫瘤如此重要從試驗結果可見一斑：當線粒體未受到抑制時，線粒體所在的腫瘤就會停止生長。
  11. Furthermore, when normally induced 3t3 - l1 preadipocytes were treated with pd98059 ( an inhibitor of mek - 1 ), to block the activation of erk1 and 2, the mitotic clonal expansion was blocked, while adipocyte differentiation was not affected

    我們用有絲分裂原激活蛋白激酶激酶（ mapkk / mek1 ）的抑制劑pd98059與誘導分化激素及藥物一起對細胞誘導分化，克隆擴增不發生，但細胞分化未受影響。
  12. Though the growth of sp - d, sp - d ( l ) and sp - z ( sl ) is distinctly inhibited by ampicillin, they are still alive in the cultures ; whereas sp - j, sp - j ( l ), sp - b and sp - fl are lethal in this condition. it means that they are most sensitive to ampicillin

    其中即上和sps對青霉素的抗性較強； spc其次： sp 、 sp mq 、 sp刁8q的生長雖受到青霉素的明顯抑制，但未能完全致死： sp j 、 8卜j0xspb 、 spf可被全部致死，因此它們對青霉素的敏感性最強，抗性最小。
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