染毒 的英文怎麼說
中文拼音 [rǎndú]
染毒
英文
[軍事] contamination-
Expression of caspase - 3 in myocardial cell of carbon disulfide poisoned mice
3在二硫化碳染毒小鼠心肌細胞中的表達Develop as the illness, because cancerous swollen canker, exsanguine, infection, toxin is sucked, close etc, the patient can appear the systemic symptom such as anaemic, angular, lack of power, low heat
隨著病情發展，由於癌腫潰爛、失血、感染、毒素吸收等，病人可出現貧血、消瘦、乏力、低熱等全身癥狀。We found nuclear deformation lymphocytes with increased heterochromatin and impaired thymus epithelium cells with increased lysosomes and deformation of mitochondrias. ( 4 ) effects on mouse liver of so2 challenge : so2 can cause significant liver injury. he staining showed several kinds of necrosis of liver including spot necrosis, focal necrosis and submassive necrosis infiltrated with lymphocytes, monocytes, few neutrophils and eosinophils ; tem observation showed fatty degeneration with dispersion of fatty droplets and dilation of rough endoplasmic reticulums, acid degeneration with significant hyperplasia of mitochondrias, necrosis of hepatocytes with karyorrhexis and other organelles losing their normal structure
（ 4 ）二氧化硫染毒對小鼠肝臟的組織學結構有明顯影響，可引起肝臟點狀壞死、灶狀壞死甚至片狀壞死，伴隨不同程度的炎性細胞浸潤；透射電鏡觀察發現二氧化硫可引起肝細胞脂肪變性、嗜酸性顆粒變和壞死，脂肪變肝細胞中可見大小不等的脂滴存在，嗜酸性顆粒變肝細胞中可見線粒體明顯增生，壞死肝細胞可見細胞核結構破壞，細胞器減少，細胞膜不完整。Dna damages caused by so2 and lead acetate were studied with the single cell microgel electrophoresis technique ( or comet assay ) in order to confirm the damaging degree of lead ( as an important component of atmosphere particle matter ) on dna from male mice exposed to so2. the migrating distances of dna of brain, lung, spleen and kidney cells of mice increased significantly, compared to the control group under conditions of single and combined poisoning of so2 ( 42mg / m3 ) and lead acetate ( 0. 2 % ), and lead could strengthen dna damage degree by so2 in nuclear dna of brain, kidney, spleen cells. damaging degree of so2 on nuclear dna of lung cell of mice was more severe than that of lead
為了明確大氣顆粒物中的重要組分? ?鉛在二氧化硫所致dna損傷中的作用程度，利用單細胞凝膠電泳技術（ singlecellgelelectrophoresis ， scge ，或稱彗星實驗， cometassay ）研究了鉛與二氧化硫的聯合污染，結果表明在42mg m ~ 3so _ 2和0 . 2醋酸摘要一abstract鉛單獨及聯合染毒條件下，小鼠腦、肺、腎、脾細胞dna遷移距離均比對照顯著增加;鉛加劇了50 :對腦、腎、脾細胞核dna的損傷程度; 50 :對肺細胞核dna的損傷程度要比鉛的損傷大，小鼠肺細胞核dna遷移距離在50 :和醋酸鉛聯合作用組與醋酸鉛單獨作用組間有極顯著性差異（ p < 0 . 01 ） ，而與502單獨作用組間沒有顯著性差異。In acute toxicity test, male and female wistar rats received sodium selenite and mercuric chloride singly or associatively. the values of the ldso show an obvious antagonism between mercury and selenium, and the ldso of the females are higher than that of the males in associated dose but contrary in the single dose
建立了wistar大鼠急性灌胃染毒試驗模型，得到了hgclz 、 nazseo3單獨作用和聯合作用的ld50 ， ld50表明汞和硒之間毒性作用為典型的拮抗作用，雌性大鼠拮抗作用更強。The oxidant injury induced by phosgene of lung in mice
光氣染毒造成小鼠肺臟的氧化損傷They sequenced the genetic code of a sars virus taken from one of the first known carriers of the disease, and of sars viruses found in four people in singapore who had had direct or secondary contact with that individual
他們排序了sars病毒的遺傳密碼，這些病毒取自於最先發現的病人中的一位，也取自於在新加坡直接或二次接觸這些病人而染毒的4位病人。Acute toxic effect of sodium cyanide on cerebral cortex of rats and its mechanism
氰化鈉急性染毒對大鼠大腦皮層損傷的作用機制Effects of taurine against damage induced by 60co - ray irradiation
亞急性樂果染毒對自發性高血壓大鼠心血管系統的影響Changes of several biochemical parameters of rat - treated by trinitrotoluene
三硝基甲苯亞慢性染毒大鼠某些生化指標的改變Protection against synergistic hepatocarcinogenesis of hepatitis b virus expression and aflatoxin b1 by antioxidant 2 - tert - 4 - hydroxyanisole in hbv large envelope transgenic mice
抗氧化劑對黃麴黴毒素染毒的轉基因小鼠肝腫瘤的防護The antioxidant abilities of protective enzymes were enhanced in restricted range by hg2 + stress in the early period and the lower concentrations, which were weaken because of the further exposure, especially in liver
抗氧化酶系統在受到低濃度及染毒初期的一定限度內能被誘導而加強，但隨著重金屬毒害加深而減弱，尤其是肝組織。Then were electrophored. the extent of dna migration were measured. index - percentage of " comet " cell and " comet tail " were analysed that indicated when vero cells were treated with 2 u g / ml quinocetone vero cells got midium - grade damage, and were treated with 6 u g / ml olaquindox vero cells got midium - grade damage
喹乙醇染毒劑量在2 10 g ml時細胞存活率90 ，以2 10 g ml劑量的喹乙醇染毒處于對數生長期的vero細胞，后經過電泳，通過分析彗星樣細胞發生率和彗尾長短等指標，結果表明染毒劑量在6 g ml對dna造成中度損傷。Index - percentage of " comet " cell and " comet tail " were analysed that indicated the optimum time is 2 ~ 3h. the degree of dna damage by quinocetone and olaquindox were detected by scge assay, when vero cells were treated with 1 ~ 5 ug / ml quinocetone the percentage of live cells was above 85 % and with 2 - 10ug / ml olaquindox the percentage of live cells was above 90 %
喹烯酮染毒劑量在1 5 g ml時細胞存活率85 ，以1 5 g ml劑量的喹烯酮染毒處于對數生長期的vero細胞，后經過電泳，通過分析彗星樣細胞發生率和彗尾長短等指標，結果表明染毒劑量在2 g ml對dna造成中度損傷。( 5 ) effects on mouse cerebrum cortex of so2 challenge : we found no pathological changes of mouse cerebrum cortex challenged by so2 by he staining and tem observation, in vitro tests of so2 derivatives challenge showed that : ( 1 ) so2 derivatives has significant toxicity on mouse spleen cells, the lc50 of 4h and 24h exposure is 1. 1293 mmol / l and 1. 3777mmol / l respectively
二氧化硫代謝衍生物體外實驗結果表明: （ l ）二氧化硫代謝衍生物對小鼠脾細胞具有明顯的細胞毒性作用，染毒4小時和24小時細胞的半數存活抑制濃度lcs 。分別為1 . 12932r以mol幾和1 . 37777 ~ ol幾。We also investigated the pathological changes of mouse liver, thymus and cerebrum cortex challenged by so2 inhalation by in vivo tests. we studied the apoptotic induction on mouse spleen cells and cytotoxicity of human embryo lung fibroblasts of so2 derivatives by in vitro tests. in vivo tests of sulfur dioxide inhalation showed : ( 1 ) effects on mouse lung of so2 challenge : we found no significant apoptotic changes induced by so2 inhalation but obvious pathological changes of lung with vacuolating of osmiophilic multilamellar bodies which maybe related with the decrease of surfacant and decrease of microvillus of type ii alveolar cells ; we also found thickening of part of basement lamina between type i alveolar cells and capillary endothelium cells which may inhibit the dispersion of oxygen and contribute to lung dysfunction
二氧化硫熏氣染毒的體內實驗結果表明，在本次實驗的濃度范圍內（ 56mg m ~ 3 、 112mg m ~ 3 、 168mg m ~ 3低、中、高三個濃度） ： （ 1 ）通過透射電鏡、 dna凝膠電泳分析和流式細胞分析發現二氧化硫吸入染毒一周對小鼠肺臟沒有明顯的凋亡誘導作用，但通過透射電鏡觀察發現二氧化硫可引起肺臟明顯的超微結構改變，引起型肺泡上皮細胞板層體空泡化，微絨毛減少，線粒體緻密化或腫脹變性；肺泡血管內皮細胞和型肺泡上皮細胞之間基膜增厚，使氧氣彌散功能出現障礙，從而降低肺功能。Mutagenicity of joint exposure to formaldehyde and benzene in mice
苯聯合染毒對小鼠的致突變作用Effect of selenium exposure on the immunological function in mice
亞硒酸鈉染毒對小鼠免疫功能的影響Effects of formaldehyde on organs and blood cells in rat offspring
甲醛染毒對大鼠子代臟器及血液細胞的影響Changes of hemoglobin in rats during co - exposure to aluminium and fluorine
氟聯合染毒大鼠血紅蛋白含量的變化分享友人