生死細胞 的英文怎麼說

中文拼音 [shēngbāo]
生死細胞 英文
germocyte
  • : Ⅰ動詞1 (生育; 生殖) give birth to; bear 2 (出生) be born 3 (生長) grow 4 (生存; 活) live;...
  • : Ⅰ動詞(失去生命) die Ⅱ形容詞1 (不顧生命; 拚死) to the death 2 (達到極點) extremely; to death...
  • : 形容詞1 (條狀物橫剖面小) thin; slender 2 (顆粒小) in small particles; fine 3 (音量小) thin ...
  • : Ⅰ名詞1 (胞衣) afterbirth2 (同一個國家或民族的人) fellow countryman; compatriot Ⅱ形容詞(同胞...
  • 生死 : life and death生死比率 birth- death ratio
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  1. Histology of the tumor revealed necrosis of salivary glands, squamous metaplasia of salivary ducts and acini with bland appearance of the squamous cells which showed occasional nuclear atypia

    病理特徵為:唾腺壞、唾腺管與腺泡的鱗狀化、其鱗狀外觀平淡無奇,但偶可見異常核。
  2. Comprehensive cellular responses was found in human amnion fl cells following exposure to low concentration of mnng, such as the lowering of dna replication fidelity resulted from alteration of dna polymerase profile ; activation of a lot of transcription factors, such as api, creb, nf - kb etc ; clustering of egfr ( epidermal growth factor receptor ) and tnfr ( tumor necrosis factor receptor ) and activation of camp - pka - creb and jnk / sapk signal pathways

    我們發現,低劑量mnng處理后的人羊膜fl有廣泛的反應,並有多個信號轉導通路的激活和基因表達的改變。例如dna復制保真度下降, dna聚合酶譜發改變,應用報告基因技術和底物磷酸化檢出技術證明一系列轉錄因子如ap1 、 creb 、 nf b等被激活,表面受體如表皮長因子受體、腫瘤壞因子受體發聚簇,信號轉導通路camp - pka - creb和jnk sapk被激活。
  3. The b lymphocyte stim lator ( blys ) also known as baff, tall - 1, thank, ztnf4, is the most recent addition to the tumor necrosis factor family ( tnf ) ligands. blys induces both in vivo and in vitro b cell proliferation, differentiation and immunoglob lin secretion. meanwhile, it also strongly suppresses the growth of tumor cell lines. the over - production of blys is associated with the development of certain autoimmune disease, such as systemic lupus erythaematosus ( sle ), rheumatoid arthritis, myasthenia gravis, sj greh ' s syndrome

    人b淋巴刺激因子( hblys )是1999年發現的腫瘤壞因子超家族成員。最早發現其可誘導腫瘤凋亡,之後發現它是一個強有力的b共刺激因子,它在體外及轉基因動物體內可明顯刺激b長,免疫球蛋白分泌,並導致動物自身免疫性改變。
  4. Growth factors can act on many kinds of cells as mitotic promoter and deprival of growth factors can cause cease of cell growth or cell death. thus growth factors are usually thought relative to cell growth and differentiation

    長因子可以作為促分裂劑作用於多種,剝奪長因子可以導致長停止或亡,因此通常認為長因子與促進的增殖和存活有關。
  5. Detachment of the dead cells produces erosions and ulcers.

    的脫落,產糜爛和潰瘍。
  6. Facial treatment includes cleansing, peeling, ozone steam, pore cleansing, high frequency, diathermy applications, facial massage, facial mask and shoulder massage

    面部護理包括潔膚、去皮、臭氧噴霧、清潔毛孔、高頻電療、電腦去斑活化護理、面部按摩、面膜及肩膊按摩。
  7. For example, we have found the clustering of egfr ( epidermic growth factor receptor ) and tnfr ( tumor necrosis factor receptor ) and the activation of camp - pka - creb and jnk / sapk pathways after mnng treatment

    例如表面受體如表皮長因子受體、腫瘤壞因子受體發聚簇,信號轉導通路camp kacgyb和jnk sapk被激活。
  8. The roots of many orchids and other epiphytes developed a sheath of dead cells, the velamen, which helps to absorb water from the atmosphere

    許多蘭科植物的根和一些附植物的鞘以及根被都可以幫助植物從空氣中吸收水分。
  9. The cause that causes dark skiing ulcer has a lot of, have endocrine, food, genetic factor, the one mite bug that returning bug having mite also is the main reason that causes dark skiing ulcer lives inside wool bursa and sebaceous glands, it absorbs the nutrition of wool bursa and sebaceous glands cell, discrepancy skin carries a lot of bacteria, it secretes the dead damage body of excretive material and mite bug to be able to cause the skin organize inflammation to cause dark skiing ulcer

    引起暗瘡的原因有很多,有內分泌、飲食、遺傳的因素,還有蟎蟲也是引起暗瘡的重要原因之一蟎蟲活在毛囊和皮脂腺內,它吸收毛囊和皮脂腺的營養,出入皮膚帶入很多菌,它分泌排泄的物質和蟎蟲的亡殘體都會造成皮膚組織發炎引起暗瘡。
  10. We found nuclear deformation lymphocytes with increased heterochromatin and impaired thymus epithelium cells with increased lysosomes and deformation of mitochondrias. ( 4 ) effects on mouse liver of so2 challenge : so2 can cause significant liver injury. he staining showed several kinds of necrosis of liver including spot necrosis, focal necrosis and submassive necrosis infiltrated with lymphocytes, monocytes, few neutrophils and eosinophils ; tem observation showed fatty degeneration with dispersion of fatty droplets and dilation of rough endoplasmic reticulums, acid degeneration with significant hyperplasia of mitochondrias, necrosis of hepatocytes with karyorrhexis and other organelles losing their normal structure

    ( 4 )二氧化硫染毒對小鼠肝臟的組織學結構有明顯影響,可引起肝臟點狀壞、灶狀壞甚至片狀壞,伴隨不同程度的炎性浸潤;透射電鏡觀察發現二氧化硫可引起肝脂肪變性、嗜酸性顆粒變和壞,脂肪變肝中可見大小不等的脂滴存在,嗜酸性顆粒變肝中可見線粒體明顯增,壞可見核結構破壞,器減少,膜不完整。
  11. The results of biological tests have demonstrated that allantoic fluid of the first passage virus did n ' t produce macroscopic pathogenic role to chicken embryos and after passaged for four times, gross lesions were observed in chicken embryo. the virus showed typical coronavirus under electron - microscope and it could n ' t form plaque in cef cells and could hemagglutinates chicken red blood cells after treatment with 1 % trypsin. to surprise, the virus replicated in cef cells also showed hemagglutination activity to chicken red blood cells. in addition, the spf chickens which inoculated with the virus isolated from the chicken damaged tissue showed clinical sign and grow lesion, but it ' s gross lesion did n ' t resemble to those of field outbreaks

    物學特性:雞胚尿囊液經離心、磷鎢酸負染后,電鏡觀察該病毒為典型的冠狀病毒;該毒株的第一代尿囊液對雞胚無肉眼可見的致病作用,當繼代到第5代后,胚體嚴重病變;病毒在雞胚中隨著接種時間的延長,其效價增高, 96h可達到48h的2倍;該毒株可在cef上長,但不能形成明顯的蝕斑;經1胰酶處理后可凝集雞紅;雞胚的第四代尿囊液病毒回歸動物體,病雞腎臟呈典型的花斑腎,腺胃則未見肉眼可見的病變。
  12. Those cells that do die, perhaps in the transmutation process ( along with other toxins unnecessary to the crystalline form ) are filtered out by two of these four new organs which grow slightly above the hipbone, expanding the size of the lower belly

    那些的確亡的,也許在轉化過程中(以及那些對水晶體而言不需要的毒素)被這4個新器官中的2個過濾走,它們長在髖骨的稍上方,擴展了腹部下半部的尺寸。
  13. Investigators have found that the knapweed ' s roots exude a toxin that builds up in the soil

    研究者發現,這種矢車菊根部分泌的一種毒素會滲進土壤里,使得其他植物的大量亡,並抑制萌芽與長。
  14. It was also found that the nuclear envelope disappeared earlier at the micropylar end than that at the chalazal end. the speed of the programmed cell death were different in three nonfunctional megaspores at the micropylar end and their relationships with the neighboring cells were also different. the biological significance was discussed about these phenomenon

    還發現了大孢子母合點端的核膜先於珠孔端的消失,並且珠孔端3個大孢子的亡時間不同,與相鄰的關系也不同,進而討論了這些現象的物學意義。
  15. During al treatment or recovery culture without al, the irreversible inhibition of cell elongation or growth was resulted from cell death

    在鋁脅迫或無鋁恢復培養過程中產伸長或長的不可逆抑制起源於亡。
  16. Most of the newborn cells die, but some successfully migrate to the damaged area and have been reported to become adult neurons

    大多會亡,但有一些可以成功遷移到受損的區域,而且曾有報告指出,它們會發育為成熟的神經
  17. We identify, for the first time, a number of gene candidates that are induced by dht with or without ischemia, many of which could account for cell death through enhanced inflammation, dysregulation of blood - brain barrier and the extracellular matrix, apoptosis, and ionic imbalance

    我們首次發現,許多候選基因在有無缺血時都由dht誘導,這其中的許多基因能夠解釋通過增加炎癥、血腦屏障和外基質失調、凋亡和離子失衡等原因產亡。
  18. Besides from the above - mentioned typical characters of hypersensitive reaction, the host cells also produced defense structure in cell walls and secondary metabolic materials

    除了上述典型的過敏性壞反應特徵以外,寄主還產壁防禦結構和次物質。
  19. When the oral cleaning function of saliva is reduced, dead cells will accumulate on the tongue, gums and cheeks. these cells then decompose and cause odour

    由於唾液有清潔口腔的功能,唾液流量不足的話,口腔內的壞便會聚積在舌頭、牙肉及面頰,並且腐壞和產口臭。
  20. According to the characteristics of morphology, biochemistry, and molecular biology of cells, the apoptotic cells can be distinguished from the necrotic cells

    根據的形態學、物化學和分子物學特徵,可以將凋亡與壞區別開來。
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