甲狀旁腺增生 的英文怎麼說

中文拼音 [jiǎzhuàngbàngxiànzēngshēng]
甲狀旁腺增生 英文
parathyroid hyperplasia
  • 甲 : Ⅰ名詞1 （天乾的第一位） the first of the ten heavenly stems2 （爬行動物和節肢動物身上的硬殼） she...
  • 狀 : Ⅰ名詞1 （形狀） form; shape 2 （情況） state; condition; situation; circumstances 3 （陳述事件或...
  • 旁 : 名詞1 （旁邊） side 2 （漢字的偏旁） lateral radical of a chinese character （e g 亻 氵 etc ）Ⅱ形...
  • 腺 : 名詞（生物體內能分泌某些化學物質的組織） gland
  • 生 : Ⅰ動詞1 （生育; 生殖） give birth to; bear 2 （出生） be born 3 （生長） grow 4 （生存; 活） live;...
  1. If hyperparathyroidism is present, removal of hyperplastic or adenomatous parathyroids is required

    如存在甲狀旁腺功能亢進，就需行甲狀旁腺增生或腺瘤切除。
  2. Objective to report 16 cases of intracranial aneurysms with calcified vascular wall and study the relationship between vascular calcification and pathogenic mechanism of intracranial aneurysms. methods spiral ct scan, clinical data and serum calcium, phosphate, glucose, lipid, renal function of the 16 patients were studied in retrospect. all aneurysms were clipped, in which 8 were resected after clipping. results the location of aneurysms with aneurismal wall calcification was, on posterior communicating artery in 3, on middle cerebral artery in 2, on anterior communicating artery in 2, on basilar artery in 5 and on basilar artery in 4. all patients showed normal serum calcium, phosphate, glucose and normal renal function. serum lipid of patients had been elevated. all patients recovered with no significant neurological deficits. conclusion intracranial vascular calcification is an active and regulated process in close relation to atherosclerosis, serum calcium, phosphate, lipid, thyroid, parathyroid function and other factors. intracranial vascular calcification has close relationship with the pathogenesis and management of intracranial aneurysms

    目的報告16例瘤壁有鈣化的顱內動脈瘤病例的治療，分析顱內血管鈣化與顱內動脈瘤病理發生機制的關系.方法回顧性分析16例瘤壁有鈣化的顱內動脈瘤病例的影像學、臨床資料及手術治療.結果瘤壁有鈣化的顱內動脈瘤的位置是: 3例位於后交通動脈， 2例位於大腦中動脈， 2例位於前交通動脈， 5例位於椎動脈， 4例位於基底動脈.所有患者血鈣、磷酸鹽、血糖、腎功能正常. 6例患者血脂增高. 16例患者均行動脈瘤夾閉術，其中8例術中切除動脈瘤，治療效果良好.結論動脈瘤瘤壁鈣化與其病理發生機制有關，常見于巨大或大動脈瘤，瘤壁血管鈣化是與動脈粥樣硬化、血鈣、磷酸鹽、血脂、甲狀腺、甲狀旁腺等因素有關的主動耗能，多因素調控的病理過程
  3. The high parathormone levels increase osteoclast activity and produce irregular bone resorbtion with microfractures and hemorrhage and macrophage proliferation and fibrous connective tissue proliferation

    高甲狀旁腺激素水平提高了破骨細胞的活性並導致不規則的骨溶解吸收，伴隨微骨折、出血、巨噬細胞增殖及纖維性結締組織增生。
  4. In parathyroid hyperplasia, there is little or no adipose tissue, but any or all cell types normally found in parathyroid are present

    甲狀旁腺增生很少有或沒有脂肪組織存在，但各類細胞正常。
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