缺鉀癥 的英文怎麼說
中文拼音 [quējiǎzhēng]
缺鉀癥
英文
kaliopenia-
The first sign of potassium deficiency is a white speckling or freckling of the leaf blades.
缺鉀的最初病癥是葉片呈現白色斑痕或斑點。See also plate 32 manganese deficiency and plate 20 potassium deficiency
同樣注意圖32缺錳癥和圖20缺鉀癥。However, because of the position on the plant, the symptoms were not considered as being due to k deficiency
然而因其在植物上出現的位置,並未認為這是缺鉀癥狀。At first, plants would turn dark green but soon the top leaves would progress through typical k symptoms to bronzing and leaf curl
最初植株變得深綠,但很快上部葉片就會經歷典型缺鉀癥狀出現青銅癥和卷葉。Leaf : threshold iron supply with potassium deficiency ; symptoms generally as in 27 or 28 above
番茄增加鐵供應加劇鉀缺乏癥狀:葉片:增加鐵的供應可使鉀缺乏,一般癥狀如27或28所述。Leaf to right : same potassium supply with additional ( luxury ) iron supply ; symptoms of potassium deficiency delayed in older leaves and especially evident in later younger leaves finally becoming more severe with increased iron supply
從左到右:鉀的供應伴隨著鐵的供應,缺鉀癥會延遲出現在老葉上,後期的幼葉尤其明顯,最後隨著鐵供應的增多,缺鉀癥狀更加嚴重。Pre - mixed potassium chloride iv infusion these preparations are used in the treatment of potassium deficiency. in acute hypokalaemia, parenteral administration of potassium is necessary
治療鉀缺乏癥,以及在急性低鉀血癥中須將鉀作非口服的注射However, researchers challenged by these symptoms, by the lack of correlation with surface soil k levels and prompted by ppi staff scientists, set out to determine the cause of the problem
但受到這些癥狀、其與表土鉀水平缺乏相關的挑戰,受ppi科學家鼓勵的研究人員著手確定問題的根源。Previous studies have showed that sulfonylureas possess a bidirectional effect on cardiovascular system : they can raise the coronary artery tone, aggravate ischemic injury to the cardiovascular tissues, and accelerate cardiac hypertrophy through closing cardiovascular atp - sensitive k ( superscript - ) channels ; meanwhile they can also regulate glucolipid metabolism, modulate inflammatory reaction and inhibit atherosclerosis through activating peroxisome proliferators - activated receptors
摘要基礎研究表明,磺脲類藥物對心血管系統有雙向影響:磺脲類藥物能夠關閉心血管系統atp敏感性鉀通道,從而增高冠脈張力,加重缺血對心血管組織的損傷和促進心臟肥厚的發生;另一方面,磺脲類藥物能夠激活過氧化物酶體增殖物激活受體,從而調控糖脂代謝,調節炎癥反應和抑制動脈粥樣硬化的發生。分享友人