肝毒性作用 的英文怎麼說
中文拼音 [gāndúxìngzuòyòng]
肝毒性作用
英文
hepatotoxic effect- 肝 : 名詞(人和高等動物的消化器官之一) liver
- 毒 : Ⅰ名詞1 (對生物體有害的性質或物質; 毒物) poison; toxin 2 (毒品) drug; narcotics 3 (姓氏) a s...
- 性 : Ⅰ名詞1 (性格) nature; character; disposition 2 (性能; 性質) property; quality 3 (性別) sex ...
- 用 : Ⅰ動詞1 (使用) use; employ; apply 2 (多用於否定: 需要) need 3 (敬辭: 吃; 喝) eat; drink Ⅱ名...
- 毒性 : [藥理學] toxicity; virulence; poisonousness毒性測定 toxicity test
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Through expert s analysis, aloe contains rich natural protein, vitamin, chlorophyl and the neccessary microelements. it has effects of laxative, stomach care, detoxifcation, detumescence, acesodyne and diminish inflammation. so aloe is usually used to treat astriction, cold, cough, headach, car sickness, bronchia, gastric ulcer, liver disease, hypertension, diabetes, eczema, fleck, chilblain, scald, cancer, etc
經科學分析,它含有大量天然蛋白質、維生素、葉綠素、洛性酶和人體必需的微量元素及蘆蔡大黃素等七十多種成份,具有催瀉、健胃、通經、解毒、消腫止痛、清熱抗炎等作用,對便秘、感冒、頭痛、咳嗽、暈車、支氣管、胃瘍病、小兒厭食癥、肝病、出血癥、高血壓、糖尿病、濕疹、雀斑、凍瘡、燙傷、刀傷、癌癥等數十種疾病有療效。Human augmenter of liver regeneration ( halr ) is a novel cytokine which stimulates specifically hepatic cell proliferation and is able to rescue acute liver failure caused by hepatotoxin for example carbon tetrachloride and galactosan amine et al
人肝再生增強因子( humanaugmenterofliverregeneration , halr )是一種新的細胞因子,能特異性地刺激肝源細胞的增殖,並對四氯化碳、半乳糖胺等肝毒劑引起的肝損傷有治療作用。Using cd, cu, zn and cr as experimental toxicants and crucian as experimental creature, we employed the environmental biotechniques ( flame atomic absorption spectrophotometry, the method of improved pyrogallol autoxidation and electron microscopy ) to study the acute toxicity and secure concentrations of heavy metals to crucian ; the accumulation and distribution of mixed heavy metals to fingling crucian ; the activities of superoxide dismutase ( sod ) of gill and liver tissues and the hispathological and ultrastructural change of superfine structure of liver and kidney of crucian after cadmium exposure. through which, we intended to fully and systemically study the toxic influence of heavy metals to fish, and preliminarily discuss the influence of heavy metals pollution to the diversified level of fish such as individual, organ, tissue, cell and molecule. thus we tried to provide scientific evidence to establish preventative management measures, avoid and relieve the harm of heavy metals pollution to aquicolous ecosystem in time
隨后以這幾種重金屬作為實驗毒物,以鯽魚為實驗動物,應用環境生物技術、火焰原子吸收分光光度法、鄰苯三酚自氧化法、透射電鏡等技術研究了重金屬cu 、 zn 、 cd和cr對鯽魚的急性毒性及其安全濃度評價、混合重金屬在鯽魚幼體組織內的積累和分佈、 cd對鯽魚鰓和肝臟中sod活性的影響、 cd對鯽魚肝細胞和腎細胞超微結構的影響等,全面和系統地研究了水環境中重金屬暴露對魚類的毒性影響,初步探討了重金屬污染對魚類的個體? ?器官? ?組織? ?細胞? ?分子等各水平層次的影響,為制定漁業生產上預防性的管理措施提供科學依據,及時避免或減輕重金屬污染對水生生態系統造成的損害。Even so, by truncating hbv pres gene, we finally obtained some useful " " bailors ", either nontoxic or self - activating, and used them to fish dna fragments of hbv pres interacting protein ( s ) from an ad vector constructed human embryonic cdna library
我們通過第回軍巨大學碩士學位論文對pres基因分段截短的方法,獲得了對酵母細胞即無毒性作用,又沒有自激活作用的「誘餌」 ,通過它在酵母雙雜交系統中篩選構建於ad載體的人胎肝。The main study in this paper included as follows : the content and distribution of heavy metals in sediments and benthic organisms from the sewage stream in guangzhou city ; the acute toxicity and joint toxicity of mercury and selenium to swordtail fish ( xiphophorus helleri ) ; the damage of mercury to the indexes of antioxidant system in the gills and livers in swordtail ( including the measurement of the activities of total antioxidative capacity [ t ~ aoc ], superoxide dismutase [ sod ], glutathione peroxidase [ gsh - px ] and the concentration of malondiald - ehyde [ mda ] ) and the relief effects of selenium on it, as well as the physiological damage of mercury on the tissues, namely : the antagonistic effect of na + - k + ~ atpase activity on the tissues between selenite and mercury, and the ultrastructural damage under the exposure of mercury
研究內容主要有:廣州市河涌沉積物及底棲生物體內重金屬含量及評價;汞和硒對劍尾魚的急性毒性和聯合毒性及安全濃度的評價;汞對劍尾魚鰓和肝臟中抗氧化系統的毒性,包括對總抗氧化能力、超氧化物歧化酶、谷胱甘肽過氧化物酶活力及丙二醛含量的測定及硒對其保護作用;汞對劍尾魚組織生理毒性即:汞對na ~ + - k ~ + atpase活力的影響及硒的保護作用和汞和對劍尾魚組織超微結構的損傷等。以高氯酸?硝酸消化法和火焰原子吸收分光光度法測定了廣州市河涌沉積物和底棲生物中重金屬含量。Protective effect of ethanol extracts of ginkgo biloba leaves on mice hepatic toxic injury induced by isoniazid and rifampicin
銀杏葉醇提取物對異煙肼和利福平肝毒性保護作用的實驗研究Exploring the possible mechanisms is an important content of bioinorganic chemistry and ecotoxicology, which has signality in modifying contamination effect to animals. this thesis studies on the chemical speciation of mercury and selenium in dolphin livers, sub - chronic toxic effect of wistar rats of sodium selenite and mercuric chloride and investigates the antagonism mechanism from there aspects : 1. redistribution of mercury in the presence of selenium, 2
本文以海豚肝臟為對象,研究其積累的高濃度汞硒化合物的化學形態和結構,並結合動物實驗的研究,從汞硒相互作用的總體生物學表現,汞硒在動物體內分佈變化和結合形態的變化以及汞硒化合物的結構等方面探討兩元素毒性拮抗作用機理。Antioxidant effect of scutellarin and its antagonistic action on the liver toxicity of se in rats
燈盞花素的抗氧化作用及拮抗硒對大鼠肝臟毒性的研究The variety of the activity of na + / k + - atpase, ca2 + - atpase and sod by increase calcium concentration in the water, and the effects of cd2 +, estradiol - 17 and ca2 + on the lymphocyte proliferation from blood and head kidney of ctenopharyngodon idellus were also investigated the significance of this paper is that to research the toxicity of cadmium to the fish and to search the mechanism of the cadmium toxicity to the fish. and the protection of calcium on cd2 + toxicity
本論文研究了鎘對草魚的毒性作用,比較了在不同鈣濃度條件下,其鰓、肝臟、腦等組織中sod 、 na ~ + k ~ + ? atp酶、 ca ~ ( 2 + ) ? atp酶的活性變化規律以及鎘和17 ?雌二醇對草魚血液及頭腎淋巴細胞增殖的影響,評價鎘對魚類的毒害作用,探索鎘的毒害機理。Scientists at erasmus university in rotterdam, the netherlands, conducted a six - year study of 5, 395 people aged 55 and over who did not have signs of dementia
過量飲酒的不良作用是毫無疑問的。除了損傷肝臟,許多研究已證明,過量飲酒可使大腦中毒。飲酒過量者最終會導致大腦萎縮,造成癡呆。甚至醫學上就專門有"酒精中毒性癡呆"一說。Wide - spectrum sterilizing effects. can be widely used for the sterilizing of home, hospital, restaurant, hotel., foodstuff processing, beauty and hairdressing as well as the cleaning of color stains and white clothes
本品為次氯酸鈉溶液與特殊表面活性劑復配而成,具有殺毒及洗滌作用,能迅速殺滅肝炎流感等多種傳染病毒,具有快速高效廣譜的殺菌效果,可廣泛用於家庭賓館飯店醫院食品加工美容美發等用具的消毒和有色污漬白色衣物的漂洗。Milk thistle has no major hepatotoxic effects, and n - acetylcysteine, an agent used to attenuate acetaminophen - mediated hepatotoxicity, is also unlikely to be a factor in his illness
乳薊無主要的肝毒性作用, n -乙酰半胱氨酸常用於中和對乙酰氨基酚引起的肝毒性作用,所以也不太可能是患者的致病因素。Discussion on the social educational function of the national library from constructing the harmonious society
幾種螯合劑對鎘致小鼠肝臟毒性解毒作用比較Yellow dock root is useful for many chronic and acute skin conditions. it is both cooling and detoxifying, a blood purifier aiding liver and intestinal functioning
皺葉酸模對于長期和急性的皮膚問題都很有效,它有清涼和排毒作用,可清血毒、促進肝臟和腸道功能。Prevention and cure action of hugan jiedu chongji on experimental liver fibrosis
護肝解毒沖劑對實驗性肝纖維化的防治作用The results showed that the interaction between selenium and mercury in rats mostly is the antagonism on the both acute toxicity tests and sub - chronic ones. liver, kidney, gland, heart, spleen, lungs, stomach and testis of rats are the target organs of mercury, selenium and their joint poison. and liver, kidney and spleen are three most important target organs
研究結果表明,大鼠體內硒與汞的急性和亞慢性相互作用主要表現為拮抗作用;大鼠肝臟、腎臟、胸腺、心臟、脾、肺、胃和睪丸等組織器官都是硒、汞及其聯合毒物的靶器官,其中肝臟、腎臟、脾臟是三個主要靶器官;肝臟中汞與硒的含量呈高度正相關,腎臟中汞與硒的含量呈負相關,但相關性不甚好。In order to understand the mechanism of mtx further and to investigate the genotoxic target organs, we studied the dna damage and the correlation with dose of mtx by using the alkaline single cell gel electrophoresis ( comet ) assay. liver, spleen, bone marrow, thymus, kidney, testicle, stomach and peripheral lymphocytes of mice were isolated at lh, 3h, 6h, 12h, 24h after 5mg / kg mtx intraperitoneal injection
為了進一步了解甲氨蝶呤( mtx )的作用機制,探測其作用的遺傳毒性靶器官,為應用mtx治療過程中的臨床監測和副作用防治提供理論依據,我們以小鼠為研究對象,用單細胞凝膠電泳技術檢測了mtx腹腔注射染毒1h 、 3h 、 6h 、 12h 、 24h后對肝、脾、骨髓、胸腺、腎、睪丸、胃和外周血淋巴細胞的dna損傷作用及損傷程度與mtx劑量間的關系。Among the joint dose experiments, the antagonism of selenium and mercury is the most evident in the rats with 2 : 1 mol - ratio of selenium and mercury but the least in the rats with 1 : 2 mol - ratio of selenium and mercury. the results also showed that there are two essential conditions for good antagonistic effect of selenium and mercury and 1 : 1 mol - ratio relationship of selenium and mercury in the most organs ( liver, gland, spleen, stomach and lungs ). one is that the content of mercury is more than 100nmol / g, and another is the 2 : 1 dose ratio of selenium and mercury
總體而言,雌性大鼠對硒、汞毒性的拮抗作用與雄性大鼠相比時更為敏感;相同性別的大鼠體內,聯合高劑量時硒、汞的拮抗作用最明顯,聯合中劑量時,拮抗效果相對不明顯,而聯合低劑量時,拮抗效果最差;聯合摩爾劑量比為2 : 1 ( se : hg )和汞的積累濃度大於100nmol g ,是大鼠體內大部分組織器官(肝臟、胸腺、脾、胃、肺)中四川大學碩士學位論文硒、汞等摩爾比積累的兩個必要條件,同時也是硒、汞的相互作用產生最佳桔抗效果的兩個重要前提。The results showed that within the range of the dosages of exposure, the activities of brain ache and liver esterases were found to be inhibited by treatment i - iii ( fipronil - triazophos and cd2 + ), treatment iv - vi ( fipronil - - triazophos and pb2 + ), and triazophos, and the inhibitions of the enzyme increased with the concentration of these compounds, showing a statistically significant dose - response relationship, which can, therefore, be used as a practical biomarker for combined exposure of organophosphorous ( op ) insecticides and metals
處理i一vi在24h時對ache的聯合作用基本表現為增毒作用。處理i一m和處理iv一vi對肝臟gst活性的影響趨勢基本相似, 48h時均達到誘導效應最強,隨著時間推移,表現為抑制作用。在% h時,處理v和vi對肝臟gst活性的抑制率分別為46 . 6 % 、 53 . 7 % ,明顯高於處理11和111 。After yellow aspergillus toxin enters the person ' s liver, be released to issue the part that has active by splitting decomposition, action at liver cell, have the effect that cause cancer
黃麴黴毒素進入人的肝臟后被裂解釋放出具有活性的成分,作用於肝細胞,具有致癌作用。分享友人