肝臟毒物 的英文怎麼說
中文拼音 [gānzāngdúwù]
肝臟毒物
英文
hepatotoxic agent- 肝 : 名詞(人和高等動物的消化器官之一) liver
- 臟 : 臟形容詞(不幹凈) dirty; filthy; foul
- 毒 : Ⅰ名詞1 (對生物體有害的性質或物質; 毒物) poison; toxin 2 (毒品) drug; narcotics 3 (姓氏) a s...
- 物 : 名詞1 (東西) thing; matter; object 2 (指自己以外的人或與己相對的環境) other people; the outsi...
- 肝臟 : hepar; liver肝臟創傷 liver trauma
- 毒物 : toxic; toxicant; poison; poisonous substance
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Using cd, cu, zn and cr as experimental toxicants and crucian as experimental creature, we employed the environmental biotechniques ( flame atomic absorption spectrophotometry, the method of improved pyrogallol autoxidation and electron microscopy ) to study the acute toxicity and secure concentrations of heavy metals to crucian ; the accumulation and distribution of mixed heavy metals to fingling crucian ; the activities of superoxide dismutase ( sod ) of gill and liver tissues and the hispathological and ultrastructural change of superfine structure of liver and kidney of crucian after cadmium exposure. through which, we intended to fully and systemically study the toxic influence of heavy metals to fish, and preliminarily discuss the influence of heavy metals pollution to the diversified level of fish such as individual, organ, tissue, cell and molecule. thus we tried to provide scientific evidence to establish preventative management measures, avoid and relieve the harm of heavy metals pollution to aquicolous ecosystem in time
隨后以這幾種重金屬作為實驗毒物,以鯽魚為實驗動物,應用環境生物技術、火焰原子吸收分光光度法、鄰苯三酚自氧化法、透射電鏡等技術研究了重金屬cu 、 zn 、 cd和cr對鯽魚的急性毒性及其安全濃度評價、混合重金屬在鯽魚幼體組織內的積累和分佈、 cd對鯽魚鰓和肝臟中sod活性的影響、 cd對鯽魚肝細胞和腎細胞超微結構的影響等,全面和系統地研究了水環境中重金屬暴露對魚類的毒性影響,初步探討了重金屬污染對魚類的個體? ?器官? ?組織? ?細胞? ?分子等各水平層次的影響,為制定漁業生產上預防性的管理措施提供科學依據,及時避免或減輕重金屬污染對水生生態系統造成的損害。The main study in this paper included as follows : the content and distribution of heavy metals in sediments and benthic organisms from the sewage stream in guangzhou city ; the acute toxicity and joint toxicity of mercury and selenium to swordtail fish ( xiphophorus helleri ) ; the damage of mercury to the indexes of antioxidant system in the gills and livers in swordtail ( including the measurement of the activities of total antioxidative capacity [ t ~ aoc ], superoxide dismutase [ sod ], glutathione peroxidase [ gsh - px ] and the concentration of malondiald - ehyde [ mda ] ) and the relief effects of selenium on it, as well as the physiological damage of mercury on the tissues, namely : the antagonistic effect of na + - k + ~ atpase activity on the tissues between selenite and mercury, and the ultrastructural damage under the exposure of mercury
研究內容主要有:廣州市河涌沉積物及底棲生物體內重金屬含量及評價;汞和硒對劍尾魚的急性毒性和聯合毒性及安全濃度的評價;汞對劍尾魚鰓和肝臟中抗氧化系統的毒性,包括對總抗氧化能力、超氧化物歧化酶、谷胱甘肽過氧化物酶活力及丙二醛含量的測定及硒對其保護作用;汞對劍尾魚組織生理毒性即:汞對na ~ + - k ~ + atpase活力的影響及硒的保護作用和汞和對劍尾魚組織超微結構的損傷等。以高氯酸?硝酸消化法和火焰原子吸收分光光度法測定了廣州市河涌沉積物和底棲生物中重金屬含量。Exploring the possible mechanisms is an important content of bioinorganic chemistry and ecotoxicology, which has signality in modifying contamination effect to animals. this thesis studies on the chemical speciation of mercury and selenium in dolphin livers, sub - chronic toxic effect of wistar rats of sodium selenite and mercuric chloride and investigates the antagonism mechanism from there aspects : 1. redistribution of mercury in the presence of selenium, 2
本文以海豚肝臟為對象,研究其積累的高濃度汞硒化合物的化學形態和結構,並結合動物實驗的研究,從汞硒相互作用的總體生物學表現,汞硒在動物體內分佈變化和結合形態的變化以及汞硒化合物的結構等方面探討兩元素毒性拮抗作用機理。Poiycyclic aromatic hydrocarbons ( pah ) are priority trace organic pollutants in the clrtap ( convention on long - range transboundary air poliution ) pops ( persistent organic poilutants ) protocol. both laboratory experiments and fie1d surveys unequivocally demonstrated that pah adversely affect estuarine and marine organisms. pah are implicated in the development of iesions and tumors in fish, they produce biochemical disruptions and cell damage that iead to mutations, developmental malformations and cancel although many marine organisms do not exhibit acute responses to pah " exposure, but suffer sublethal effects
本論文以湄洲灣海域和廈門西海域養殖區為研究區域,採用現代分析技術,從分子水平上系統研究了多環芳烴( pah )在海水、沉積物、養殖魚類中的含量分佈:通過現場實驗,實驗室暫養、投毒和恢復實驗系統測定了養殖魚類的肝臟、膽汁、鰓、全血等不同器官、組織的生化指標及養殖魚類肝臟、膽汁的化學指標,探討了這些指標對水環境多環芳烴污染的生物指示作用。Aflatoxin can cause both acute and chronic toxicity in animals. effects such as acute liver damage, liver cirrhosis, induction of tumours and teratogenic and other genetic effects are well documented
黃麴黴毒素可引致動物急性及慢性中毒,其影響包括急性肝臟受損肝硬化引起腫瘤形成畸胎及其他遺傳影響。Because of this, meat stays in our intestines longer than it should, and it can produce toxic substances, thus overloading the liver. consequently, this essential organ can be damaged
如果肉類在我們的腸道停留太久,就會引起有毒物質產生,而造成肝臟的過度負荷,最後造成這個重要器官的損壞。Though the clinical respiratory illness did not appear, the pathological changes were observed in lungs from the most of the challenged animals. portions of the lungs were tan in color and partly collapsed, with occasional anteroventral areas of congestion and consolidation
盡管攻毒后動物沒有表現出明顯的呼吸道癥狀,但在剖檢后發現在肺臟有不同程度的病理變化,主要表現為粉紅色肉樣變和灰色肝樣變,顯微鏡下觀察表現為!The models mentioned above not only can be used to investigate the mechanisms of drug toxicology, but also the high throughout screening of drug toxicology
上述模型除了可以應用於藥物肝臟毒性機制的研究之外,還可以用於藥物毒性的高通量篩選。New study demonstrates telbivudine s superiority to adefovir in hepatitis b treatment both newly diagnosed hepatitis b patients and those who have previously taken adefovir, a widely prescribed treatment for the disease, will see improved viral suppression with telbivudine, according to a new study presented at the asian pacific association for the study of liver apasl meeting in kyoto, japan
亞太肝臟學會( apasl )在日本京都發表最新研究報告指出,無論是乙型肝炎新癥以及一些正在服用傳統藥物阿德?偉( adefovir )的乙型肝炎病人,在服用新一代的口服乙肝藥制劑替比夫定( telbivudine )后,可更有效抑制病毒復制,為乙型肝炎患者的新選擇。Provides a method for the body to eliminate toxic substances that are broken down by the liver
提供機體一個排除由肝臟產生的毒物的方法。For the future, there is a need for better culture conditions and more complete system of the drug toxicology research
在今後的研究中,如何改善肝臟體外模型的培養條件及完善藥物肝臟毒性研究體系是急需解決的課題。The results showed that the interaction between selenium and mercury in rats mostly is the antagonism on the both acute toxicity tests and sub - chronic ones. liver, kidney, gland, heart, spleen, lungs, stomach and testis of rats are the target organs of mercury, selenium and their joint poison. and liver, kidney and spleen are three most important target organs
研究結果表明,大鼠體內硒與汞的急性和亞慢性相互作用主要表現為拮抗作用;大鼠肝臟、腎臟、胸腺、心臟、脾、肺、胃和睪丸等組織器官都是硒、汞及其聯合毒物的靶器官,其中肝臟、腎臟、脾臟是三個主要靶器官;肝臟中汞與硒的含量呈高度正相關,腎臟中汞與硒的含量呈負相關,但相關性不甚好。Hepatic transaminases alone are not a reliable marker of liver pathology, and liver histology is essential before consideration for antiviral treatment
肝臟轉氨酶本身對于肝臟的病理學改變不是一個可靠的標記物,肝活檢對于考慮進行抗病毒治療的患者是至關重要的。The virus could multifly on the mdck cells. and induce regular cytopathic effect ( cpe ). observed by negative staining electron microscope, the virus could be seen with coroniform spike. in the ultrathin sections, the viruses were found to multiply and induce inclusion body forming in the cytoplasm
本研究從某動物園兩只死亡大熊貓的肝、脾等臟器分離病毒,經mdck細胞適應培養,獲得一株病毒(命名為dxmv ) ,在該細胞上可形成有規律的病變。Aflatoxin belongs to a group of fungal toxins known as mycotoxins, and is widespread in agricultural products and food. aflatoxin is associated with both acute and chronic toxicity in animals and humans including acute liver damage, liver cirrhosis, and liver cancers. chronic toxicity associated with ingestion of low dose aflatoxin in peanuts, cereals and other at risk items are of greater concern
黃麴黴毒素是泛指一些由真菌產生的毒素,它廣泛存在於農作物及食物中,黃麴黴毒素可引致動物及人類產生急性及慢性中毒,其影響包括急性肝臟受損肝硬化及腫瘤,長期從花生穀物及其他相關的食物中攝取少量的黃麴黴毒素而引發之慢性毒害較令人關注。A variety of exogenous toxins, like alcohol, drugs and endogenous toxic metabolites are modifed by liver into less toxic or soluble forms. 3. metabolic function
體內新陳代謝產生的有毒物質如酒精及部份藥物等,都由肝臟處理,化解成為無毒、毒性較輕或容易被溶解的物質,再經膽汁或尿液排出體外。The liver also aids in the metabolism and detoxification of drugs and other foreign substances
肝臟還輔助藥物和其他外來物質的代謝與解毒。分享友人