肺氧中毒 的英文怎麼說
中文拼音 [fèiyǎngzhōngdú]
肺氧中毒
英文
oxygen toxicity-
Dna damages caused by so2 and lead acetate were studied with the single cell microgel electrophoresis technique ( or comet assay ) in order to confirm the damaging degree of lead ( as an important component of atmosphere particle matter ) on dna from male mice exposed to so2. the migrating distances of dna of brain, lung, spleen and kidney cells of mice increased significantly, compared to the control group under conditions of single and combined poisoning of so2 ( 42mg / m3 ) and lead acetate ( 0. 2 % ), and lead could strengthen dna damage degree by so2 in nuclear dna of brain, kidney, spleen cells. damaging degree of so2 on nuclear dna of lung cell of mice was more severe than that of lead
為了明確大氣顆粒物中的重要組分? ?鉛在二氧化硫所致dna損傷中的作用程度，利用單細胞凝膠電泳技術（ singlecellgelelectrophoresis ， scge ，或稱彗星實驗， cometassay ）研究了鉛與二氧化硫的聯合污染，結果表明在42mg m ~ 3so _ 2和0 . 2醋酸摘要一abstract鉛單獨及聯合染毒條件下，小鼠腦、肺、腎、脾細胞dna遷移距離均比對照顯著增加;鉛加劇了50 :對腦、腎、脾細胞核dna的損傷程度; 50 :對肺細胞核dna的損傷程度要比鉛的損傷大，小鼠肺細胞核dna遷移距離在50 :和醋酸鉛聯合作用組與醋酸鉛單獨作用組間有極顯著性差異（ p < 0 . 01 ） ，而與502單獨作用組間沒有顯著性差異。We also investigated the pathological changes of mouse liver, thymus and cerebrum cortex challenged by so2 inhalation by in vivo tests. we studied the apoptotic induction on mouse spleen cells and cytotoxicity of human embryo lung fibroblasts of so2 derivatives by in vitro tests. in vivo tests of sulfur dioxide inhalation showed : ( 1 ) effects on mouse lung of so2 challenge : we found no significant apoptotic changes induced by so2 inhalation but obvious pathological changes of lung with vacuolating of osmiophilic multilamellar bodies which maybe related with the decrease of surfacant and decrease of microvillus of type ii alveolar cells ; we also found thickening of part of basement lamina between type i alveolar cells and capillary endothelium cells which may inhibit the dispersion of oxygen and contribute to lung dysfunction
二氧化硫熏氣染毒的體內實驗結果表明，在本次實驗的濃度范圍內（ 56mg m ~ 3 、 112mg m ~ 3 、 168mg m ~ 3低、中、高三個濃度） ： （ 1 ）通過透射電鏡、 dna凝膠電泳分析和流式細胞分析發現二氧化硫吸入染毒一周對小鼠肺臟沒有明顯的凋亡誘導作用，但通過透射電鏡觀察發現二氧化硫可引起肺臟明顯的超微結構改變，引起型肺泡上皮細胞板層體空泡化，微絨毛減少，線粒體緻密化或腫脹變性；肺泡血管內皮細胞和型肺泡上皮細胞之間基膜增厚，使氧氣彌散功能出現障礙，從而降低肺功能。Clinical analysis on 6 cases of acute pulmonary edema caused by nitrogen oxides
急性氮氧化物中毒性肺水腫6例臨床分析This study was to investigate the effects of sulfur dioxide inhalation at different concentrations on some glutathione - related enzymes such as glutathione s - transferase ( gst ), glucose 6 - phosphate dehydrogenase ( g6pd ) and glutathione reductase ( gred ) in brain, lung, heart, liver, kidney and spleen of mice by the technology of biochemical toxicology. the results were showed as follows, so2 exposure at different concentrations caused the changes of glutathione redox system. moreover, the activities of antioxidative enzymes and the contents of reduced glutathione ( gsh ) were decreased significantly in different tissues at higher concentrations of soa
本研究利用生化毒理學技術研究了不同濃度二氧化硫吸入（ 22 2mg m ~ 3 ， 64 3mg m ~ 3 ， 148 23mg m ~ 3 ）對純系昆明小鼠腦、肺、心、肝、腎、脾六種組織的谷胱甘肽還原酶（ glutathionereductase ， gred ） 、谷胱甘肽硫轉移酶（ glutathiones - transferase ， gst ）和葡萄糖- 6 -磷酸脫氫酶（ glucose6 - phosphmedehydrogenase ， g6pd ）活性的影響，結果表明so _ 2吸入使小鼠不同組織的谷胱甘肽氧化還原系統發生了改變，表現為隨著so _ 2吸入濃度的增加，該系統中的抗氧化酶活性的顯著變化和抗氧化物質水平的顯著降低，且存在著組織差異性。The phase ii trial was conducted by the south african medical research council in durban, south africa, in patients with newly diagnosed pulmonary tuberculosis with and without hiv co - infection. it was designed to measure the anti - tuberculosis activity of the treatment in the first two months of therapy when compared to standard who recommended treatment and two other similar regimens which contained either ofloxacin or moxifloxacin
南非醫學研究理事會在南非德班對新近確診的肺結核患者（不論是否合併感染艾滋病毒）進行了二期試驗，旨在衡量這種療法在治療的頭兩個月中的抗結核效力，並與世衛組織建議的標準療法以及含有氧氟沙星或莫西沙星的另外兩種類似治療方案作比較。分享友人