腫瘤控制 的英文怎麼說

中文拼音 [zhǒngliúkòngzhì]
腫瘤控制 英文
cancer control journal of the moffitt cancer center
  • 腫 : Ⅰ名詞（隆起處） swelling Ⅱ動詞（突起） swell; be swollen
  • 瘤 : 名詞（瘤子） tumour
  • 控 : 動詞1 （告發;控告） accuse; charge 2 （控制） control; dominate 3 （使容器口兒朝下 讓裏面的液體慢...
  • 制 : Ⅰ動詞1 （製造） make; manufacture 2 （擬訂; 規定） draw up; establish 3 （用強力約束; 限定; 管束...
  • 腫瘤 : tumour; core （綿羊體內的）; neoplasm; phyma
  • 控制 : control; dominate; regulate; govern; manage; check; cybernate; manipulate; encraty; rule; rein; c...
  1. This commensal organism is part of the gastrointestinal tract flora and can become extremely virulent, often in the setting of immuno - suppression such as neutropenia, occult malignancy ( commonly caecal ) and poorly controlled diabetes

    這種（與人類）共生生物是胃腸道菌群的一部分，可以變得毒性巨大，通常發生於機體免疫受抑制，如中性粒細胞減少癥，隱蔽的腫瘤（一般如盲腸） ，或者控制不佳的糖尿病。
  2. Hormone-dependent tumour may sometimes be controlled by the artificial administration or deprivation of hormones

    依賴激素的腫瘤有時可用人為的服用或去除的方法得到控制。
  3. Hormone-dependent tumour may sometimes be controlled by the artificial administration or deprivation of hormones.

    依賴激素的腫瘤有時可用人為的服用或去除激素的方法得到控制。
  4. Protein kinase a is camp - dependent kinase, one of the most important signal transduction pathways, plays a pivotal role in growth, differentiation, tumor occur, cell cycle control, etc. pka activators such as camp, 8 - br - camp or phosphodiesterase ( pde ) inhibitor isomethyl butyl xanthine ( imbx ) or purified pka catalytic subunit all can inhibit germinal vesicle breakdown ( gvbd ) and meiotic maturation in mouse oocytes, also in xenopus oocytes

    蛋白激酶a （ proteinkinasea ， pka ）是依賴于camp的絲蘇氨酸蛋白激酶，是重要的信號傳導途徑之一，廣泛參與許多生命過程，包括生長、分化、腫瘤發生、細胞凋亡、細胞周期調控等。以小鼠卵母細胞為實驗對象，給予pka激動劑camp 、 8 - br - camp或磷酸二酯酶抑制劑imbx （ isomethylbutylxanthine ）均可抑制小鼠卵母細胞的胚泡破裂及減數分裂成熟。
  5. Also have due to illness, the result of parasitism of bug enroach on, " exciter " disturbed the normal dissension of plant cell, and cause a cell cannot proper motion pilot breaks up, at this moment pathological changes also can appear tumor

    也有因病、蟲侵害寄生的結果， 「刺激物」擾亂了植物細胞的正常分裂，而造成細胞無法自行控制的分裂，這時病變也會出現腫瘤。
  6. Curcumin has been shown to induce a wide variety of tumor cells by several mechanisms, mainly including modulation of the expression of oncogenes and cancer suppressor genes, down - regulation the activation of transcription factors, via many signal transduction pathways, and the modulation of cell cycle. provides an overview of domestic and international apoptosis mechanisms of malign tumor cells induced by curcumin in order to better explore and open up new avenues of cancer treatment

    姜黃素能夠誘導多種腫瘤細胞系凋亡，其機制主要是調控癌基因和抑癌基因，下調多個轉錄因子活性，通過多種信號轉導途徑，誘發細胞周期停滯而誘導細胞凋亡.對近年來姜黃素誘導細胞凋亡的機制進行綜述，以便更好地探求和開辟治療惡性腫瘤的新途徑。
  7. The mechanism of the underlying disorder of genetic control in such tumours is unknown

    這種腫瘤遺傳控制的潛在性紊亂機制尚不清楚。
  8. This treating instrument is going to the clinic experiment period. at the same time, the research work of the second generation of the instrument had begun, including the circuit design of intelligent testing temperature, safety guarding board and intelligent

    同時，第二代腦腫瘤射頻熱療儀也已開始著手研究，主要包括智能測溫電路板，安全控制板，智能控制板等部分的完善性設計。
  9. Combining high voltage new technology and power electronics technology, this paper developed a controllable steep pulse generator ( cspg ) that can control the energy of output pulse accurately. applied with this generator, a lot of cell experiments and animal experiments were carried out. based on these works, it ' s easier to research mechanism of irreversible electrical breakdown ( ireb ) of malignant tumor cell under steep pulse, and it may be helpful to generalize energy controllable steep pulse therapy ( ecspt ) on cancer treatment

    為了研究能量可控陡脈沖對惡性腫瘤細胞不可逆性電擊穿的機理，並將能量可控陡脈沖療法應用於惡性腫瘤的臨床治療，本論文研製出了一套能對輸出陡脈沖的能量進行精確控制的陡脈沖發生裝置，並應用該裝置進行了大量的醫學細胞實驗和動物實驗。
  10. Dendritic cells ( dc ) is the most powerful apc, which can markedly increase the antigen - presentation capacity by maximizing the pepitide - mhc complexes on the cell surface and upregulating the co - stimulatory ligands b7 - 1 and b7 - 2, adhesion moleculees such as il - 12 that promote full activation of lymphocytes. full activation of antigen - specific t cells requires two signals - one signal coming via the tcr and the other signal through engagment of co - stimulatary molecules. t cells receiving one signal via their tcr are turned off by mhc ( major histocompatibility complex ), via t cell cd28 binding to b7 on the dc induce tlymphokine and t cell proliferatiion

    T細胞介導的細胞免疫在控制腫瘤生長方面發揮著重要作用， t細胞在發揮抗瘤效應（分泌細胞因子和直接殺傷）之前必須先經過活化，體內專職抗原提呈細胞（ apc ）細胞並使其活化，樹突狀細胞（ dendriticcell ， dc ）為t細胞的激活提供雙重信號， t細胞藉助tcr識別由dcmhc分子遞交的抗原肽后，通過tcr - cd3復合體傳遞抗原特異性識別信號（第一信號） ，以cd28為主的t細胞表面輔佐分子識別dc表面b7分子，傳遞非特異性協同刺激信號（第二信號） ，在機體抗腫瘤免疫應答中處于核心地位。
  11. The tremendous developments in radiation therapy technology open a new prospect for improvements in local tumor control

    摘要近幾年來，由於放射治療科技的高度發展，已大幅提升了局部腫瘤的控制率。
  12. Sleep / waking cycle is a complex network modulation and many factors such as interleukin - 1 ( il - 1 ), tumor necrosis factor ( tnf ), growth hormone releasing hormone ( ghrh ), vasoactive intestina polypeptide ( vip ) and many conventional neurotransmitters such as serotonin ( 5 - ht ), acetylcholine ( ach ), norepinephrine ( ne ), dopamine ( da ) and gamma - aminobutyric acid ( gaba ) were involved in it. recent evidence has shown that no synthesized in neurons in several areas of the brain can induce the release of neurotransmitters. in the rat central nervous system, the anatomical distribution of nos - containing neurons is now well established, and it was reported that nos is co - localized with neurotransmitters well known for their involvement in sleep mechanisms, i. e. 5 - ht, ach, da and gaba

    鄭州大學2003屆碩士畢業論文gaba受體激動劑消除no合成酶抑制劑對大鼠睡/醒周期的影響睡/醒周期的形成是一個復雜的網路調控的結果，體內許多因子都參與了這一調控網路，這些因子如白介素一1 （ il一1 ） 、腫瘤壞死因子（ tnf ） 、生長激素釋放激素（ ghrh ） 、血管活性腸膚（ vip ）以及經典的神經遞質如5一輕色氨（ 5一ht ） 、乙酞膽堿（ ach ） 、去甲腎上腺素（ ne ） 、多巴胺（ da ）和卜氨基丁酸（ gaba ）等，它們在睡眠的發生和調節中也發揮著重要作用。
  13. Method used in analyzing and controlling dosage of hifu for tumor therapy

    高強度聚焦超聲腫瘤治療劑量分析和控制系統
  14. In the high risk areas of cancers, such as cervical, esophageal, gastric, liver and nasopharyngeal cancers, the secondary prevention could be carried out as more effective strategy for cancer prevention in china

    在宮頸、食管、胃、肝及鼻咽等腫瘤的高發現場，可將二級預防作為當前腫瘤控制的重要手段加以推行。
  15. Several genes and cytokines take part in the accommodation of these changes and influence the severity of brain injury, such as tumor necrosis factor a, bel - 2 gene family and ced gene family ( cystein - dependent aspartate - specific protease, caspase ). they are all has relationship with cell death after tbi and control the different cascade of cell death. there are two kinds of cell death after traumatic brain injury ( tbi ) that is necrosis and apoptosis

    腦外傷后腦組織發生的這些改變由許多基因、細胞因子參與調節並影響著損傷程度，除腦水腫外，腦損傷程度主要取決于細胞因子，這些因子包括腫瘤壞死因子- （ tumornecrosisfactor - ， tnf - ） ， b細胞淋巴瘤白血病- 2 （ b - celllymphoma leukemia - 2 ， bcl - 2 ）基因中的bcl - 2 、 bax ， ced基因家族中的半胱天冬酶（ cystein - dependentaspartate - specificprotease ， caspase ）等，它們都與腦損傷后的神經細胞死亡有關，控制著引起細胞死亡的不同層面。
  16. It is a new strategy to explore the mechanism of tumor carcinogenesis, and to regulate the network, key molecules, and drug target by combined biology effects

    用綜合生物學效應來闡明疾病發生發展機制、相關調控網路、關鍵靶分子和藥物靶點，為腫瘤的診斷、分型、藥物研製提供新思路。
  17. Chicago ( reuters ) - cancer cells often have a way of outsmarting new targeted drug therapies, but u. s. researchers said on thursday a combination of targeted drugs could shut down a tumor ' s backup plan, resulting in much more effective treatments

    芝加哥（路透社新聞）新的靶向藥物治療是癌癥細胞常常可以控制的一種方法，但是美國研究者說：在周四，靶向藥物聯合治療能攻克腫瘤支持計劃，結果好於很多有效治療。
  18. Neoplasia, or uncontrolled cellular proliferation, can result either from mutations that " turn on " the oncogenes that stimulate growth, or from mutations that result in loss of tumor suppressor genes and their products that inhibit growth

    腫瘤或失去控制的細胞增生是由於突變使癌基因啟動從而促使增生，或者由於腫瘤抑制基因和腫瘤抑制基因產物的缺失。
  19. Professor fung said, " the data from the pre - clinical studies indicates that the anti - cancer product developed by ck life sciences has the ability to strengthen the immune system in tumor - bearing mice and suppress the growth of tumors in mice in a time - and dose - dependent manner - an encouraging result indeed.

    馮國培教授指出：一系列的臨床前測試初步顯示長江生命科技之抗癌產品確能增強免疫功能，亦能在一定劑量及時間下，控制癌腫瘤的生長，這是令人鼓舞的訊息。
  20. Results and conclusion : the extracted esophageal boundary variations between adjacent time frame sequences can be applied to estimate the tumor motions as well as the required safe margins

    結論：移動評估的結果，有利於治療計畫中安全邊距的縮小，在腫瘤治療劑量增加時仍能有效控制正常組織所接受之劑量，降低正常組織副作用發生機率及其發生的嚴重程度。
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