腫脹細胞 的英文怎麼說

中文拼音 [zhǒngzhàngxìbāo]
腫脹細胞 英文
bloated cell
  • 腫 : Ⅰ名詞（隆起處） swelling Ⅱ動詞（突起） swell; be swollen
  • 脹 : 動詞1. （膨脹） expand; dilate; inflate 2. （脹大） swell; distend; bloat
  • 細 : 形容詞1 （條狀物橫剖面小） thin; slender 2 （顆粒小） in small particles; fine 3 （音量小） thin ...
  • 胞 : Ⅰ名詞1 （胞衣） afterbirth2 （同一個國家或民族的人） fellow countryman; compatriot Ⅱ形容詞（同胞...
  • 腫脹 : swelling; plumping; tympanites; puff; turgescence; turgidity; turgidness; turgor; lump; [中醫] oe...
  • 細胞 : cell; sytes; bioplast; cella; [口語] gene; [生物學] cellule; cellule cellulli cellulo ; cello ; k...
  1. Abstract : the anti - inflammatory effect of n - butanol extract of xanthocerassorbifolia bunge ( bex ) was studied in this paper. the ear edema resulted from dimethyl benzene, paw edema induced by carrageenin, the increase of vascular permeability caused by acetic acid, the chemotaxis of wbc induced by cmc and the weight of cotton granuloma in mice and hind paw edema induced by fresh egg white in rats were inhibited significantly by ig bex daily for 7 days, respectively. the carrageenin - induced paw edema was also inhibited markedly in adrenal - ectomiced mice. no changes in weight of adrenal and the concentration of vitamin c were observed, indicating that the anti - inflammatory effect of bex was not related to pituitary - adrenal system

    文摘:文冠木正丁醇提取物對二甲苯致小鼠耳腫脹、蛋清致大鼠足腫脹、角叉菜膠致小鼠足腫脹、醋酸致小鼠腹腔毛細血管通透性增加、小鼠羧甲基纖維素囊中白細胞遊走、小鼠棉球肉芽腫生長均有顯著的抑制作用，同樣劑量下，文冠木正丁醇提取物對角叉菜膠致去雙側腎上腺小鼠足腫脹仍有顯著的抑制作用，且對小鼠腎上腺重量及腎上腺中維生素c的含量沒有明顯影響。
  2. Fig. 4 anoxia reperfusion group with the addition cerium ions : mitochondria swelling and cristae vague appear in the cell. cerium deposits can be seen in the mitochondria

    圖4缺氧再灌注加鈰組：心肌細胞內線粒體腫脹，嵴模糊不清。線粒體內可見鈰沉積顆粒。
  3. Results the major morphologic changes were as follows : histologically, alveolar inflammation and interstitial fibrosis were observed. electron microscopic findings were : 1. alveolar type i cells were degenerated 、 broken - down and desquamated, endothelial cells were swelled, with inter cellular tight junction shortened, alveolar type ii cells hyperplastic, basement membrane thinned and deformed ; 2. alveolar macrophages and interstitial macrophages were hyperplastic ; 3. mast cells were infiltrated and degranulated ; 4. electron - dense deposits were present at alveolar wall ; 5. myofibroblasts 、 fibroblasts 、 collagen and basement membrane like material were hyperplastic

    電鏡觀察可見： （ 1 ） i型肺泡上皮細胞變性、崩解和脫落，內皮細胞腫脹，細胞間緊密連接短小， ii型肺泡上皮細胞增生，基底膜變薄和破壞； （ 2 ）肺泡巨噬細胞、間質巨噬細胞增多； （ 3 ）肥大細胞浸潤並見脫顆粒現象； （ 4 ）肺泡壁電子緻密物沉積； （ 5 ）肌纖維母細胞、纖維母細胞、膠原原纖維及基底膜樣物質增生。
  4. Ultrastructurally, karyopyknosis and karyolysis occured in lymphocytes, and the mitochondria, rough endoplasmic reticulum of lymphocytes in the immune organs were damaged. the nucleus were deformity, the rmtochondrial and rough endoplasmic reticulum were swelling, and the ribosomes were shed in reticular cells of thymus in zinc - deficient ducklings

    超微結構顯示缺鋅組淋巴細胞核固縮或溶解，線粒體、粗面內質網受損；胸腺網狀細胞核受損，粗面內質網擴張，線粒體腫脹、嵴斷裂。
  5. Halocin c8 appeared to have a very wide activity spectrum, including most haloarchaea and even some haloalkaliphilic rods, but it showed no inhibitory activity against bacterial strains tested. when a sensitive strain of halorubrum saccharovorum atcc29252 was exposed to halocin c8, the treated cells swelled at the initial stage, the cell wall appeared to be nicked and the cytoplasm was extruded out afterwards, and the whole cell was eventually completely lysed

    將敏感菌株halorubrumsaccharovorumatcc29252的細胞用嗜鹽菌素c8處理后，採用電鏡觀察，發現處理2小時后細胞開始腫脹、膨大，接著在細胞的兩端出現裂口，細胞內容物開始向外溢出，隨著時間延長，裂口越來越大，發生這種變形的細胞也越來越多，最後，幾乎所有的細胞完全裂解。
  6. Endothelial cells may be plump, but no atypia is present

    可有內皮細胞腫脹，但無不典型性。
  7. Because lung cancer cells may have some special hormone ( heterologous hormone ), and antigen enzyme, the role of these substances in the operation of bone joints, a result of bone and joint swelling pain, often involving the tibia, recife, ulnar and radial and bone and joints, often terminal expansion toes were clubbed fingers, x - ray radiography examination showed periosteal proliferation

    由於肺癌細胞可產生某些特殊的內分泌激素（異源性激素） 、抗原和酶，這些物質運轉作用於骨關節部位，而致骨關節腫脹疼痛，常累及脛、腓、尺、橈等骨及關節，指趾末端往往膨大呈杵狀指， x線攝片檢查可見骨膜增生。
  8. We also investigated the pathological changes of mouse liver, thymus and cerebrum cortex challenged by so2 inhalation by in vivo tests. we studied the apoptotic induction on mouse spleen cells and cytotoxicity of human embryo lung fibroblasts of so2 derivatives by in vitro tests. in vivo tests of sulfur dioxide inhalation showed : ( 1 ) effects on mouse lung of so2 challenge : we found no significant apoptotic changes induced by so2 inhalation but obvious pathological changes of lung with vacuolating of osmiophilic multilamellar bodies which maybe related with the decrease of surfacant and decrease of microvillus of type ii alveolar cells ; we also found thickening of part of basement lamina between type i alveolar cells and capillary endothelium cells which may inhibit the dispersion of oxygen and contribute to lung dysfunction

    二氧化硫熏氣染毒的體內實驗結果表明，在本次實驗的濃度范圍內（ 56mg m ~ 3 、 112mg m ~ 3 、 168mg m ~ 3低、中、高三個濃度） ： （ 1 ）通過透射電鏡、 dna凝膠電泳分析和流式細胞分析發現二氧化硫吸入染毒一周對小鼠肺臟沒有明顯的凋亡誘導作用，但通過透射電鏡觀察發現二氧化硫可引起肺臟明顯的超微結構改變，引起型肺泡上皮細胞板層體空泡化，微絨毛減少，線粒體緻密化或腫脹變性；肺泡血管內皮細胞和型肺泡上皮細胞之間基膜增厚，使氧氣彌散功能出現障礙，從而降低肺功能。
  9. Histologically, diffuse distribution of eosinopholic cardiocytes with pyknosis was noted. immunohistochemical staining for myoglobin discovered wide and diffuse loss of myoglobin throughout the ventricular myocardium. ultrastructurally, the myocardial cell with myoglobin loss were characterized by the presence of swollen mitochondria and packed cellular constituents

    組織學檢查注意到彌散性分佈伴有核碎裂的嗜酸性變性，肌紅蛋白組化染色發現整個心室肌廣泛性脫失，超微結構研究表明脫失肌紅蛋白的心肌細胞存在線粒體腫脹和細胞內成分的堆積。
  10. During the early treatment time, nuclear outer membrane was swelling, nucleolus was located near the nuclear membrane, mitochondrions were fused into a mitochondrial ball, myofibril was expanded partly

    結果表明：在汞處理初期，心肌細胞核膜突出，核仁分佈於近核膜區，線粒體聚集成線粒體球，肌原纖維有部分腫脹。
  11. A very rare case of hepatocellular carcinoma ( hcc ) extending within the common bile duct ( cbd ) and left intrahepatic duct ( ihd ) with no detectable primary hepatic tumor in a 52 - year - old taiwanese man is reported

    摘要肝細胞癌（肝癌）是國內惡性腫瘤死因中的第一位，住院的大型肝癌患者常見癥狀包括腹痛、食欲不振、體重減輕、腹脹、全身虛弱、腹部腫塊、發燒等。
  12. Fig. 3 anoxia reperfusion group with the addition cerium ions : myocardial cell structure shows vague, mitochondria swell, deposits of cerium are scattered throughout the cytoplasm, some parts appear confluent

    圖3缺氧再灌注加鈰組：心肌細胞結構不清，線粒體腫脹，胞漿內充滿大量的鈰沉積顆粒，有些部位融合成片。
  13. The results showed that, at 12 48 hrs post infection, the bursal epithelial cells bore swollen and necrotic shape. the lymphoid cells in the medulla and cortex of lymphoid follicles had various degrees of degeneration and necrosis. the lymphoid follicles showed glandular structures or cystic vacuoles owing to the prolifetation of bursal epithelium and lymphocyte depletion. lymphoid follicles from bursa at 72 144 hrs post inoculation became atropy. marked proliferation of interfollicular and interstitial connective tissue was present

    結果表明，病毒感染后12 48小時，雛雞法氏囊粘膜上皮細胞腫脹、壞死脫落，淋巴濾泡髓質部及皮質部淋巴細胞不同程度變性、壞死、排空，形成腺管樣結構或囊狀空泡，接毒后72 144小時，法氏囊淋巴濾泡淋巴細胞壞死排空，淋巴濾泡萎縮，網狀結締組織大量增生，而胚胎發育時期，法氏囊粘膜上皮腫脹變性，法氏囊淋巴濾泡形成延遲或不完整，淋巴濾泡內淋巴細胞缺乏或空虛。
  14. ( 6 ) pathological morphology : the vascular permeability, and tissue swell of cim group was slighter than the other groups in 6h after irrigation. the necrosis tissue was mixed by normal tissue ; 3d after irrigation, the speed of repair of cim was faster than the other groups. in cim, blood capillary began to form from the edge of wound

    （ 6 ）病理形態學變化:沖洗后6h ， cim組的血管通透性、組織腫脹程度較另外兩組輕，壞死組織中夾雜有較多正常組織;沖洗后3d ，組織修復加快，表現為毛細血管從傷口邊緣長入形成血液循環，與血管新生的同時，鄰近的成纖維母細胞跨越切口，將傷口連接，而後膠原繼續增加。
  15. This step - by - step, physiological narrative of patient rescues will immerse viewers in the stories that began as events unfold outside and the human body and progress inside ; teetering on the brink of death to reveal these incredible stories

    醫生原以為是並發性偏頭痛，但再次發作時，以先進的核磁共振掃描檢查才發現，雪莉其實是中風，而且大腦受損部分嚴重腫脹，壓迫到正常腦細胞。
  16. Fibroblast covered the wound and joined it. however, in the other groups, inflammatory reaction remains active, and there still were lots of degeneration and necrosis cells

    而另兩組仍處于炎癥反應的活躍階段，可見大量的組織細胞變性壞死，細胞破碎，毛細血管內皮腫脹加重。
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