致瘤病毒 的英文怎麼說

中文拼音 [zhìliúbìngdú]
致瘤病毒 英文
oncogenic virus
  • 致 : Ⅰ動詞1 （給與;向對方表示禮節、情意等） deliver; send; extend 2 （集中於某個方面） devote （one s ...
  • 瘤 : 名詞（瘤子） tumour
  • 病 : Ⅰ名詞1 （疾病; 失去健康的狀態） illness; sickness; disease; malum; nosema; malady; morbus; vitium...
  • 毒 : Ⅰ名詞1 （對生物體有害的性質或物質; 毒物） poison; toxin 2 （毒品） drug; narcotics 3 （姓氏） a s...
  • 病毒 : [醫學] virus; inframicrobe （濾過性）
  1. Common ground is told, the immune system that aids is human body is destroyed by aids virus, make human body lost counteractive ability to all sorts of pathogen of minatory life, produce a variety of infection or tumor thereby, cause a kind of of death serious infection finally

    通俗地講，艾滋病就是人體的免疫系統被艾滋病病毒破壞，使人體對威脅生命的各種病原體喪失了反抗能力，從而發生多種感染或腫瘤，最後導致死亡的一種嚴重傳染病。
  2. Scientists discovered that this particular variety of rabbit was susceptible to a fatal virus disease, myxomatosis.

    科學家們發現，這種特殊品質的兔子易患一種叫做「多發性粘液瘤」的致命病毒性疾病。
  3. Scientists discovered that this particular variety of rabbit ( and apparently no other animal ) was susceptible to a fatal virus disease, myxomatosis

    科學家們發現，這種特殊品種的兔子（顯然不包括別的動物）易患一種叫「多發性粘液瘤」的致命毒性疾病。
  4. Pp38 was found to have immunosuppressive effect on chicken in vivo and the copy number of 132 - bpr was found to be associated with the attenuation of the virus in vitro, meq was believed to be a potential oncogene, based on its leucine zipper structure and proline - rich domain characteristic of jun / fos family of transcription factors, and may plays an important role in the pathogenicity or oncogenicity of mdv

    其中， meq與132 - bpr兩個基因是mdv - 1所特有，已證實132 - bpr的拷貝數與毒株的體外致弱程度有關， pp38則被證實可抑制機體的免疫應答。 meq基因由於具有與致瘤基因jun fos家族類似的分子結構，因此，我們有理由認為meq基因在mdv致病、致瘤中可能發揮重要的作用。
  5. Differential diagnosis included cerebral toxoplasmosis, cytomegalovirus ( cmv ) encephalitis, primary cns lymphoma, progressive multifocal leukoencephalopathy, fungal abcess due to candida, aspergillus, or cryptococcus, varicella - zoster virus encephalitis or vasculitis, herpes simplex encephalitis, tuberculosis ( m. tuberculosis ), and kaposi ' s sarcoma

    其它的診斷包括：腦弓形體病，巨細胞病毒（ cmv ）型腦炎，原發中樞系統淋巴瘤，漸進性多灶性腦白質病，假絲酵母菌，麴菌或隱球菌所致真菌性膿腫，水痘帶狀病毒型腦炎或脈管炎，單純皰疹腦炎，肺結核（多發性結核） ，和卡波西肉瘤。
  6. Infectious agents such as human papillomavirus can lead to cellular transformation as well

    感染性因素如人類乳頭狀瘤病毒能導致細胞轉化。
  7. Apoptin ( vp, ) can selectively induce apoptosis in human transformed and malignant cells but not in normal diploid cells. apoptin induces a p53 - independent, bcl - 2 - insensitive type of apoptosis in various human tumor cells

    其中， vp _ 3蛋白質是雞貧血病毒的致病因子，能選擇性的誘導腫瘤細胞和轉化細胞發生凋亡，但對人和鼠的正常二倍體細胞無影響，稱為凋亡素（ apoptin ） 。
  8. The env protein deduced from env gene encodes the hydrophilic surface protein ( su ) and the hydrophobic transmembrane domain ( tm ) that determine the specific interaction between virus particles and cell surface receptors during retroviral entry. the su of retroviruses is a highly variable genetic element, containing receptor binding sites and major antigenic determinants. exjsrv - specific dna probes were derived. by using these dna probes in tissue hybridization. we successfully identified jsrv mrna expression and proviruses dna in sheep lung tissues infected with jsrv and control group has no postive signals, validating the use of exogenous virus - specific dna probes in the analysis of oncogenic proviral integration sites and identification of integrated exogenous proviral sequences

    用地高辛隨機引物法標記exjsrv特異的env片段，制備探針，原位雜交檢測spa肺組織中的rna及前病毒dna ，結果表明spa患羊肺組織內有jsrvenv基因mrna的表達，同時也檢測到了前病毒dna ，而相應的陰性對照卻無陽性信號，證實外源性病毒特異的dna探針在致瘤性前病毒的整合位點和整合的外源性前病毒的檢測中具有可信度。
  9. A gene that causes the transformation of normal cells into cancerous tumor cells, especially a viral gene that transforms a host cell into a tumor cell

    致癌基因導致正常細胞轉變為癌變細胞的基因，尤其是一種把寄主的細胞變為腫瘤細胞的病毒基因
  10. Professor anthony chan, director of the hong kong cancer institute and chairman of the department of clinical oncology at the chinese university of hong kong, and professor qian tao, head of the cancer epigenetics and tumour virology laboratory in the sir yk pao centre for cancer, have successfully demonstrated for the first time in a group of npc patients who have failed standard therapies, that demethylation of the ebv genome can be achieved using azacytidine, a study published in the journal of clinical oncology with a leading editorial describing the work as " a model for future trials in solid tumors "

    香港中文大學腫瘤學繫系主任兼香港癌癥研究所總監陳德章教授及腫瘤學系副教授陶謙教授，最近首次成功地在一組常規治療已經無效的鼻咽癌患者身上，利用氮胞? （ azacytidine ）達致eb病毒基因的去甲基化，有關研究報告發表在《臨床腫瘤學報》上，而該期的一篇社論更形容是項研究是將來腫瘤臨床試驗的典範。
  11. It is well known that tumor necrosis factor a ( tnfa ) can play very different character in defence mechnism of body and pathologic injury, in which it can inhibit or kill tumor cell, and induce inflammation against infection ; on the other hand, tnfa can be as a very important mediator to cause some serious pathologic processes such as septic shock, gvhr and some autoimmune diseases

    腫瘤壞死因子（ tumornecrosisfactor ， tnf ）具有對某些惡性腫瘤的抑制或殺傷作用，也是致內毒素性休克、急性移植物抗宿主病及一些自身免疫病的重要效應因子，因此tnf的改造和tnf拮抗劑的研究受到各國學者的重視。
  12. Aids is a severe immunological disorder caused by the retrovirus hiv, resulting in a defect in cell - mediated immune response that is manifested by increased susceptibility to opportunistic infections and to certain rare cancers, especially kaposi ' s sarcoma

    愛滋病是一種由逆轉錄酶病毒引起的嚴重免疫系統混亂，導致細胞中介免疫反應的缺失，表現為容易受感染和受某些罕見癌癥的侵襲，尤其是卡波濟氏腫瘤。
  13. The pathogenesis of gastrointestinal tract infected with hiv

    卡波西肉瘤相關的皰疹病毒致病分子機制
  14. The comparative analysis of sequences indicated that the sequences of meq in different pathotypes are relatively conserved and the homology of the amino acid sequences is very high. the significant differences include two mutations in both mdv - 1 vaccine strains cvi988 / rispens and 814 strain : the deletion of a proline ( no. l93aa ), and this mutation is just exactly located in a 15 - amino - acid ( eelcaqlcstppppi ) repeat sequence within the c - terminal transactivation domain of meq protein ; and a point mutation with a shift from alanine ( a ) of all virulent strains to serine ( s ) was occurred on the no. 71 aa

    結果發現， mdv不同致病型的meq基因序列相對比較保守，它們相互間核苷酸和氨基酸序列的同源性均很高；但是，與所有七個致瘤性的mdv毒株相比，二個型弱毒疫苗cv1988 rispens株和814株均出現了兩個特徵性的突變：即第194位的p缺失性突變和第71位氨基酸由a變成了s的位點突變；缺失性突變恰恰位於meq基因中轉錄激活域內的一個多脯氨酸的重復序列（ pppp ）之中。
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