血管重塑 的英文怎麼說

中文拼音 [xiěguǎnzhòng]
血管重塑 英文
vascular remodeling
  • : 血名詞(血液 多用於口語) blood:吐血 spit (up) blood; 血的教訓 a lesson paid for [written] in b...
  • : Ⅰ名詞1 (管子) pipe; tube 2 (吹奏的樂器) wind musical instrument 3 (形狀似管的電器件) valve;...
  • : 重Ⅰ名詞(重量; 分量) weight Ⅱ動詞(重視) lay [place put] stress on; place value upon; attach im...
  • : 動詞(塑造) model; mould
  • 血管 : blood vessel; vascellum; vessel; rete
  • 重塑 : remodeling
  1. Anp is a circulating hormone with asodilatory and diuretic properties that inhibits the renin - angiotensin - aldosterone system, which has been suggested to modulate cardiac sympathetic nere actiity and cardiac remodeling

    Anp是循環系統中的一種激素,其舒張和利尿特性能夠抑制腎素?緊張素?醛固酮系統。普遍認為腎素?緊張素?醛固酮系統能夠調節心臟交感神經活性和心臟
  2. Significance of main matrix metalloproteinase in cardiovascular matrix remodeling

    主要基質金屬蛋白酶在心基質中的意義
  3. Exercise could lead to remodeling of icn, of which normal exercise load might lead to proportional increase of cardiac and icn, adapting oneself ot transmit of contractility between myocardial bundles and cardiac muscle cell ( cmc ) under conditions of myocardial hypertrophy ; and exercise overload might lead to increase of a large number of different collagen among myocardial bundles, cmc, arteriole and capillary, that could lead to increase of stiffness and descent of systolic and diastolic function of heart

    運動可引起心肌間質膠原網路結構的,一般運動負荷可使心肌與間質膠原網路同步增長,以適應心肌細胞肥大收縮力增加從而保證肌束間及心肌細胞間力的傳遞;運動超負荷可使心肌束間、心肌細胞間以及小動脈和毛細間不同類型膠原大量蓄積增生,導致心臟的僵硬度增加,舒縮功能降低。
  4. The pathophysiologic mechanism of ppar and its activators on ventricular remodeling and heart failure

    頸動脈粥樣硬化斑塊形成及其血管重塑的力學機制
  5. The result of the study demonstrate the pt autograft which have blood supply can promote the reversion of biomechanics properties, the action mechanism is maybe related with the ischemic necrosis and revascularization of the graft. transplanted autogenous patellar - tendon grafts undergo a process of ischemic necrosis, revascularization, proliferation, and remodeling

    其作用機理可能與移植物缺,壞死和再化有關,移植物在向前交叉韌帶樣變過程中,移植物的修復均經歷缺壞死,建,細胞增殖,再型期。
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