血脫證 的英文怎麼說

中文拼音 [xiětuōzhèng]
血脫證 英文
syndrome of blood depletion
  • 血 : 血名詞（血液 多用於口語） blood:吐血 spit （up） blood; 血的教訓 a lesson paid for [written] in b...
  • 脫 : Ⅰ動詞1 （脫落） cast; shed; drop; fall off 2 （取下; 除去） take off; strip; cast off 3 （脫離） ...
  • 證 : Ⅰ動詞（證明） prove; verify; demonstrate Ⅱ名詞1 （證據） evidence; proof; testimony; witness 2 （...
  1. Chronic hepatitis b tcm syndrome type peripheral blood mononuclear cells

    慢性乙型肝炎中醫證型外周血單個核細胞乙肝病毒脫氧核糖核酸
  2. But recent eidence suggests that drug - eluting stents carry a risk of producing potentially dangerous blood clots

    但是最近的證據顯示藥物洗脫支架具有潛在的導致血液凝集的危險性。
  3. Ren shen can generates fluids and reduce thirst, for xinqixu ( heart qi deficient ) palpitations with instant sweating and anxiety, insomnia, dizziness / headache, forgetfulness, impotence, diabetes, bleeding in the vagina not during period, seizures in children, chronic weakness, restlessness. any qixu ( qi deficient ) xue xu ( blood deficient ) or fluid deficient syndrome

    用於心氣虛之陣跳，倦怠，反胃吐食，大便滑泄、虛咳喘促，自汗暴脫、驚悸、健忘、眩暈頭痛、陽痿尿頻、消渴、婦女崩漏，小兒慢驚，及久虛不復，不安，一切氣虛血虛津液不足之證。
  4. To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation

    為了驗證上述假設，本文分別在整體和離體水平，採用大腦中動脈缺血（ middlecerebralarteryocclusion ， mcao ）和體外氧?糖剝奪（ hypoxiaandglucose - deprivation ， hgd ）缺血模型，首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況，觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的磷酸化水平變化，然後通過外源性給予vegf蛋白，在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白磷酸化水平的影響，最後利用vegf反義脫氧寡核苷酸（ oligodeoxynucleotide ， odn ）特異阻斷內源性vegf蛋白的表達，觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白磷酸化中的作用，以進一步明確vegf缺血保護效應與其調節kv1 . 2蛋白磷酸化之間的關系。
  5. Objective to study the relationship between tcm syndrome type and hbvdna in serum and peripheral blood mononuclear cells pbmcs in chronic hepatitis b patients

    目的探討慢性乙型肝炎患者血清乙肝病毒脫氧核糖核酸hbvdna及外周血單個核細胞pbmcs中hbvdna感染與中醫證型的關系。
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