骨節作響 的英文怎麼說
中文拼音 [gǔjiézuòxiǎng]
骨節作響
英文
sounding of the joints-
Hiding essence of life is the main physiology function of kidney, the essence in kidney is angry, be airframe life activity this, to airframe the physiology activity of each respect all is having all in all effect ; kidney advocate water fluid, basically be the aerification function that the essence in pointing to kidney enrages, to the body fluid inside body be defeated cloth and excrete, maintain the balance that the body fluid inside body metabolizes, the adjustment with rising very important action ; kidney advocate bone gives birth to pith, it is spirit of the energy in kidney has stimulative airframe to grow actually a of growth function main component, the rise and fall that the essence in kidney enrages, affect the growth of bone and growth not only, and the plentiful that also affects spinal cord and encephalon and development ; " tine is bone ", tine and bone give one cause together, the tooth also is filled by institute of gas of the energy in kidney raise, spirit of the energy in kidney is abundant, fall off the tooth is solid and not easily, if kidney is medium air of essence of life is inadequate, criterion the tooth becomes loose easily, inchoate even fall off ; kidney have one ' s ideas straightened out at 2 shade ( external genital organs and anal ), accordingly, frequent micturition, enuresis, urinary incontinence, make water little or anuria, all the aerification function with kidney is wrong about
藏精是腎的主要生理功能，腎中精氣，是機體生命活動之本，對機體各方面的生理活動均起著極其重要的作用；腎主水液，主要是指腎中精氣的氣化功能，對于體內津液的輸布和排泄，維持體內津液代謝的平衡，起著極為重要的調節作用；腎主骨生髓，實際上是腎中精氣具有促進機體生長發育功能的一個重要組成部分，腎中精氣的盛衰，不僅影響骨的生長和發育，而且也影響脊髓和腦髓的充盈和發育； 「齒為骨之餘」 ，齒與骨同出一源，牙齒也由腎中精氣所充養，腎中精氣充沛，則牙齒堅固而不易脫落，若腎中精氣不足，則牙齒易於松動，甚至早期脫落；腎開竅於二陰（外生殖器和肛門） ，因此，尿頻、遺尿、尿失禁、尿少或尿閉，均與腎的氣化功能失常有關。In this thesis, four types of steel beam - rectangular cfst column connections, including normal welded flange plate ( wfp ) connection, bolted flange plate ( bfp ) connection, stiffened end plate ( sep ) connection and double split - tee plate ( dst ) connection, were designed based on the configuration of steel frame connection. total 8 models, 2 models of each kind of connections were tested under low - reversed cyclic loading at the end of cfst columns. the relationships between force and displacement at the end of columns, the relationships between the moment and rotation of the joints, degradations of strength and stiffness, ductility, failure mechanism and failure characteristics of these four connections under different axial - compression ratios were presented
本文借鑒鋼框架節點構造，設計了四種類型矩形鋼管混凝土柱與鋼梁連接節點，包括常規栓焊（ wfp ）節點、翼緣全螺栓連接（ bfp ）節點、雙t板連接（ dst ）節點以及加勁端板連接（ sep ）節點，進行了四類節點8個模型試件在柱端低周反復荷載作用下的抗震性能試驗研究，比較了不同軸壓比下節點的滯回性能、強度與剛度退化、延性、破壞機理與破壞特徵，主要結論有： 1 、節點的位移滯回曲線與轉角滯回曲線為塊型分佈，沒有或略有捏攏現象，耗能能力強； 2 、軸壓比對節點滯回曲線有顯著影響，全部節點都有顯著的剛度退化； 3 、位移與轉角骨架曲線在峰值荷載後有較長的水平或下降段，具有良好的延性性能； 4 、從整體抗震性能上看，翼緣全螺栓連接節點、雙t板連接與加勁端板連接節點都優于常規栓焊節點，可在實際工程中加以推廣。Pseudo - static force testing of 1 : 2 scale models of a steel frame with dog - bone connections is first carried out in china in order to obtain elastic - plastic behavior of dog - bone connections steel frames under cyclic loading as well as to analyze collapse characteristic after structural ultimate strength has lost. the influence of dog - bone connection on the coming order of plastic hinges and failure characteristic of steel frame, and the influence of dog - bone ' s shape and position on the behavior of structure is obtained
通過試驗考察了狗骨式連接平面鋼框架結構在循環荷載作用下的彈塑性性能;了解狗骨式連接對結構塑性鉸出現的順序以及結構破壞形態的影響;了解節點處與狗骨處應力發展狀況，探討狗骨削弱的形狀和削弱位置對節點性能的影響。Therefore zo - 2 established a linker between tight junction and cytoskeleton. zo - 2 may regulate the formation or function of tight junction and contact between tight junction and cytoskeleton
因此zo 2的生物學作用可能參與緊密連接形成和功能調節，經細胞骨架而影響細胞的形態和運動。The results suggested : the differential gene expression in the adrenal gland under acute hyperglycemia / hypoglycemia was related to blood glucose ; these genes might regulate blood glucose by influencing the metabolism and secretion of hormones, cytoskeleton constitution and signal transduction in the adrenal gland ; bbs4 gene might participate in glucoregulation via the activities of adrenal medulla ; the differential genes expressed under acute vs chronic hyperglycemia might play a basic role of the structural and functional changes in the adrenal gland, lead to the process of chronic complications of diabetes or accommodate to the status of hyperglycemia
結果提示：腎上腺基因差異性表達和血糖變化有關；在急性高低血糖情況下，這些基因可能通過影響腎上腺內激素的分泌和代謝、細胞骨架構成和信號轉導來調節血糖； bbs4可能參與血糖調節，其作用可能和腎上腺髓質的活動有關；慢性高血糖情況下的腎上腺差異表達不同於急性高血糖，這種表達差異可能與腎上腺慢性的結構和機能改變相關，或參與了糖尿病慢性並發癥的發生發展、高血糖狀態的調節。分享友人