cell necrosis 中文意思是什麼

cell necrosis 解釋
細胞壞死
  • cell : n 1 小室，單室；隔間，艙；〈詩〉茅舍；（單個的）蜂窩，蜂房。2 〈詩〉墓穴，墓。3 （大修道院附屬的...
  • necrosis : n. （pl. -ses ） 【醫學】壞死,壞疽；骨疽；【植物；植物學】枯斑,壞死。necrotic adj.
  1. The b lymphocyte stim lator ( blys ) also known as baff, tall - 1, thank, ztnf4, is the most recent addition to the tumor necrosis factor family ( tnf ) ligands. blys induces both in vivo and in vitro b cell proliferation, differentiation and immunoglob lin secretion. meanwhile, it also strongly suppresses the growth of tumor cell lines. the over - production of blys is associated with the development of certain autoimmune disease, such as systemic lupus erythaematosus ( sle ), rheumatoid arthritis, myasthenia gravis, sj greh ' s syndrome

    人b淋巴細胞刺激因子（ hblys ）是1999年發現的腫瘤壞死因子超家族成員。最早發現其可誘導腫瘤細胞凋亡，之後發現它是一個強有力的b細胞共刺激因子，它在體外及轉基因動物體內可明顯刺激b細胞的生長，免疫球蛋白分泌，並導致動物自身免疫性改變。
  2. Tumor necrosis factor ( tnf ), a cytkine that exerts many pro - atherosclerotic effects in vivo, causes up - regulation of acat - 1 gene expression in human blood monocytes. by luciferase activity assay, tnf - a enhanced acat - 1 p7 promoter activity in thp - 1 monocytic cell line

    細胞因子tnf -即腫瘤壞死因子大量存在於人動脈粥樣硬化斑塊中，本實驗通過熒光素酶活性和rt - pcr測定結果表明， tnf -處理人血單核細胞和單核細胞株thp - 1均增強acat - 1基因p7啟動子活性。
  3. The histologic lesions were characterized by the remarkable atrophy of intestinal villi, basal vacuolation of enterocytes, increment of the crypt and the villus - to - crypt ratio had significantly decrease. compared with that of noninoculated control chicks. besides. intestinal villi become thin and flat. or cubic, also accompanied with necrosis. many villous enterocytes were detached from the lamina propria and the lamina propria contained a diffuse infiltrate of lymphocytes, plasma cells, macrophages. the characteristic ultrastructural changes observed by electronmicroscope were swelled mitochondria and dilated endo - plasticreticulum. ribosome on the surface of endo - plasticreticulum fall off and the number of ribosome within the cell decrease

    動物回歸試驗中接種兩周齡spf雞， 24h后陸續發病，表現為持續性水樣腹瀉；剖檢可見病雞脫水、小腸內有大量的液體和氣泡、腸粘膜變薄；組織學變化為腸絨毛上皮壞死、脫落，絨毛平均長度減少而隱窩深度增加，固有層中淋巴細胞浸潤。其臨床癥狀及病理組織學變化與自然發病相同。
  4. The characteristics of this method are : a, directly counting cell number without the influence of the metabolic state of the cells ; b, discrimination of target cells from effector cells in cell - mediated cytotoxicity assay ; c, less treatment step, and free - radioactivity ; d, high sensitivity and reliability. 2, using the above assay, immunofluorescent labeled technique, and flow cytometry, the pbmc proliferation, apoptosis, necrosis, cell cycle, activation, cytokines and membrane marker were detected. the results showed that the number of pbmc reduced, but the activity of pbmc increased dose - dependently ; the reduction of cell number resulted from necrosis and apoptosis ; the supernatant of k562 cell lines were not able to block the cell cycle, but to promote it ; the ratio of t cell subset and the expression of thl and th2 cytokines increased

    結合以上創建的方法和免疫熒光流式細胞術，用k562細胞株可溶性分泌物（上清）對外周血單個核細胞（ pbmc ）進行培養以模擬體內微環境，然後分別從細胞增殖、凋亡、壞死、細胞周期、活性、細胞因子和表面抗原表達等方面進行研究，結果發現用腫瘤上清培養的pbmc細胞數量下降明顯，但同時對其有激活作用，且呈劑量依賴性；細胞數的下降主要是由細胞壞死和凋亡引起的，腫瘤上清對細胞周期沒有阻斷作用，反而略有促進作用； t細胞亞群比例增加，並促進表達th1 、 th2細胞因子。
  5. There is lobular disarray with focal hepatocyte necrosis, giant cell transformation, lymphocytic infiltration, kupffer cell hyperplasia, and cholestasis ( not seen here )

    圖中可見具有病灶肝細胞壞死、巨細胞轉化、淋巴細胞浸潤、枯否細胞增生和膽汁淤積（此圖未顯示）的肝小葉紊亂。
  6. This large renal cell carcinoma shows cyst formation and foci of hemorrhage and necrosis

    本例腎細胞癌瘤體巨大，伴發小囊腫形成、局灶性出血和壞死。
  7. A mononuclear inflammatory cell infiltrate extends from portal areas and disrupts the limiting plate of hepatocytes which are undergoing necrosis, the so - called " piecemeal " necrosis of chronic active hepatitis

    單核細胞浸潤從門脈區延伸，破壞正在壞死（也稱為慢性活動性肝炎的「碎片狀」壞死）的肝細胞界板。
  8. Several genes and cytokines take part in the accommodation of these changes and influence the severity of brain injury, such as tumor necrosis factor a, bel - 2 gene family and ced gene family ( cystein - dependent aspartate - specific protease, caspase ). they are all has relationship with cell death after tbi and control the different cascade of cell death. there are two kinds of cell death after traumatic brain injury ( tbi ) that is necrosis and apoptosis

    腦外傷后腦組織發生的這些改變由許多基因、細胞因子參與調節並影響著損傷程度，除腦水腫外，腦損傷程度主要取決于細胞因子，這些因子包括腫瘤壞死因子- （ tumornecrosisfactor - ， tnf - ） ， b細胞淋巴瘤白血病- 2 （ b - celllymphoma leukemia - 2 ， bcl - 2 ）基因中的bcl - 2 、 bax ， ced基因家族中的半胱天冬酶（ cystein - dependentaspartate - specificprotease ， caspase ）等，它們都與腦損傷后的神經細胞死亡有關，控制著引起細胞死亡的不同層面。
  9. It is well known that tumor necrosis factor a ( tnfa ) can play very different character in defence mechnism of body and pathologic injury, in which it can inhibit or kill tumor cell, and induce inflammation against infection ; on the other hand, tnfa can be as a very important mediator to cause some serious pathologic processes such as septic shock, gvhr and some autoimmune diseases

    腫瘤壞死因子（ tumornecrosisfactor ， tnf ）具有對某些惡性腫瘤的抑制或殺傷作用，也是致內毒素性休克、急性移植物抗宿主病及一些自身免疫病的重要效應因子，因此tnf的改造和tnf拮抗劑的研究受到各國學者的重視。
  10. Microscopically, graft versus host disease is one of the best examples of a process called " apoptosis " or single cell necrosis

    鏡下，移植物抗宿主反應是說明「程序性細胞死亡」或單個細胞壞死（即調亡）過程的最好例子。
  11. Apoptosis is a more orderly process of cell death in which there is individual cell necrosis, not necrosis of large numbers of cells

    細胞凋亡是一種程序性細胞死亡，屬于單個細胞死亡，而不是大量細胞死亡。
  12. These results indicated low concentration of no is necessary for cell physiological activities, whereas high concentration of no will impare cell physiological activities and even result in cell necrosis

    這說明低劑量的no是pbmc細胞生理活動所必須，而高濃度no會損害pbmc細胞甚至導致死亡。
  13. Expression of p53 and bcl - 2 proteins in the cell apoptosis of adenoid cystic carcinoma induced by tumor necrosis factor - alpha tnf

    450表氧化酶對腫瘤壞死因子誘導的內皮細胞凋亡的影響
  14. We introduce a new method of autologous stem cell transplantation for the avascular necrosis of femoral head and believe it is an effective pathway in treatment

    本文介紹的採用自體骨髓幹細胞移植技術為早期股骨頭缺血性壞死的治療提供了另一新的更為有效的治療途徑。
  15. O. osmm of snp could activate the pbmc, while above 0. 5 mm of snp could significantly induce pbmc necrosis, nevertheless, the higher concentration of snp could not induce significant change of cytokine production, cell cycle, and ratio of pbmc subsets

    0刀smm的swh有活化pbmc的作用，大於0 sinm的snp會大量誘導pbmc的壞死，但對細胞因子、細胞周期、細胞亞群的比例均影響不大。
  16. Effect of cytochrome p450 expoxygenases on vessel endothelial cell apoptosis induced by tumor necrosis factor alpha

    腫瘤壞死因子凋亡誘導配體誘導白血病細胞凋亡中抵抗機制的初步探討
  17. Cell death by apoptosis and necrosis

    凋亡和壞死引起的細胞死亡
  18. The high concentration of no production induced by k562 cell lines supernatant may only play the role of induction of pbmc necrosis

    在k562上清對pbmc作用中，產生的高濃度no所起的作用也許僅僅是誘導了pbmc的壞死。
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