death gene 中文意思是什麼
death gene
解釋
死亡基因-
The characteristics of tm - 22 expression presented in transgenic tobacco : 1 ). virus specificity in either homozygote or heterozygote ; 2 ) tm - 22 gene integrated in tobacco genomic dna with single copy and in inheritance and segregation to progenies on the mendel role ; 3 ). transgenic line with tm - 22 promoter ( ptm47 ) showed infected symptoms with cell death distinguished to one with 35s promoter ( ptm49 ) after inoculation with tomv - 2a
其次，通過氨基酸序列和結構的比較，確定tm - 2 ~ 2基因的編碼蛋白與tomv病毒在抗病反應中相互識別的特異氨基酸及其功能；然後，應用重組dna技術，互換tm - 2 ~ 2基因和tm - 2基因的對應結構域，構建嵌合基因，獲得嵌合蛋白表達的轉化體，驗證tm - 2 ~ 2編碼蛋白中變異氨基酸的作用。 -
Gene for best vitelliform macular dystrophy. dr. dorothy shu - ping fan, associate professor of the department of ophthalmology & visual sciences of cuhk, and director of genetic counseling of lim por - yen eye genetics research centre, remarked, " retinoblastoma can cause not only bilateral blindness but also death if not treated early enough
香港中文大學眼科及視覺科學學系副教授兼林百欣遺傳眼疾研究中心遺傳輔導主任范舒屏醫生指出：視網膜胚細胞瘤若未能及早發現，不單可以引致雙目失明，更可導致死亡。 -
These included mutations in the sodium channel gene scn5a, preiously associated with lqts and other unstable heart rhythm conditions and sudden death
這其中包括鈉通道基因scn5a的突變，該基因以前發現與lqts 、其他不穩定心律失常以及猝死相關。 -
Several genes and cytokines take part in the accommodation of these changes and influence the severity of brain injury, such as tumor necrosis factor a, bel - 2 gene family and ced gene family ( cystein - dependent aspartate - specific protease, caspase ). they are all has relationship with cell death after tbi and control the different cascade of cell death. there are two kinds of cell death after traumatic brain injury ( tbi ) that is necrosis and apoptosis
腦外傷后腦組織發生的這些改變由許多基因、細胞因子參與調節並影響著損傷程度，除腦水腫外，腦損傷程度主要取決于細胞因子，這些因子包括腫瘤壞死因子- （ tumornecrosisfactor - ， tnf - ） ， b細胞淋巴瘤白血病- 2 （ b - celllymphoma leukemia - 2 ， bcl - 2 ）基因中的bcl - 2 、 bax ， ced基因家族中的半胱天冬酶（ cystein - dependentaspartate - specificprotease ， caspase ）等，它們都與腦損傷后的神經細胞死亡有關，控制著引起細胞死亡的不同層面。 -
This strain ' s virulence was judged by mean death time of chick embryos ( mdt ), intracerebral pathogenicity index in day - old chicks ( icpi ) and intravenous pathogenicity index in 6 - week - old chickens ( ivpi ) and it was found to be the virulent strain. at last, it was tested by the recurrent infection and found that it was the newcastle disease virus ( ndv ), and it was named hbg - 1 strain. in order to find the difference of the cleavage site of this strain with f48e9 and ? 30 strain, a part of the cleavage site of fusion protein gene fragment was amplified by rt - pcr using a primer and sequenced. the sequence analysis showed this strain had low homology with f4ge9 and cso. a phylogenetic tree based on the published sequences of ndv reference strains was constructed and showed the isolated strain hbg - 1 belonged to the genotype w ndv, a novel genotype ndv
為了進一步探尋分離株與標準株的異同，又採用rt - pcr方法，擴增獲得分離株f _ o裂解位點附近的基因片段，經測序后與國際上已發表的新城疫病毒的核酸序列進行比較，結果表明其與標準株和疫苗株之間的同源性較低，僅為82 86之間。經系統發育進化樹分析后，判定該分離株為新城疫病毒（ ndv ）基因型。運用計算機軟體對其裂解位點處的氨基酸序列進行預測和分析，結果表明該分離株為新城疫病毒強毒株並具有基因型的典型結構特徵。 -
Programmed cell death ( pcd ) in plants is an active and gene - regulated process which plays an important role in normal growth and development of plants
摘要植物細胞程序化死亡（ pcd ）是一種由基因控制的、主動的細胞死亡過程，它在植物正常生長發育過程中起著重要作用。 -
Latest research indicates that long - qt syndrome ( lqts ) gene ariants contribute to sudden infant death syndrome ( sids )
最新的研究表明，長qt綜合征基因變異導致嬰兒猝死綜合征。 -
We concluded that excessive expression of exogenous htr gene may compete with the endogenous telomerase rna and prevent rna template from combining with telomeric dna, thus repressing the elongation of telomeric dna ( telomeres ) and causing cell aging and cell death. - 6 - 3. some modifications have been made to overcome the limitation of conventional telomeric repeat amplification protocol ( trap ) assay
分析其原因，可能是htr基因的過表達在數量和空間效應上同細胞內的端粒酶rna組分產生竟爭，一定程度上阻礙了端粒酶rna模板區與端粒dna的結合，從而抑制端粒dna的延伸，導致細胞凋亡。 -
We identify, for the first time, a number of gene candidates that are induced by dht with or without ischemia, many of which could account for cell death through enhanced inflammation, dysregulation of blood - brain barrier and the extracellular matrix, apoptosis, and ionic imbalance
我們首次發現，許多候選基因在有無缺血時都由dht誘導，這其中的許多基因能夠解釋通過增加炎癥、血腦屏障和細胞外基質失調、凋亡和離子失衡等原因產生的細胞死亡。 -
Tracking the evolutionary path of genes and pseudogenes helps molecular biologists to uncover instances of gene birth and death just as the study of mineral fossils tells paleontologists about the creation and extinction of species
追蹤基因和偽基因的演化路徑，讓分子生物學家得以了解基因的出生和死亡，就像古生物學家利用化石來推斷物種的誕生和滅絕一樣。
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