endothelial tumor 中文意思是什麼

endothelial tumor 解釋
內皮瘤
  • endothelial : 內皮的
  • tumor : n. 【醫學】腫瘤,癌,疙瘩,贅疣。 a benign [malignant] tumor 良[惡]性腫瘤。adj. -ous 腫瘤的;誇張的 (tumorous growth 【農業】陡長)。
  1. Now it has been found the recruitment and homing of circulating endothelial cells ( cecs ) or their progenitors ( ceps ) released from the bone marrow is very important in tumor angiogenesis. we detected the cecs and ceps in 64 pb ( peripheral blood ) of tumor patients. we found the amount of cecs and ceps is higher in tumor patients than normal controls

    本項目應用流式細胞法對64例腫瘤患者及15例正常人,外周血中的循環血管內皮細胞( circulatingendothelialcell , cec )以及骨髓來源的循環血管內皮前體細胞( circulalingendothelialprecursor , cep )進行了檢測,證實了外周血中cec / cep的存在,發現腫瘤患者cec / cep含量高於正常對照組,提示腫瘤患者體內存在血管內皮及其前體細胞的動員。
  2. Studies of the crystal structure of endostatin have shown a compact globular fold, with one face particularly rich in arginine residues acting as a heparin - binding epitope, this site was recently shown to be involved in the inhibition of induced angiogenesis. experimental studies show that recombinant endostatin specifically inhibits the proliferation of endothelial cells in a dosedependent fashion. recombinant endostatin from bacteria is largely insoluble, but still efficient in arresting tu mor growth after injection into mice. intermittent therapy with recombinant bacterially produced endostatin reduces several experimental tumors, including lewis lung carcinoma, to a dormant state. no sign of drug induced resistance has been reported and, in the original study, the treatment dormancy appeared to persist even when therapy was discontinued. sowe regard endostatin as a promising anti - tumor drug

    許多研究表明重組內皮抑素特異性抑制內皮細胞增殖,而且這種抑制作用呈劑量依賴性。細菌表達產物內皮抑素大部分以不溶形式存在,將這種混懸液注射治療老鼠仍可以抑制腫瘤生長。于小鼠皮下重復注射內皮抑素重組蛋白,幾乎完全抑制鼠lewis肺癌等多種腫瘤生長,並無耐藥性產生,即使中斷治療腫瘤也不再復發。
  3. Vascular endothelial growth factor and the effects of endostatin on the inhibition of tumor angiogenesis in treating hepatocarcinomas

    血管內皮生長因子和內皮抑素在肝癌抗腫瘤相關血管治療中的作用
  4. No, a first gas information molecule discovered in human being, is a typical endothelial - derived relaxant and mediates endothelium - dependent relaxation of blood vessels. in the pathogesis of endotoxin shock vec is one of the major target cells of lps and lps - induced proinflammatory cytokine such as tumor necrosis factor and interlukin 1 and activated. in vec inducible nitric oxide synthase ( inos ) is induced and lead to an increase in production of no, the while endothelial nitric oxide synthase ( enos ) is inhibited and elicit decrease in no formation, both of which are demonstrated to induce the

    在內毒素休克過程中vec是lps及其誘導機體產生的多種促炎細胞因子如tnf 、 il - 1作用的主要靶細胞, vec誘導型一氧化氮合酶( induciblenitricoxidesynthase , inos )激活、 no大量誘生而內皮型一氧化氮合酶( endothelialnitricoxidesynthase , enos )活性被抑制、 no生成障礙,是血管反應性異常變化、血管調節機制紊亂的重要發病環節。
  5. Methods to extend the migration of endothelial cells on sprouts from four directions - moving up, down, left or right - in previous models to random direction in the present model, and develop a relevant 2d discrete model to simulate the process of tumor angiogenesis and compare the simulation results with some corresponding experimental data

    方法將新生血管芽尖的內皮細胞遷移方向由先前模型的上、下、左、右四個方向擴展為任意方向,建立相應的二維離散模型,數值模擬實體腫瘤微脈管系統的生成過程,比較模擬結果與相關實驗數據的符合程度。
  6. Angiostatin, a multiple kringle ( 1 - 4 ) - containing fragment of plasminogen, is an effective natural inhibitor of tumor angiogenesis, which is a potent inhibitor of angiogenesis in vivo, and selectively inhibits endothelial cell proliferation and migration in vitro

    血管抑素angiostatin是纖溶酶原的內源片段,包括四個kringle結構,是一種重要的內源性新生血管生成抑制劑。
  7. His research work mainly concerns microcirculation, shock, fibronectin, tumor necrosis factor, dysfunction of microvascular endothelial cells and mechanism of weight - reducing. more than 200 scientific papers were published in domestic or abroad journals

    近年來主要從事微循環、休克、纖維連接蛋白、腫瘤壞死因子、血管內皮細胞功能障礙及減肥機制等方面的研究,在國內外雜志上發表論文200餘篇。
  8. The ceps can differentiate and expand into mature endothelial cells. and cecs / ceps and vegf are helpful in tumor matastasis, prognosis and gene therapy research

    同時,血管內皮細胞體外分化擴增方法的建立,為血管內皮細胞功能、以及抗血管基因治療等的深入研究奠定了基礎。
  9. Discussions angiogenesis, an essential process for tumor growth, is regulated by endothelial proliferation factors and their inhibitors such as angiostatin

    討論血管生成對于腫瘤生長來說是一個必需的過程,受內皮細胞增殖刺激因子和抑制因子的共同調控。
  10. Angiostatin is a angiogenesis inhibiting factor derived from tumor, which can potently inhibit endothelial cell proliferation and migration and has an important application value in the treatment of solid tumor

    摘要血管生成抑制素是腫瘤衍生的血管生成抑制因子,能有效地抑制血管內皮細胞的增生和遷移,在實體瘤的治療中有重要的應用價值。
  11. To gain a molecular understanding of tumor angiogenesis, we compared gene expression patterns of endothelial cells derived from blood vessels of normal and malignant colorectal tissues

    為了能夠在分子水平上了解腫瘤的血管造影照片,我們對比了從正常和惡性結腸癌組織的血管分離到的上皮細胞的基因表達模式。
  12. Recent advances in biomedical research, such as vascular growth factors, clonality of hemangioma endothelial cells, immunophenotype, endothelial progenitor cells ( epcs ), somatic mutations of ih, provide insights into a deeper understanding of the etiology of the tumor of childhood

    近年來對與該病相關的血管形成生長因子、單克隆生長、免疫表型、內皮祖細胞、基因缺陷等多方面進行了較深入的研究。
  13. Inhibition of tumor - induced angiogenesis may represent an effective cytostatic strategy. angiostatin inhibits endothelial proliferation, angiogenesis, and tumor growth., so it is very important to receive a great deal of soluble, correctly folded, biologically ac

    Angiostatin已被證實是一種很具潛力的抗腫瘤生長和轉移的因子,因而若獲得大量具有高活性、高純度的angiostatin對腫瘤的治療會有重要的意義。
  14. Angiogenesis, the formation of new capillary vessels, is an essential step in tumor growth and matastasis. it has been thought that the angiogenesis is the migration and proliferation of existing endothelial cells within vascular endothelium

    對于腫瘤血管生成,傳統的觀點認為是由已有血管通過出芽方式完成的;但最近的研究證實外周血中的血管內皮及其前體細胞的動員與募集,對腫瘤血管生成具有十分重要的作用。
  15. [ objective ] tumor angiogensis is a critical step for the growth and metastasis of solid tumors. vascular endothelial growth factor ( vegf ) is a specific and potent angiogenic factor that mediates vascularization by binding to the kinase domain receptor ( kdr )

    [目的]本實驗試圖通過能中和vegf生物活性的單抗從噬菌體隨機7肽、 12肽文庫中篩選一種能抑制vegf與血管內皮細胞表面受體kdr ( kinasedomainreceptor )結合的vegf模擬短肽。
  16. Effect of cytochrome p450 expoxygenases on vessel endothelial cell apoptosis induced by tumor necrosis factor alpha

    腫瘤壞死因子凋亡誘導配體誘導白血病細胞凋亡中抵抗機制的初步探討
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