surfactant activity 中文意思是什麼

surfactant activity 解釋
表面活性劑效能
  • surfactant : n. 【化學】表面活化劑。
  • activity : n. 1. 活動；活躍；動作；活動力；能動性。2. 活性；放射性。3. 機能，功能。4. 〈美國〉機構。5. 〈pl. 〉 活動范圍。
  1. The length of alkyl, carbon chain about betaine type amphoteric surfactant affects its surface activity and other properties

    摘要甜菜堿表面活性劑的烷基碳鏈的長度對其表面活性及其它性質有不同程度的影響。
  2. The addition of surfactant to etchant can protect surface from intense dissolution, and keep a relative similarity of dissolving activity between surface and tunnel walls. in order to gain a satisfactory enlargement of tunnels, the etchant temperature should be controlled at 70 - 90

    3a / cm ，嚴重，使比容降低;溶液中添加表面活性劑可以有效地抑製表面溶解，提高比容;適宜的擴孔條件為:溫度80oc ; h +濃度lmol / l0
  3. The main effect factors include : 1. microbial species, which seem to have different degradation rate under varied circumstances and n - alkanes ; 2. physical and chemical properties of crude oil itself, which have an important effect on biodegradation, such as conglutination, boiling point, refractive index, ingredient and concentration of the oil ; and 3. culture condition of microorganism, under which the degrading activity of microorganism will be affected by surfactant, light condition, sorbent, nutrition, co - metabolite, oxygen, temperature and salinity during the inoculation of selected or indigenous microorganisms

    其中主要影響因素包括：菌種的影響，菌種在不同的環境中和對不同碳鏈長度的碳氫化合物表現出不同的降解效率；石油物質本身物理化學特性的影響，如石油物質在水體或土壤中的濃度以及石油的粘度、沸點、折射率等特性；生存環境條件的影響，在接種入高效率的降解菌或利用土著微生物進行降解時，降解率受到生存環境中各種條件的影響，如表面活性劑、光照條件、吸附劑的利用、營養鹽、共代謝底物、氧氣、溫度、鹽度等。
  4. Mechanism of glutamate - induced inhibition of pulmonary surfactant lipid synthesis ? mda receptor ( nmdar ) antagonist mk - 801 could reverse glutamate - induced inhibition of [ 3h ] - choline incorporation in lung explants ; ? he expression of nmdar1 subtype existed in alveolar type ii epithelial cells ; ? he effect of glu on pc synthesis required l - arginine in cultured lung explants, nos activity and no production were increased significantly in lung explants with glu treatment ; and mk - 801 and nos inhibitor l - nna could block the increase of nos activity and no production induced by glu ; l - nna also could reverse the inhibition of [ 3h ] - choline incorporation induced by glu ; ( 4 ) mda content increased and sod level decreased in cultured lung explants of glu treament groups, mk - 801 and l - nna could reverse these effects ; ( 3 ) mk - 801, l - nna could block the release of ldh induced by glu

    谷氨酸對肺表面活性物質脂質合成抑制的機制研究nmda受體拮抗劑mk - 801能逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應；肺泡上皮細胞有nmda受體nmdar1亞單位的表達； glu對ps脂質合成的影響依賴l -精氨酸的存在， glu可使肺組織勻漿中nos活性、 no合成量升高； mk - 801 、 nos的抑制劑l - nna均可阻斷glu所致的這些效應； l - nna可逆轉glu所致肺組織[ ~ 3h ] -膽堿摻入減少的效應； glu可使肺組織勻漿中mda碩士學位論文中文摘要含量增高、 sod水平下降。 mk七0 、 l nna能阻斷q所致的這些效應； mk80 、 l nna還能阻斷gill誘導的ldh釋放增多的效應。
  5. 3. calcitonin gene - related peptide attenuates glutamate - induced inhibition of pulmonary surfactant lipid synthesis ( d cgrp could reverse the decrease of [ 3h ] - choline incorporation induced by glu with dose - dependence ; ( 2 ) cgrp could block the decrease of the content of cct a mrna induced by glu in lung tissue ; ( 3 } cgrp could reduce the impairment of the ultrastructure of at ii cells induced by glu ; ( 4 ) cgrp could reverse the increase of mda content and decrease of sod level induced by glu in cultured lung explants, respectively ; ? grp had no significant effect on nos activity and increase of no production induced by glu

    降鈣素基因相關肽減輕谷氨酸所致肺表面活性物質脂質合成抑制的保護作用降鈣素基因相關肽grp ）可顯著減輕0所致肺組織h一膽堿摻入pc量的降低，並且呈劑量依賴保護效應； cgrp可逆轉gill所致cctqinrna含量的降低； cgrp可減輕o所致肺11上皮細胞超微結構的損傷； cgrp可逆轉o所致肺組織勻漿中mda含量增多、 sod水平降低的效應，並可逆轉q所致肺組織ldh釋放增多的效應； cg販對gill引起的nos活性和no含量的升高均沒有顯著性影響。
  6. Methods : in cultured lung explants without serum, the lipid component synthesis of pulmonary surfactant was evaluated in [ 3h ] - choline incorporation ; mrna content of phosphocholine cytidylyltransferase ( cct ) in lung explants was investigated in rt - pcr ; the changes of the ultrastructure of the at ii cells were observed with electron microscope ; the expression of nmdar1 subtype was observed in immunohistochemistry staining ; nitric oxide synthase ( nos ) activity, nitric oxide ( no ) content, superoxide dismutase ( sod ) level, malondialdehyde ( mda ) content and lactae dehydroase ( ldh ) level were determined by biochemistry methods. results : 1. influence of glutamate on synthesis of the lipid component of pulmonary surfactant ? with l - arginine, glu inhibited [ 3h ] - choline incorporation with good dose - dependence and time - dependence ; ( 2 ) mrna content of cct of the glu treatment groups was decreased ; ( 3 ) glu increases the release of ldh in cultured lung explants ; ( dwith electron microscope histochemistry, glu induced the changes of the ultrastruture of at ii iv cells

    方法：採用成年大鼠肺組織無血清培養，運用[ ~ 3h ] -膽堿摻入法測定ps主要脂質磷脂酰膽堿（ pc ）合成量； rt - pcr擴增檢測肺組織中pc合成限速酶磷酸膽堿二胞苷酰基轉移酶（ cct ） mrna含量；透射電子顯微鏡法觀察肺泡型上皮細胞和ps系統超微結構的變化；免疫組織化學染色檢測glu的受體nmdar1亞單位的表達；生化測定肺組織乳酸脫氫酶（ ldh ）釋放量和肺組織勻漿中一氧化氮合酶（ nos ）活性、一氧化氮（ no ）生成量、超氧化物歧化酶（ sod ）水平以及丙二醛（ mda ）含量。
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