transcription complex 中文意思是什麼

transcription complex 解釋
轉錄復合物
  • transcription : n 謄寫,抄寫;轉錄;抄本,繕本,副本;轉寫,翻譯;【音樂】樂曲改作;【無線電】錄音;錄音廣播;(...
  • complex : adj 1 復雜的,錯綜的。2 合成的,綜合的;【化學】絡合的。3 【語法】復合的;含有從屬子句的。n 1 復...
  1. At rest, the heterodimeric rel / nf - k b complex is located in the cytoplasm bound to an inhibitory factor, i k b. upon stimulation, i k b is phosphorylated and degraded, free nf - k b then translocates into the nucleus where it binds to k b site to regulate transcription

    靜息時, re皿4bh聚體與抑制蛋白kbs結合,在細胞漿中保持無活性。受到刺激, ikb分子迅速磷酸化並降解,釋放nf兒b轉位入核,通過與kb位點結合調節轉錄。
  2. 13 chatterjee - kishore m, wright k l, ting j p, stark g r. how stat1 mediates constitutive gene expression : a complex of unphosphorylated stat1 and irf1 supports transcription of the lmp2 gene. embo j., 2000, 19 : 4111 - 4122. 14 mahboubi k, pober j s. activation of signal transducer and activator of transcription 1 stat1 is not sufficient for the induction of stat1 - dependent genes in endothelial cells

    例如, jak - stat中, socs1 ppn以及stat1蛋白對于系統輸出的影響相對其他蛋白來說要明顯的多,這三個蛋白主要控制著jak - stat信號通路的「信號幅度」和響應強度response magnitude ,最大響應值。
  3. The elicitor of this pathway is ca2 +, ca2 + binds to sos3, which leads to interaction with sos2, a ser / thr protein kinase of 446 amino acids, and activated the kinase. the transcription and post - transcription of sosl, an putative na + / h + antiporter on the membrane is controlled by the sos3 - sos2 complex

    這條途徑的起始為外界高鹽刺激使細胞內ca ~ ( 2 + )水平發生變化, ca ~ ( 2 + )作為第二信使與sos3結合,然後導致sos3與sos2相互作用激活sos2的激酶活性,形成sos2 - sos3激酶復合體。
  4. Abstract : plant responses to salt stress via a complex mechanism, including sensing and transducing the stress signal, activating the transcription factors and the corresponding metabolizing genes. since the whole mechanism is still unclear, this review emphasize the biochemical events during the plant adaptation to salt stress referring to an index of importance : the homeostasis in cytoplasm, the biosynthesis of osmolytes and the transport of water. most of these biochemical events were elucidated by study of halophyte and salt - sensitive mutations, also many important genes involved were cloned and used to generate stress - tolerance phenotypes in transgenic plants. on the other hand, about the molecular mechanism in signal transduction, the research of arabidopsis mutations and yeast functional complementation provided helpful traces but not full pathway

    摘要植物對鹽脅迫的耐受反應是個復雜的過程,在分子水平上它包括對外界鹽信號的感應和傳遞,特異轉錄因子的激活和下游控制生理生化應答的效應基因的表達.在生化應答中,本文著重討論負責維持和重建離子平衡的膜轉運蛋白、滲調劑的生物合成和功能及水分控制.這些生理生化應答最終使得液泡中離子濃度升高和滲調劑在胞質中積累.近年來,通過對各種鹽生植物或鹽敏感突變株的研究,闡明了許多鹽應答的離子轉運途徑、水通道和物種特異的滲調劑代謝途徑,克隆了其相關基因並能在轉基因淡水植物中產生耐鹽表型;另一方面,在擬南芥突變體及利用酵母鹽敏感突變株功能互補篩選得到一些編碼信號傳遞蛋白的基因,這些都有助於闡明植物鹽脅迫應答的分子機制。
  5. In eukaryotes it may bind to proteins in a transcription complex to inhibit its action, or it may bind to a transcription factor

    在真核生物中,它可能與蛋白質結合形成一種轉錄復合體,抑制轉錄作用,或與轉錄因子結合在一起。
  6. By combining the information from the cdna sequences with functions of their homologs, we suggest that two kinds of hormones may play important roles during flower regeneration in a few ways : ( i ) proteolytic system of ubiqutin / 26s proteosome complex, ( ii ) actin - dependent vesicular cycling of auxin efflux carrier, ( iii ) regulating transcription of genes, ( iv ) phosphorylation and dephosphorylation

    經過進一步的篩選,結合序列特徵和同源基因的功能信息對這些基因功能進行了初步分析。認為兩種外源激素可能通過泛素26s蛋白酶體復合體對調節蛋白的降解機制,生長素運輸載體肌動蛋白依賴的小泡運輸,基因轉錄調節,磷酸化和去磷酸化等調節過程誘導風信子花分生組織的再生。
  7. Encapsulate some complex reaction types as objects, including elementary mechanisms, enzymatic mechanisms and gene expression mechanisms, simplify the description of complex biochemistry reactions ; at the same time encapsulate typical biological processes, such as transcription and translation, so it can avoid a lot of repeated workload

    把復雜生物化學反應封裝成對象,包括基本的化學反應類型,酶反應機制和基因表達機制等,簡化了復雜生化反應的描述;同時對經常使用的典型生物過程,例如轉錄、翻譯過程等,封裝成對象,避免了大量重復性的工作,方便用戶的使用。
  8. It is well documented that they jointly take part in gene transcription that led by rna polymerase ii. however, whether baf complex and nf1 / ctf are also involved in the formation of rna polymerase ii - mediated transcription initiation complex is still uncertain, and a number of the currently proposed models still lack the support of experimental data

    哺乳動物swi snf ( baf )復合物和轉錄因子( nf1 ctf )都是轉錄活動的重要調控因子,許多實驗證實,它們共同參與了基因轉錄調控活動。 baf復合物與轉錄因子nf1 ctf是否一同參加了由rna聚合酶介導的基因轉錄起始復合物的形成,目前還沒有比較一致的認識,已有的模型仍缺乏實驗支持。
  9. 3, by immunofluoresent microscopy, we also found that brg1 did not clearly co - localize with rna polymerase ii small subunit b6, but partially co - localized with rna polymerase ii small subunit b8 ; nf1 / ctf clearly co - localized with rna polymerase ii small subunit b6 and partially with b8. these data implies that brg1 may be partially associated to b8 and nf1 / ctf may be involved in the formation of transcription initiation complex through associating to brg1 and b6

    3 、通過兔疫熒光實驗我們還發現, brg和rna聚合酶d亞基b6之間沒有明顯共定位現象,與rna聚合酶11小亞基bs有部分的共定位現象:呷1兒仆與刪a聚合酶小亞基孤有比較明顯的共定位,與rna聚合酶11小亞基bs有部分的共定位。
  10. Human elongator complex serves a role in chromatin remodeling through modifying the lysine residues of histone h3 and h4 during transcription elongation in higher cells. the main results and conclusions are as followings. 1 using depletion strain of yeast as material, we have established a system for complementation test and gene expression analysis of human elongator complex

    4 、利用點突變和plasmidshuffle等技術構建了含h3k14一r 、 h4ks ~ r單突變和h3k14一側h4ks一r雙突變的e1p3乙菌株,通過功能互補實驗分析少凡p3對突變株的補償功能,證實酵母elongator可對h314k / h4sk進行乙酞化修飾。
  11. This data indicated the expression of baf complex and nf1 / ctf is closely related to cell activation and gene transcription activities

    以上實驗表明, baf復合物和轉錄因子nf1 ctf在細胞中的表達是與細胞活化和基因轉錄活動密切相關的。
  12. An understanding of the complex chain of neurotransmitters, “ second messengers, ” transcription factors, genes and other miscellaneous molecules needed to make a long - term memory is leading to drugs that may ultimately help more than those beset with alzheimer ' s or more benign forms of dementias that plague the aged

    了解參與長期記憶的復雜過程,包括神經傳遞物、二級傳訊分子、轉錄因子、基因與其他五花八門的分子,最後引導我們研發的新藥物,不只有助於受到阿茲海默癥或其他失智癥問題困擾的年邁患者而已。
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