核苷一磷酸 的英文怎麼說

中文拼音 [līnsuān]
核苷一磷酸 英文
nucleoside monophosphate nmp
  • : 核構詞成分。
  • : 名詞[化學] (有機化合物的一類) glucoside
  • : 名詞[化學] phosphorus (15號元素,符號p)
  • : 酸構詞成分。
  • 磷酸 : [無機化學] orthophosphoric acid; phosphoric acid磷酸胺 [化學] phosphamide; ammonium phosphate; 磷...
  1. Ligase an enzyme that catalyzes the bond formation between two substrates at the expense of the breakdown of atp or some other nucleotide triphosphate

    連接酶:可將兩個底物結合在起的酶,此過程需要atp或其他供能。
  2. Biochemistry ) a nitrogenous molecule, either a pyrimidine or a purine, that combines with a pentose sugar and phosphoric acid to form a nucleotide, the fundamental unit of nucleic acids

    2堿基:種含氮的分子,嘌呤堿或者是嘧啶堿,它們和戊糖還有起構成的基本結構單位。
  3. Adp ( adensine diphosphate ) a nucleotide consisting of adenine and ribose with two phosphate groups attached. the second phosphate is attached to the first by a high - energy bond

    腺(嘌呤種由腺嘌呤和糖再加上兩個組成的。其中第二個以高能鍵與第結合。
  4. Adenosine triphosphate : an adenosine - derived nucleotide, c10h16n5o13p3, that supplies large amounts of energy to cells for various biochemical processes, including muscle contraction and sugar metabolism, through its hydrolysis to adp

    種由腺衍生的c10h16n5o13p3 ,通過它的水解作用到二可為細胞進行各類生化過程提供大量能量,包括肌肉收縮及糖份新陳代謝。
  5. To make clear the hypothesis, a middle cerebral artery occlusion ( mcao ) and hypoxia and glucose - deprivation ( hgd ) ischemic models were used in in vivo and in vitro study, respectively. we first studied the cellular localization of kvl. 2 and the co - localization of kvl. 2 protein and vegf receptors flk - 1 and flt - 1, observed the effect of mcao on kvl. 2 expression and phosphrylation in the rat brain in vivo, then investigated the effect of vegf on ischemia / hypoxia cell damage and tyrosine phosphorylation of kvl. 2 in sh - sy5y cells. finally, in order to further elucidate the relationship between vegf ' s neuroprotection and its regulation on kvl. 2 phosphorylation, we used a specific antisense oligodeoxynucleotide ( odn ) to knockdown the expression of endogenous vegf to observe its role in ischemia / hypoxia cell damage and regulation of kvl. 2 phosphorylation

    為了驗證上述假設,本文分別在整體和離體水平,採用大腦中動脈缺血( middlecerebralarteryocclusion , mcao )和體外氧?糖剝奪( hypoxiaandglucose - deprivation , hgd )缺血模型,首先了解了kv1 . 2蛋白的細胞定位及與vegf受體flk - 1和flt - 1的共存情況,觀察了整體mcao后缺血再灌不同時間大鼠腦內kv1 . 2蛋白的化水平變化,然後通過外源性給予vegf蛋白,在sh - sy5y細胞株上觀察其對缺血細胞存活率及kv1 . 2蛋白化水平的影響,最後利用vegf反義脫氧寡( oligodeoxynucleotide , odn )特異阻斷內源性vegf蛋白的表達,觀察內源性vegf蛋白在缺血細胞損傷及調節kv1 . 2蛋白化中的作用,以進步明確vegf缺血保護效應與其調節kv1 . 2蛋白化之間的關系。
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