毒性出現時間 的英文怎麼說

中文拼音 [dúxìngchūxiànshíjiān]
毒性出現時間 英文
toxic effect time
  • 毒 : Ⅰ名詞1 （對生物體有害的性質或物質; 毒物） poison; toxin 2 （毒品） drug; narcotics 3 （姓氏） a s...
  • 性 : Ⅰ名詞1 （性格） nature; character; disposition 2 （性能; 性質） property; quality 3 （性別） sex ...
  • 現 : Ⅰ名詞1 （現在; 此刻） present; now; current; existing 2 （現款） cash; ready money Ⅱ副詞（臨時; ...
  • 時 : shí］Ⅰ名1 （比較長的一段時間）time; times; days:當時at that time; in those days; 古時 ancient tim...
  • 間 : 間Ⅰ名詞1 （中間） between; among 2 （一定的空間或時間里） with a definite time or space 3 （一間...
  • 毒性 : [藥理學] toxicity; virulence; poisonousness毒性測定 toxicity test
  • 時間 : time; hour; 北京時間十九點整19 hours beijing time; 上課時間school hours; 時間與空間 time and spac...
  1. I often left nostril gives a few nosebleed, and basic it is halitosis, mouth every day ache of suffering, waist, and radical have a bowel movement is jalf congealed every time sodden, talking around gives birth to sore, do not want to drink water, mouth weak, chang youbai is phlegmy in larynx wall, tongue thick in vain, left nostril often relapses instead answer bleed one year between half, right nostril never has shed blood, examination of classics hospital nose division slants for bazoo rhinitis of music, serious irritability, but it is good to ate medicine to disappear, also have the thing of suffer from excessive internal heat rarely, can be meeting above expression, the thing that had very much cool and refreshing relieve internal heat or fever also has vanished, can deficiency of yin with irritability or be deficiency of yang, irascibility, internal heat or lobar fire

    我經常左鼻孔出少量鼻血、而且基本天天都是口臭、口苦、腰酸痛、而且基本大便每次都是溏爛、口舌生瘡、不想喝水、口淡、常有白痰在喉壁、舌厚白、刷牙出血、 （附早上煩噪、口乾上火、刷牙出血、且出左鼻孔出血癥狀更明顯，左鼻孔經常反反復復流血一年半時間了，右鼻孔從沒流過血，經醫院鼻科檢查為鼻偏曲、嚴重過敏性鼻炎，但吃了藥不見好，也很少吃上火的東西，可就是會以上表現，吃了很多清涼解毒的東西也不見好，會不會陰虛或是陽虛、肝火、心火或肺火呢？ ）
  2. In trpsin tolerance assay. this virus could resist to 1 % trpsis at 37 in an hour. in acid tolerance assay, this virus was resistant to ph3. 0 and ph5. 0 at 37 in 2 hours, and the average infection litre of the virus decreased little. in heat assay, at 50, the virus was processed from 5 minutes to 150 minutes and at each condition the viral virulence reduced to some certain degree. among these conditions, when at 50 in 30 minutes. the average infection litre of this virus decreased over 2 tilre. and when al 50 in an hour, cpe of ihis virus disappeared. when time was set for an hour. but with processed in different temperature as 50 60 70, 80, the virus losl the multiplication capacity complelely. in biological assay, we selected different cell lines to cultivate this virus by laking advantage of possesional cells at that time in our laboratory. then we found that fcwf cell line was the most sensitive to dxmv and mdck was the second. with f81 cell line, after passaged for 12 times continuously with low concentration of fcs. the virus could produce cpe. however, with vero cell line. the virus could not procuce any cpe after many passages. the hemagglutination and lumadsorption reaction test proved that this virus had no any reaction to erythrocyte of pig, fowl and cavy. by neutrolizaion assay, dxmv could be identified as a kind of ccv

    理化學研究表明，該病毒為rna病毒，對氯仿、乙醚敏感；胰酶試驗中，經37 、 1小時處理的病毒，仍然能夠在貓源細胞fcwf細胞上生長，並且毒力基本保持不變；耐酸性試驗中，病毒分別在ph5 . 0和ph3 . 0經37作用2小時，毒力僅下降一個滴度；耐熱性試驗中，該病毒在恆定溫度50 ，設定不同時間，從5分鐘到150分鐘，毒力均有不同程度下降，其中， 50作用30分鐘，病毒平均滴度下降2個單位； 50 ， 60分鐘， cpe消失；恆定時間1小時，設定不同溫度（ 50 - 60 - 70 - 80 ） ，病毒在細胞上完全喪失增殖能力， cpe消失。生物學試驗，利用實驗室現有條件，選擇不同的細胞系對該病毒進行培養，發現該病毒對貓源細胞fcwf最敏感； mdck細胞次之； f81細胞經多次傳代，亦可出現cpe ；而vero細胞則不敏感。血凝試驗表明，該病毒對豬、雞、人及豚鼠的紅細胞均無血凝性。
  3. The specification is affected toward virus of liver having second already history or the convalescence after second liver virus is affected, had appeared protection antibody has immune power to second liver virus namely, do not need to medication, do not need vaccine of inject second liver, generally speaking, the protection antibody duration that this kind of natural infection forms is long, often be lifetime

    說明既往有乙肝病毒感染史或者乙肝病毒感染后恢復期，已經出現保護性抗體即對乙肝病毒有免疫力，不需要藥物治療，不需要注射乙肝疫苗，一般來說，這種自然感染形成的保護性抗體持續時間長，往往是終生的。
  4. The apoptosis induced by extract of russula subnigricans hongo was investigated in little white rat liver and kidney cells by agarose gel electrophoresis. the result showed that agarose gel electrophoresis of dna extracted from poisoned little white rat liver and kidney cells revealed typical 180 ~ 200bp integer - fold " ladder " " bands. apoptosis induced by extract of russula subnigricans hongo was dose - and time - dependentthe result indicated that extract of russula subnigricans hongo could induce apoptosis in little white rat liver and kidney cells

    4 .用電泳技術研究亞稀褶黑菇粗毒液誘導小白鼠肝腎細胞凋亡，小白鼠亞稀褶黑菇抽提液中毒后，肝腎細胞. dna經瓊脂糖凝膠電泳出現180一200bp整數倍的ona梯形帶， 19 . 09 / l一28 . 59 / l范圍內，亞稀褶黑菇提取液誘導肝腎細胞凋亡表現出時間和劑量依賴性
  5. The results showed that the function of mitochondrial began to decline after induction for 8h, and the declining rate was typical in time - dependent manners

    結果表明，在接種病毒后的8h時sl - 1細胞的線粒體出現了功能的變化，隨著時間的延長，功能逐漸降低且呈時間依賴性。
  6. Patients oer 70 years old were more likely than younger patients to hae a dose delay of at least a week at some point during the trial, the authors report, but there was no difference in the median number of cycles administered, dose reductions, or grade 3 - 4 toxicity

    作者們報道，大於70歲的病人與較年輕的病人相比，在該試驗的某一時間更可能出現至少一個星期以上的劑量延遲，而監控周期的中位數、劑量減少和3 - 4級毒性都沒有出現差異。
  7. The toxicity peaks always lagged the growth peaks, furthermore, the greater the biomass, the longer the lagging days. both mc - rr and mc - lr gave rise to their maximum value at the end of every single bloom and soon after a new bloom began. in general, the ratio of mc - rr and mc - lr remained comparatively constant during every bloom, but no obvious change characteristic was observed on mc - yr content. in the discussion, in was found that toxin - producing of microcystis was related to biomass, nutrients and water temperature

    分析各個階段的水華，我們發現毒性峰總是滯後於生長峰，且生物量越大，滯后的時間越長； mc - rr與- lr的含量在每個階段水華的末期達到最大，之後新一輪的水華開始出現，並且mc - rr與- lr的比例保持基本恆定，但是mc - yr的變化無明顯的規律。
  8. Thirdly, this paper compared the different responses of plants to these two stresses, the change of the defensive enzyme activity and the rate of losing water of leaves was different at two different stresses. the rate of losing water of leaves at drought stress was more rapid than that at salt stress, the leaves would wither and the change of activity of enzymes notably at drought stress, these results showed that the damage to plants was result from the oxidation at drought stress. at salt stress, the content of relative water of leaves changed slightly as well as did the activity of enzymes, these results showed that the damage to plants was caused by poison of ion and lack of nutrition

    三、通過研究兩種冰草植物對兩種脅迫的反應指出乾旱脅迫和鹽脅迫通過不同的途徑對植物進行傷害，主要表現在葉片的失水速率和保護酶活性的變化，乾旱脅迫短時間內葉片失水速率很快，葉片萎蔫，引起保護酶活性顯著變化，表明乾旱脅迫主要是通過氧化脅迫對植株造成傷害，而鹽脅迫下葉片相對含水量幾乎保持不變，表明鹽脅迫對植物的傷害主要通過離子毒害和營養虧缺造成。
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