白色突變體 的英文怎麼說
中文拼音 [báishǎitūbiàntǐ]
白色突變體
英文
albino mutant-
The topics include : structure and function of genes, chromosomes and genomes, biological variation resulting from recombination, mutation, and selection, population genetics, use of genetic methods to analyze protein function, gene regulation and inherited disease
主題包括：基因、染色體與基因組的結構和功能；來自於基因重組、突變和篩選的生物變異；族群遺傳學；運用遺傳學的方法分析蛋白質的功能，基因的調控和遺傳性疾病。The main results are as follows : 1 effects of light quality and kt on cotyledon enlargement 5 - d - old seedlings of arabidopsis wild type and mutants were harvested and the cotyledons were cut to use as excised cotyledon or the whole seedlings were used as intact cotyledon. excised and intact cotyledons were incubated in the medium ( ph 6. 0 ) with or without kt under white light ( wl ), red light ( rl ), far - red light ( fr ) and uv - b at 21 ? ? for some periods of time before cotyledon area were determined by a delta - t area measurement system
主要結果如下：一、光質和ky對于葉擴大的影響白光、紅光、藍光明顯促進5天齡幼苗離體和連體子葉擴大，遠紅光對子葉的擴大效應較弱， uv - b對子葉的擴大有抑制；對突變體的研究表明，紅光下光敏色素b 、遠紅光下光敏色素a 、藍光下隱花色素1對子葉的擴大起主要調節作用。In this experiment, three dead sheep from spa were been as materials to make histopathological and ultrastructural observation. the results showed the primary lesion was the resultant transformation of type ii epithelial alveolar cells. tumors were composed mostly of single layers of cuboidal epithelial cells arranged in an acinar pattern, but all rumors had areas where the tumor cells piled up, forming papillary projections into the alveolar spaces
本試驗以某羊場病死羊肺臟作為病料，製成病理組織切片，通過病理組織學檢查，結果表明：肺臟主要病變是肺泡細胞變為立方形或低柱狀細胞，這些細胞沿肺泡壁排列，突入肺泡腔內形成乳頭狀腺瘤結構；在氣管，支氣管腔內有灰白色泡沫狀液體，嚴重時使肺泡變為腺管狀等。The finding of smc proteins is a great breakthrough in the study of higher - order chromosome structure
因此， smc蛋白的發現是當前研究染色體高級結構變化的分子機制的重要突破。( 4 ) the color of the fishes in low temperature become darker, then changed bright when we transferred the fishes in high temperature. after three days, the color of these fishes recovered
（ 4 ）從實驗中可見低溫（門）使魚體色變深，突然升溫導致魚體色變白，隨著養殖時間延長， 3天後又恢復原來的體色。In 2001, wilson and his colleagues cloned another two members - human wnk1 gene and wnk4 gene which were located on chromosome 12 and 17, respectively. the two genes are disease - causing genes responsible for a mendelian hypertension. the disease - causing mutations are the large deletions in the first intron in wnk1 gene, causing increased expression in leukocytes
2001年， wilson等克隆了此家族的另兩個成員? ?人的wnk1和wnk4基因，分別位於12號和17號染色體，這兩個基因是一種孟德爾遺傳型高血壓病的致病基因，致病突變分別是wnk1基因第一內含子的大片段缺失，導致患者白細胞的基因表達量提高5倍左右。分享友人