突觸的 的英文怎麼說
中文拼音 [tūchùde]
突觸的
英文
synaptic-
However, the advance of intracellular labeling techniques enables us not only to visualize more complete dendritic arbor for qualitative analysis, but also to examine the relation between changes in the dendritic arborization and the evoked fast postsynaptic curents - 3 - ( fpscs ) in the same neurons during the postnatal development the aim of this study was to systematically examine the postnatal changes in the configuration of fpscs evoked by the focal stimulation of the stratum radiatum of the ca1 region, and the relationship between the dendritic arborization and evoked fpscs in the rat hippocampal ca1 pyramidal neurons using whole - cell blind patch recording technique combined with biocytin intracellular labeling during the postnatal development ( postnatal day 2 - 70, p2 - p70 )
但是,細胞內染色技術的進步使我們不僅能觀察到更完整的樹突分支來用於定性研究,而且也可以在同一神經元上研究在發育過程中樹突分支的變化與誘發的快突觸后電流( fastpostsynapticcurrents , fpscs )之間的關系。因此,本研究應用盲法腦片膜片鉗記錄並結合biocytin細胞內染色方法,對發育過程中(生后2 70天)局部刺激大鼠海馬ca1區輻射層在錐體神經元誘發的fpscs的成分變化,以及ca1錐體神經元的樹突分支與誘發的fpscs的關系進行了較為系統的研究。Nmda receptors contribute to synaptic transmission in anterior cingulate cortex of adult mice
受體參與小鼠的前額扣帶回的神經突觸傳遞Take charge of 《 eeffect of sodium cytidine triphosphate on synaptic plasticity in the focal cerebral ischemia in rats 》 and 《 study of anatomical factors on cervical spondylosis of nerve root type 》, which won second prize of science and tenology in chengde ; and 《 study of anatomical factors on cervical spondylosis of nerve root type 》 won third prize of the department of science and technology in hebei province as well
主持的《三磷酸胞苷二鈉促進局灶性腦缺血大鼠突觸重塑研究》獲承德市科技獎二等獎; 《神經根型頸椎病解剖因素研究》獲承德市科技獎二等獎,同時獲河北省科技廳三等獎。Study on neural excitatory transmission by wavelet entropy analysis of postsynaptic potentials
中樞興奮性傳遞的突觸后電位小波熵分析研究The defining morphological feature of chemical synapses is the vesicle cluster in the presynaptic nerve terminal
突觸小泡即刻可釋放池的結構組織化學突觸的定義形態學特徵是突觸前神經終端小泡群。Pubmed abstract : long - term potentiation of synaptic transmission in the hippocampus is the leading experimental model for the synaptic changes that may underlie learning and memory
《公共醫學圖書館摘要》 :腦部海馬回突觸的長期電位化是與學習和記憶相關的神經突觸變化中相當重要的一個模型。The inhibitory rate was 18 ? 4 % by nocistatin ( 1 nm ; n = 6 ) and 64 ? 10 % by compb ( 1 um ; n = 7 )
在鉗制電壓為刁0mv的狀況下; nociceptin可以明顯地抑制刺激as纖維和c纖維誘發的單突觸的興奮。The answer could be “ nothing ” : the earliest conception of the immune synapse being a kind of gasket enabling immune cells to direct their secretion of cytokines to a target cell may be the sole purpose of the structure
答案可能是什麼都沒有:最早關于免疫突觸的概念是,它是個類似束帆索的東西,可以讓免疫細胞將分泌出來的細胞素導向目標細胞,而這也可能是免疫突觸唯一的目的。Glia influence the formation of synapses and help to determine which neural connections get stronger or weaker over time ; such changes are essential to learning and to storing long - term memories
神經膠細胞會影響突觸的形成,也會幫助決定神經連結隨時間而增強或減弱,這種變化是學習與儲存長期記憶所必需的。Topics include : neural induction and pattern formation, cell lineage and fate determination, neuronal migration, axon guidance, synapse formation and stabilization, activity - dependent development and critical periods, development of behavior
主題包括:神經誘導和模式形成,細胞譜系和細胞命運確定,神經元遷移,軸突引導,突觸的形成和穩定性,依賴性活動的發育和危險期,行為的發育。You call these synaptic pathways
你們稱這些為突觸的小路。A decade later these structured synapses formed by immune cells are still generating questions : about how cellular machinery or other forces produce the synaptic architecture, how the architecture, in turn, might regulate cell - to - cell communication, how its malfunction could lead to disease, and even how pathogens might exploit the mechanism to their own advantage
時間過了10年,這種由免疫細胞所形成、結構分明的突觸,仍然不斷帶來新的疑問,例如:是什麼樣的細胞機制或其他力量,造就了這些突觸的構造,而這些構造又是如何調節細胞與細胞之間的溝通;突觸構造的病變將如何引發疾病;還有病原體是否可能利用這種突觸機制,來達成自己的目的等。Thus, correlated pre - and postsynaptic activity alters not only the strength of the activated input, but also its dendritic integration with other inputs
因此,突觸前後神經元的相關活動不僅影響被激化突觸的傳遞效率,還影響樹突的信號整合特性。A change in functional properties of the nmda receptors has been invoked as a potential mechanism contributing to the loss of synaptic plasticity during brain maturation. from this, it is important to well understand the structural and functional diversity of nmda receptors in relation to nmda receptor subunit composition and spatial distribution at excitatory synaptic sites during development
近幾年來,在哺乳動物中樞神經系統中,對離子型谷氨酸受體如何定位到興浙江大學醫學院碩士論文nrzb亞單位的c末端胞內區影響n協da受體裝配、運鈞以及表面表達的分子機制奮性突觸的研究有了長足的發展。Using this system, we have studied matrine - inhibittory effect and trifluoperation - neuroprotection effect in hippocampal slices, also discussed the mechanism of long - term potentiation using anesthetic rats. the experiment results showed that matrine can inhibit the hyperactivity induced by penicillin sodium in dosage by changing the relative parameters of field potential ; trifluoperation can alter ps change with the time, enhance the degree and the ratio of ps recovery, then minis the hypoxic injury ; high frequency stimulate can increase ps amplitude and epsp slope for long time, buildup the in / out function of nerve cells, and enhance synaptic plasticity
結果表明,苦參堿能夠劑量依賴性地抑制青霉素誘導的神經元順向信號傳導激活過程,使細胞外記錄到的場電位各個參數發生相應改變;三氟拉嗪可以改變ps的時相變化,提高ps的恢復程度和恢復率,減小了神經元因缺氧引起的不可逆損傷;高頻刺激( highfrequencystimulate , hfs )可以長時間的增強ps的幅度和epsp的斜率,進而增強神經元的輸入輸出功能,增加了突觸的可塑性。Accumulating evidence indicates that nmda receptors are involved in many complex physiological and neuropathological mechanisms, such as neuronal developmental plasticity, long - term potentiation ( ltp ), learning and memory, excitotoxicity and neurodegenerative diseases etc. three gene families that encode nmda receptor subunits have been identified : nr1, nr2a - d, and nr3 subunit
已知nmda受體與許多復雜的生理和病理機制有關,如突觸的可塑性、長時程增強作用( ltp ) 、學習和記憶、興奮性神經毒性、神經退行性變性疾病等。已克隆到3個編碼nmda受體亞單位蛋白的基因家族,即nr1 、 nr2和nr3 。The lab incorporates a wide variety of staining and dynamical imaging techniques for locating synapses and characterizing their functional capabilities
本實驗室納入了廣泛的染色方法,還有針對突觸的定位、區辨突觸功能能力而使用的動態顯影技術。Serotonin is a neurotransmitter, one of the molecules that jumps the tiny gaps known as synapses between neurons to relay a signal from one such brain cell to another
血清張力素屬于神經傳遞物的一員,這種分子會在神經元之間稱為突觸的狹小間隙中跳躍。These travelling waves occur hundreds of times a night, and most commonly at a frequency, 1 cycle per second, which has been shown to depress the activity of synapses
這些擴布的電波一晚上會產生上百次,大多數的頻率是每秒鐘擴布一輪,它們使突觸的活動減弱。Discovery of the immune synapse and its ongoing exploration has been made possible by new high - resolution microscopy techniques and computer enhancement of older imaging methods
新的高解析顯微技術以及經過電腦強化的舊顯影技術,促成了免疫突觸的發現以及持續進行的研究探索。分享友人