缺失型突變體 的英文怎麼說
中文拼音 [quēshīxíngtūbiàntǐ]
缺失型突變體
英文
depletion mutant-
In most wild type e. coli strains containing heterologous pha synthase genes, the amount of pha accumulation was almost undetectable while in e. coli km32b the amount of pha accumulation were clearly visible
在多種野生型大腸桿菌中，盡管使用了各種不同的表達載體，也很難獲得可供檢測的pha ；但應用大腸桿菌的fqdb缺失突變株卻可以獲得較多量的pha 。Here we found g proteins also function in leaf, silique development and the yield of pollen microspore. we observed several traits or characters in the offsprings of gpal, agbl null mutation and gpa1 overexpression lines and found that the width of mutants " lamina is larger than that of the wild type, whereas the lamina length, petiole length and rosette diameter is smaller than the wild type, the ga overexpression lines is different from the mutants ; the silique length and the pedicel length is larger in mutants than that of wild type, and slightly smaller in overexpression lines than the control ; the morphometric character in silique tip is different in gpal from agbl mutants ; the yield of pollen microspore is larger in null mutants than wild type whereas smaller in overexpression lines
實驗中我們跟蹤觀察了多代異三聚體g蛋白a亞基超表達轉基因植株及a ， p亞基缺失突變體的表型特徵，發現突變體的葉片寬度大於對應的野生型，葉片長度，葉柄長度及蓮座直徑小於野生型，而超表達植株的上述某些特徵與突變體相反; gp時突變體的長角果長度，花梗柄部長度大於野生型，而超表達ga植株種英則略小於對照; gpal突變體長角果尖端未出現咭乙i突變體的特徵: gpal ，口gbl突變體花粉生成量大於野生型，而超表達ga植株的花粉生成量則略小於對照。Typical accident - causing " theories includes heinrich " s domino " s theory, its development by bird, adams and other people, progress of which is that it is deficiencies of management system that are the root cause in accident causation sequence. this is author " s idea resource and main method to produce the water traffic accident - causing theory. in addition, uncontrolled transferring of energy theory, applies widely in safety management scheme for water traffic ; human error accident model can be used to analyze causes of collision accident and to take preventive measures ; perturbation theory and multilinear event sequencing method is a very useful investigation method to water traffic accidents
有代表性的事故致因理論有海因里希因果連鎖理論以及博德、亞當斯等人對該理論的發展，這些發展的主要進步是突出了管理缺陷是事故因果連鎖中的基本原因，是本文分析和發展水上交通事故致因理論的主要思想來源和方法論；能量意外轉移理論，廣泛的應用在水上交通安全的各項具體管理制度中；人失誤事故模型能用於指導對碰撞事故的原因分析和預防；動態變化理論及多線性事件連鎖論對指導事故調查是較好的分析方法；軌跡交叉理論雖然以獨立的理論形式存在，但本文認為其實質仍然是因果連鎖理論的發展。The judgement formula for high and low speed landslide and a new theory of stiffness - effect instability are presented for the instability of planar - sliding slope by the catastrophe theory. it is pointed out that the limit equilibrium stability analysis method of rigidity body has a lot of defects. the relationship between the action of outside environment and the response of slope system is complicatedly nonlinear according to the nonlinear dynamics model of slope evolution built
針對斜坡平面滑動失穩問題，運用突變理論方法，給出了快速滑坡和慢速滑坡發生的判據，驗證了剛體極限平衡穩定性評價方法有較大缺陷；根據斜坡演化非線性動力學模型，發現斜坡演化過程會出現混沌現象，其通向混沌之路是通過倍周期分叉實現的。It was devoted that class ii promoter was stimulated by ie180. there is not tata box on the upstream of the promoter, but there are binding sites of protein in the upstream of the promoter. david and his company identified ie gene in the human cytomegalovirus virus dna segment
目前國內外沒有關於野生型的ie180或其突變體對sv40和cmv啟動子調控作用的報導，此研究可為ie180基因缺失突變體，在人的巨噬細胞病毒和猴空泡病毒40控制方面的應用提供理論依據。The level of cytosolic free calcium in gp mutants is not different. adding exogenous purified cam to the cultural medium increased the pollen germination, tube growth, and cytosolic free calcium concertration in both wild type and ga mutant. however, the former was more sensitive to exogenous cam than the latter
外源鈣調素（花椰菜和擬南芥11亞型， 10一7m ）處理能夠提高野生型及gpol缺失突變體中的游離鈣離子水平，但對野生型花粉細胞游離鈣離子水平的激活效應高於gpal缺失突變體。In 2001, wilson and his colleagues cloned another two members - human wnk1 gene and wnk4 gene which were located on chromosome 12 and 17, respectively. the two genes are disease - causing genes responsible for a mendelian hypertension. the disease - causing mutations are the large deletions in the first intron in wnk1 gene, causing increased expression in leukocytes
2001年， wilson等克隆了此家族的另兩個成員? ?人的wnk1和wnk4基因，分別位於12號和17號染色體，這兩個基因是一種孟德爾遺傳型高血壓病的致病基因，致病突變分別是wnk1基因第一內含子的大片段缺失，導致患者白細胞的基因表達量提高5倍左右。分享友人